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Biomedical subjects

S R Lin

Publications and source records attributed to S R Lin.

At least 127 records · Page 7Linked to original sources

Juxtasellar hyperostosis of non-meningiomatous origin.

Seven patients are described who had juxtasellar hyperostosis with visual disturbance secondary to non-meningiomatous lesions. Two had chromophobe adenomas, one craniopharyngioma, one carcinoma of the sphenoid sinus, one a thrombosed aneurysm of the intracavernous portion of the internal carotid artery, one epidermoidoma of the orbit, and one chondroblastoma of the anterior clinoid process. The diagnosis of meningioma was entertained initially on the basis of hyperostosis plus visual impairment. Careful evaluation of hyperostosis is essential for correct diagnosis of meningioma, according to our experience. Suprasellar meningiomas almost invariably produce irregular hyperostosis of the planum sphenoidale, often associated with serration and blistering. Sphenoid meningioma, when it is sclerotic, always shows thickening or expansion of the sphenoid wings. Therefore, in the absence of typical meningiomatous hyperostosis, one can readily differentiate non-meningiomatous hyperostosis from true meningioma.

Adenocarcinoma↗

Treatment of central hemangioma of the maxilla by embolization: report of case.

Some methods of treatment for cavernous hemangiomas are reviewed and briefly discussed. Artificial embolization with barium-impregnated spheres was used by us because any other method of treatment seemed to be inadvisable. Techniques of the surgical procedure were followed as programmed by Lussenhop and others, 12 Longacre and others, 2 and by Hoey and others.9 A 47-month follow-up with no radiographic evidence of residual hemangioma has indicated a satisfactory result in this case.

Adult↗

Cerebral circulation after cardiac arrest. Angiographic and carbon black perfusion studies.

Cardiac arrest of 2-15 minutes was induced in 14 dogs. When circulatory arrest lasted longer than 5 minutes, abnormal cerebral angiographic findings were seen 3-4 hours after resuscitation, consisting of (a) marked prolongation of the arterial phase in both intracranial and extracranial arteries, and (b) faint (poor) visualization of the venous phase. No significant abnormalities were noted in dogs arrested less than 5 minutes, or in the control group. Carbon black perfusion studies showed multiple focal areas of small patchy and diffuse coarse reperfusion defects at the arteriolar-capillary levels in dogs arrested longer than 5 minutes. A combination of vascular and parenchymal changes during ischemia may be the cause of these findings.

Animals↗