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At least 19 recordsLinked to original sources

[Restoration of effective artificial cardiac pacing by isoproterenol].

Since the pacing threshold of artificial pacemakers sometimes increases and causes ineffective pacing, emergency restoration of cardiac pacing may occasionally be required. In such situations sympathomimetic agents, such as isoproterenol, increase excitability and lower threshold. A 83-year-old man with acute ineffective pacing and hemodynamic instability is reported. Intravenous administration of isoproterenol restored effective, artificial cardiac pacing.

Aged↗

Artificial cardiac pacing. 1. Indications.

With increasing use of artificial cardiac pacing, criteria for selection of patients have been refined. In general, the most important indication for pacing is the presence and severity of symptoms due to bradyarrhythmias. Use of pacing in acute myocardial infarction remains controversial, but some guidelines are presented here.

Acute Disease↗

[Arterial hypotension induced by artificial cardiac pacing under general anaesthesia (author's transl)].

A case of haemodynamic decompensation is reported in a patient with a sick-sinus syndrome on a demand cardiac pacemaker, under-going surgery under general anaesthesia. While systemic arterial blood pressure showed adequate stability under the patient's own sinus drive, it consistently fell by 15 to 20 p. cent when artificial pacing was established. The possible mechanisms are discussed. The drop in arterial blood pressure with artificial pacing is probably due to the loss of the atrial contribution in maintaining adequate cardiac output. In the conscious patient, the resulting decrease in cardiac output is compensated by an increase in the peripheral resistance. Under general anaesthesia, this compensatory mechanism is lost, resulting in a drop in cardiac output and systemic arterial blood pressure. It needs to be emphasized therefore, that the patient who has a permanent pacemaker has significant under-lying cardiovascular disease and, that, continuous monitoring is indicated not only during surgery but also postoperatively until patient has stabilized.

Aged↗

Practices and outcome of artificial cardiac pacing in 154 dogs.

Artificial pacing (AP) is a treatment for symptomatic bradyarrhythmias unresponsive to medical therapy. This retrospective study was designed to define the practices and outcome of AP in dogs at 7 referral institutions participating in the Companion Animal Pacemaker Registry and Repository (CANPACERS). The indications, implantation techniques, complications, long-term outcome, and owner satisfaction were examined. One hundred fifty-four dogs were identified as undergoing AP from January 1, 1991, to January 1, 1996. Third-degree atrioventricular (AV) block (n = 91; 59%) and sinus node dysfunction (n = 45; 29%) were the most common indications for AP Transvenous endocardial AP systems were implanted in 136 dogs (88%), and epicardial systems were implanted in 18 (12%). Complications associated with AP were reported in 84 dogs (55%). Major complications occurred in 51 dogs (33%), including dislodgement of the pacing lead (n = 15; 10%), generator failure (n = 10; 6%), cardiac arrest during implantation (n = 9; 6%), and infection (n = 7; 5%). Minor complications occurred in 47 dogs (31%), including seroma formation (n = 18; 12%), muscle twitch (n = 17; 11%), and inconsequential arrhythmias (n = 15; 10%). Fourteen dogs (9%) experienced both major and minor complications. Survival analysis revealed 1-, 2-, and 3-year survival rates of 70, 57, and 45%, respectively. Age and presence of preexisting congestive heart failure (CHF) had a negative effect on survival (P = .001). Sixty percent of dogs with preexisting CHF died within 1 year of implantation, whereas 25% of dogs without heart failure died during the same period. Owners rated their satisfaction with the procedure as high in 80% of the dogs.

Animals↗

Usefulness of 24-hour recordings of electrocardiogram for the diagnosis and treatment of arrhythmias with special reference to the determination of indication of artificial cardiac pacing.

Twenty-four-hour recordings of electrocardiogram (ECG) were obtained from 1528 subjects. Sick sinus syndrome (SSS) was observed in 34 and advanced or complete AV block (CAVB) in 13 subjects. During the period studied 21 patients had died, who showed complicated ventricular premature contractions in 9 (multiform 6, short run 2, repetitive 2). Seventy-three patients with artificial cardiac pacing who were admitted at three different institutions were analyzed. The discrepancy of degree of block was observed between His bundle electrocardiographic findings and 24-hour ECG recordings. In 15 patients there was no correlation between sinus node recovery time following overdrive suppression and pauses following the termination of tachycardia (r = 0.0896, N. S.). In 49 normal subjects 24-hour heart rate ranged from a high of 170 to a low of 36 beats/min. Longest R-R interval ranged from 2.08 to 0.86 sec (mean 1.25 +/- 0.22 sec). The critical level of longest R-R interval to differentiate between normal group and groups with SSS or CAVB was about 1.6 sec. Above results suggest that Holter monitoring permits a precise diagnosis of arrhythmias and that both HBE and Holter monitoring should be used for the determination of pacemaker implantation especially in those with syncope or dizziness but without evidence of marked bradycardia.

Adolescent↗

Artificial cardiac pacing: 2. follow-up and complications.

Following insertion or implantation of an artificial cardiac pacemaker, both physician and patient are involved in aftercare to monitor the patient's cardiac status, the condition of the implantation site, and the function of the pacemaker. Among the complications that may occur are pacemaker malfunction and perforation of the ventricles. The physician and patient must also be alert to the possibility of electrical interference and to the physiologic factors that can modify pacemaker function.

Arrhythmias, Cardiac↗

Postcountershock pulseless rhythms: response to CPR, artificial cardiac pacing, and adrenergic agonists.

Clinically, countershock of ventricular fibrillation (VF) may result in asystole or a pulseless rhythm in more than 50% of attempts. We conducted a study to assess the effects of immediate artificial pacing, CPR, and adrenergic drug therapy in the management of postcountershock pulseless rhythms. Thirty-four episodes of VF followed by countershock were studied in eight anesthetized dogs. Transducer-tipped catheters were positioned in the ascending aorta (Ao) and right atrium (RA). A bipolar pacing catheter was advanced to the apex of the right ventricle and a catheter for measurement of coronary sinus blood flow (CSQ) (continuous thermodilution technique) was positioned in the coronary sinus. VF was induced electrically and a countershock at 400 J was given two minutes later; CPR was not performed during VF episodes. Countershock was followed by asystole or a pulseless rhythm in all animals. Immediate endocardial pacing (0.1 to 5 mA) of bradyarrhythmias produced electrical capture but did not result in arterial pressure pulses in any animal. After pacing, CPR was performed for two minutes or until restoration of spontaneous circulation (ROSC). During CPR, the diastolic coronary perfusion gradient (Ao-RA) was 20 +/- 7 mm Hg (mean +/- SD) and CSQ was 14 +/- 7 mL/min/100 g (53% +/- 43% of control). ROSC followed CPR of less than two minutes duration in 24% of VF study episodes. If ROSC did not follow two minutes of CPR, 1 mg epinephrine, or 50 micrograms or 100 micrograms isoproterenol was given IV.(ABSTRACT TRUNCATED AT 250 WORDS)

Animals↗