Search PubMed⌕ Search
Home/Archive/PubMed

PubMed · 895607

Brain damage and abnormal behaviour in children.

B Herzberg·L Herzberg
View original source ↗Export metadata ↓

Abstract

Behavioural problems in children with suspected brain damage may present considerable diagnostic and management problems. Methods of assessment are still imprecise and minor degrees of brain damage are seldom detectable. Data from clinical studies highlight the interrelationship between environmental and organic factors in the pathogenesis of disturbed behaviour. Children from large families of low socioeconomic status, who are subject to parental neglect, are particularly susceptible to the development of psychiatric disorder in the presence of brain damage.

Explore related subjects

Brain Damage, ChronicBrain InjuriesCerebral PalsyChildChild Behavior DisordersFemaleHumansHydrocephalusInfant, NewbornInfant, Premature, DiseasesMalePregnancyPregnancy ComplicationsJournal Article

Keep this discovery

Explore connections, maps & timelines

BibTeXRIS

B Herzberg, L Herzberg. 1977-06-04. Brain damage and abnormal behaviour in children.. https://doi.org/10.5694/j.1326-5377.1977.tb131173.x

Cite the original work for its findings. Save a collection to share your selection of sources.

KEEP EXPLORING

Related citations

PubMed2004

Multimodality imaging in partial epilepsies.

PURPOSE OF REVIEW: The rapid expansion of novel applications and the development of new techniques in structural and functional imaging modalities present a troubling challenge to keep up with and harness potential valuable information created in this critical field of epilepsy localization and non-invasive in-vivo research. RECENT FINDINGS: Recent advances in epilepsy imaging have centered on: (1) improving the localization of epileptogenic tissue beyond that of state-of-the-art structural magnetic resonance imaging; (2) monitoring the development and progression of epileptogenic pathology, particularly mesial temporal sclerosis; and (3) an investigation of the in-vivo structural and functional disturbances underlying and revealing mechanisms of partial epilepsy pathophysiology. SUMMARY: The main impact of the progress in epilepsy localization with multimodality imaging is to allow more effective presurgical evaluation and the selection of patients with intractable seizures. By combining serial imaging findings and genetic studies, the major questions surrounding the development and progression of mesial temporal sclerosis with regard to the cause and consequence of epilepsy will soon be answered. Long-standing questions concerning in-vivo metabolic and neurotransmitter disturbances associated with partial epilepsy, detected and depicted (but not understood) with magnetic resonance spectroscopy and positron emission tomography, are finally being addressed.

Brain Damage, Chronic↗
PubMed2003

[Neuromonitoring of patients following severe brain trauma].

To a considerable extent, the neurological outcome of patients with severe brain trauma is determined by the primary injury caused by the accident. Substantial progress has been made in our understanding of the pathophysiological processes resulting in secondary brain damage after brain trauma, partly as a result of the introduction of advanced techniques of neuromonitoring. Early recognition and treatment of the symptoms leading to this type of brain damage seems crucial to the effort to improve the neurological outcome in these patients. Useful modern neuromonitoring techniques include: measurement of the intracranial and cerebral perfusion pressure and continuous electroencephalography. Methods that are also reliable and readily applicable, but less relevant clinically, include cerebral microdialysis of fluid from the extracellular space, determination of the parenchymal oxygen tension, and determination of the venous oxygen saturation. Other techniques that are not clinically applicable include: determination of the cerebral blood flow, the cortical tissue perfusion or the regional cerebral oxygenation.

Brain Damage, Chronic↗