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Biomedical subjects

F Uchino

Publications and source records attributed to F Uchino.

At least 91 records · Page 5Linked to original sources
PubMed1976

Fine structures of the spleen in hereditary elliptocytosis.

Light and electron microscopic studies of the spleen and liver from a patient with hereditary elliptocytosis have been reported. Characteristic findings are congestion of the cordal space, diminished variation of the erythrocyte shape in the cord, relatively empty sinuses and erythrophagocytosis by the cordal macrophages. The same basic defect of the red cell membrane as has been documented in hereditary sperocytosis might be a possible explanation on the pathogenesis of this rare hemolytic anemia.

Elliptocytosis, Hereditary↗
PubMed1976

Pathological study on amyloidosis--the fine structure of amyloid-laden liver as revealed by freeze-etching.

Amyloid in freeze-etched specimen of liver of mice receiving subcutaneous in sodium caseinate was studied morphologically. The convex surface and cross surface of amyloid nodule were noted. The three dimentional structure of the amyloid fibrils which showed felt-like structure, globular profiles among amyloid fibrils and amyloid bundles were clearly visible. Amyloid bundles enveloped by the cytoplasmic membrane of the Kupffer cell projected from the deeply concave surface of the cytoplasmic membrane into the Disse's space and showed the appearance of ston-column-like structure. The amyloid bundles projecting from the invaginated cytoplasmic membrane were closely in contact with the cytoplasmic membrane and deeply rooted in the cytoplasm of the Kupffer cell.

Amyloid↗
PubMed1975

Type IV glycogenosis - a study of two cases.

Liver biopsy materials of two siblings with type IV glycogenosis were studied by light and electron microscopy. Biochemical analysis was added using autopsy material in one of the two cases. Two kinds of polysaccharides were noted not only in the cardiac muscle, skeletal muscles, smooth muscles and reticuloendothelial cells, but also in the neutrophils and platelets. One was glycogen and the other was similar to amylopectin. Ultrastructurally, a large amount of fibrils, 60 A in width, glycogen rosettes and glycogen granules were detected in those cells. Branching glycosyltransferase deficiency was biochemically confirmed in one case examined.

Amylopectin↗