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PubMed · 3631555

Coital death.

O Steeno
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Abstract

Generally speaking one can state that there exists insufficient information to be alarmed about coital death; indeed, there are almost no studies known about coital death of heart patients. All the same an epidemiologic research is absolutely necessary for profylaxis of heart attacks caused by sexual activity, even more whilst the present population shows an increased rate of arteriosclerosis and disposes over more spare time. It is remarkable that apart from medico-legal practicians, cardiologists and revalidation doctors, other disciplines as psychiatry and sexuology, still haven't paid any attention to the problem of coital death. It certainly is high time to draw this phenomenon out of its taboo-sphere.

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Angina PectorisCoitusCoronary DiseaseFemaleHeart ArrestHeart RateHumansMaleMyocardial InfarctionJournal Article

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BibTeXRIS

O Steeno. 1987. Coital death.. https://doi.org/10.1111/j.1439-0272.1987.tb02337.x

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Effect of a hypoglycemic agent on ischemic preconditioning in patients with type 2 diabetes and stable angina pectoris.

OBJECTIVE: Ischemic preconditioning is an increased tolerance to myocardial ischemia during the second of two consecutive exercise tests. ATP-sensitive K(+) channel blockers, such as glinides and sulfonylurea drugs, can induce loss of ischemic preconditioning. This study aimed to investigate the effects of repaglinide, a hypoglycemic agent with an affinity for myocardial ATP-sensitive K (+)channels, on the results of consecutive exercise tests in patients with diabetes and multivessel coronary artery disease. METHODS: Forty-two patients with type 2 diabetes and chronic stable angina pectoris, and two-vessel or three-vessel disease participated in this study. The patients underwent two consecutive treadmill exercise tests (phase 1). On the day after these exercise tests, 2 mg of oral repaglinide was given to the patients. One week later, two exercise tests were repeated consecutively (phase 2). RESULTS: All patients achieved 1.0-mm ST-segment depression during the four exercise tests (T1, T2, T3, and T4). In phase 2, seven patients improved in time to onset of 1.0-mm ST-segment depression. The worsening of the time to onset of 1.0-mm ST-segment depression in phase 2 demonstrated ischemic preconditioning block in 83.3% of patients (P=0.0001). Even the postexercise electrocardiographic parameters (ST-segment depression morphology and magnitude and arrhythmias) were significantly different between the groups with and without pharmacologic ischemic preconditioning block (P=0.031). CONCLUSIONS: Repaglinide, an oral hypoglycemic agent with ATP-sensitive K(+) channel-blocker activity, eliminated the myocardial ischemic preconditioning in patients with coronary disease and diabetes.

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