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Biomedical subjects

Y Tamari

Publications and source records attributed to Y Tamari.

At least 37 records · Page 2Linked to original sources
PubMed1986

Fluorometry of nanogram amounts of selenium in biological samples.

For fluorometry of selenium in human blood, hair, and liver and in leaves, we wet-ashed the samples with conventional nitric and perchloric acids, and then extracted piazselenol (complex of Se and 2,3-diaminonaphthalene) in cyclohexane. Selenium was back-extracted from the cyclohexane into nitric acid to remove the fluorometric interferences of trace amounts of organic compounds. This fluorometric method is rapid and suitable for routine analysis. We applied the method to human hair samples and compared it with the data for non-destructive neutron activation analysis of the hair.

2-Naphthylamine↗
PubMed1979

Neutron activation analysis of selenium and 17 elements in sediment.

Instrumental neutron activation analysis was performed to determine Na, Mg, Al, Cl, Sc, V, Cr, Mn, Fe, Co, Ni, Rb, Sb, Cs, Ba, Ce and Eu in the Pacific core sediment. Selenium was also determined by neutron activation analysis, by applying a solvent extraction method with 2,3-diaminonaphthalene (DAN) as a post-irradiation procedure, after the wet-ashing of the sediment. Homogeneity of trace constituents in the sediment and neutron flux corrections for accurate determination were also investigated.

Activation Analysis↗
PubMed1975

Functional changes in platelets during extracorporeal circulation.

An in vitro trauma test was conducted to determine the effects of extracorporeal circulation on platelet count and function. Fresh human blood was circulated in two identical in vitro circuits for six hours at a rate of 500 ml per minute (500 recirculations). One circuit included a G.E.--Peirce membrane lung and the other was a control. Platelet aggregation induced by adenosine diphosphate (ADP), epinephrine, or collagen was studied before and after six hours of perfusion. No important drop in platelet count occurred in the control circuit (Control-C) following bypass, but there was a 20% drop for the lung circuit (Lung-C). Platelet aggregation was reduced by about 30% for the control circuit and 65% for the lung circuit. The large decrease in platelet function accompanied by only a moderate decrease in platelet count is discussed in terms of loss of the youngest and most active platelets, platelet inhibition due to ADP released by red blood cell lysis, and platelet trauma.

Adenosine Diphosphate↗
PubMed1971

Effects of nicotine on salivary amylase secretion from rabbit parotid gland.

1. The effects of nicotine on amylase secretion induced by auriculo-temporal nerve stimulation were studied.2. Nicotine caused a transient increase in secretion as well as flow rate of amylase.3. No difference in nicotine action was found between acute sympathetic decentralization of the gland and acute denervation.4. The increase in amylase secretion due to nicotine was not inhibited by phenoxybenzamine, bretylium and chronic denervation, but was prevented by hexamethonium, propranolol and adrenalectomy.5. The increase in flow rate due to nicotine was not inhibited by propranolol, chronic denervation and adrenalectomy, but was prevented by hexamethonium, phenoxybenzamine and bretylium.6. These results show that the action of nicotine in increasing amylase secretion is neither a direct action on the ganglion nor on the nerve terminal of the cervical sympathetic nerve, but is an indirect action of catecholamines released from the adrenal medulla on the post-junctional receptors.7. The study also suggests that the initial acceleration of salivary flow due to nicotine is characterized by a mechanism different from that of amylase secretion.

Adrenal Medulla↗