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Biomedical subjects

V K Kak

Publications and source records attributed to V K Kak.

At least 91 records · Page 5Linked to original sources
PubMed1987

Congenital intramedullary spinal ependymal cyst.

A rare case of congenital intramedullary ependymal cyst of the dorsal cord in a 7-year-old boy associated with kyphoscoliosis and rachidian malformations of the dorsal spine is reported. Myelography suggested an intramedullary lesion. The posterior location and presence of a clear plane of cleavage from the medullary tissue enabled total enucleation of the cyst. Histological differentiation from other similar intradural cysts is discussed and the relevant literature is reviewed. This is the fifth such case reported in the literature.

Child↗
PubMed1987

Intramedullary spinal cysticercosis. Case report and review of literature.

Intramedullary spinal cysticercosis is extremely uncommon; only 29 cases have been reported previously. A case of solitary intramedullary spinal cysticercosis is described. Factors accounting for the rarity of intramedullary cysticerci are reviewed and the probability of reaching a preoperative diagnosis is discussed. This is the fifth such case reported from India.

Adult↗
PubMed1985

Infected intradural hydatid cyst at foramen magnum. A case report.

A case of infected intradural extramedullary hydatid cyst at foramen magnum with recurrent spinomedullary compression is described. The diagnosis was based on operation and histopathology. So far, no case of extraparenchymatous multiple cysts with cervicomedullary compression has been described in literature. Infection within an intact cyst is extremely rare.

Adult↗
PubMed1985

Chronic spinal subdural hematomas. Report of two cases.

Two patients with chronic spinal subdural hematomas are described. Both had a fluctuating clinical course, not previously documented in the literature. Surgical evacuation resulted in almost complete recovery. The pathogenesis of spinal subdural hematoma is discussed and the pertinent literature is reviewed.

Adult↗
PubMed1984

Hemibase syndrome: an unusual presentation of intracranial paraganglioma.

A case of paraganglioma of parasellar origin in a 7-year-old girl is described. She presented with hemibase syndrome with involvement of a majority of the left cranial nerves. Three times in the past 2 years she had recurrent ophthalmoplegia with complete recovery. She was treated surgically and with postoperative radiotherapy. The site of origin of this paraganglioma as well as the clinical presentation are uncommon features in this case.

Child↗
PubMed1982

Meningioma presenting as recurrent transient cerebral ischemia and intracranial hemorrhage.

Intracranial tumors are sometimes known to appear first as apoplexy. This report describes a patient with a meningioma of the falx who experienced acute onset of symptoms due to intracerebral and subarachnoid hemorrhage and following a history of transient ischemic attacks on exertion. The tumor was an angioblastic-meningioma. The probably source of bleeding and the mechanism of transient ischemic episodes provoked by exertion are discussed.

Adult↗
PubMed1981

Adult aqueductal stenosis with Recklinghausen's neurofibromatosis.

A 28-year-old man with von Recklinghausen's neurofibromatosis was first seen with a spastic-ataxic gait and epileptic seizures of recent onset. computerized tomographic scan established the diagnosis of aqueductal stenosis, rarely reported in patients with von Recklinghausen's disease. The value of computerized tomography in the etiological diagnosis of hydrocephalus in von Recklinghausen's disease is emphasized and the pathogenesis of aqueductal narrowing in neurofibromatosis is discussed.

Adult↗
PubMed1980

Cerebral metabolism following brain injury. I. Acid-base and pO2 changes.

Acid-base parameters and oxygen tension were studied in arterial blood and lumbar CSF of 45 subjects in three groups of controls, head injuries, and postoperative cases. CSF in both groups of head injury and postoperative cases showed changes of metabolic acidosis with the arterial blood changes of respiratory alkalosis. Both groups had significant hypoxaemia. CSF acidosis is an indication of cerebral metabolic disturbance. It is proportional to the severity of brain damage, and permits a prognosis for further clinical progress. It is recommended that patients with severe brain lesions should be put on controlled ventilation to relieve them of respiratory overwork and to combat cerebral oedema.

Acid-Base Equilibrium↗
PubMed1980

Cerebral metabolism following brain injury. II. Lactic acid changes.

Lactic acid levels were estimated in cerebrospinal fluid and blood of 25 normal individuals and 50 patients with varying grades of head injury. CSF lactic acid levels showed significant elevation proportionate to the degree of brain injury. Elevation of blood lactic acid was less pronounced. CSF lactic acid levels closely correlated with the prognosis of the case, and an increase above 50 mgm% was not compatible with life.

Brain Injuries↗
PubMed1980

Cerebral metabolism following brain injury, III. Transaminase changes.

Glutamic oxalacetic and glutamic pyruvic transaminase levels in CSF and blood were estimated in 25 normal individuals and 50 patients with varying grades of head injury. CSF GOT, and to a lesser extent GPT, showed significant elevation proportionate to the degree of brain injury. Serum GOT was raised in more severe injuries. CSF GOT and GPT levels showed a significant correlation with the prognosis of the case.

Alanine Transaminase↗