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Biomedical subjects

S Wolf

Publications and source records attributed to S Wolf.

At least 361 records · Page 20Linked to original sources
PubMed1978

Circadian rhythm disorders in manic-depressives.

Seven circular manic-depressives were studied through complete of cycles of mania and depression. In five subjects, there was evidence that a circadian rhythm free-ran fast (p less than 0.005 and p less than 0.002 in two of the subjects), and in five subjects, there was evidence that lithium slowed a circadian rhythm. The palliative benefit of lithium may derive from slowing or delaying an overfast circadian clock to prevent desynchronization. Two subjects whose circadian clocks seemed too slow were lithium nonresponders (p less than 0.05). As circadian clock frequency may be transmitted on an X-chromosome gene and may increase with age, a circadian etiology is consistent with the genetics and age distribution of manic-depressive illness. Affective disturbances could be evolutionary remnants of the photo-periodic seasonal responses in animals.

Adult↗
PubMed1978

Electromyographic biofeedback in the reeducation of facial palsy.

Electromyographic biofeedback techniques have been successfully applied in various neuromuscular disorders. Data on the use of this technique in reeducation of facial paralysis after initial surgical correction are scarce. The authors have developed a treatment program consisting of EMG biofeedback with the use of the Basmajian-Emory myotrainer. Two case reports are presented which show successful results after a three month training period.

Adult↗
PubMed1975

Ballistocardiographic indicators of prognosis in ischemic heart disease.

Bimonthly recordings of ultra-low-frequency acceleration ballistocardiograms overa 2 to 7 year period in 73 patients with ischemic heart disease and 50 matched control subjects within the framework of a clinical follow-up were subjected to measurement of IJ amplitude (reflecting force of contraction), IJ velocity (reflecting contractility), and IJ velocity variation coefficient. These repeated measurements were correlated with 24-hour vanillylmandelic acid (VMA) excretion in the urine, arthythemia incidence, heart rate, and clinical outcome in each subject. Moth-to-month variability of the IJ velocity was highly significantly correlated with 24-hour VMA excretionas well as with the likelihood of subsequent myocardial infartion or sudden death. Data on 24 of the patients with ischemic heart disease were available during a two-year period prior to their death from documented myocardial infarction or presumed fatal arrhythmia without a fresh infarction at autopsy. Their records were compared with those made during the same time period on individually matched control subjects and on patients with documented ischemic heart disease who survived the period of study. The mean force of cardiac contraction (IJ amplitude) was consistently lower in the group destined todie early than in the other two groups during the first 18 months. During the 6 monthsprior to death, however, their tracings show a relatively increased force of cardiac contraction and an increased product of force x pulse rate (referred to as the "drive index"), as well as an increase in the incidence of cardiac arrhythmias. None of these late increases was observed during comparable periods of time among surviving patients orcontrol subjuects.

Adult↗