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Biomedical subjects

M Takase

Publications and source records attributed to M Takase.

At least 181 records · Page 10Linked to original sources
PubMed1982

Hematocrit and blood content in the rabbit eye.

The red blood cell volume and the apparent plasma volume in the ocular tissues of the rabbit were measured using 51Cr labelled red blood cells and 125I labelled human serum albumin. On this basis, the hematocrit of the circulating blood in the eye and the blood content in the ocular tissues were estimated. In the retinal circulation, the hematocrit was 26, which was 70% of the hematocrit of the cardiac blood. This value was thought to be also applicable in the uveal tissue, although the extravascular leakage of the tracer albumin did not allow an exact determination of the hematocrit of the circulating blood in this tissue. The blood content per 1 gram tissue was calculated from the red blood cell volume and the hematocrit determined. It was about 63 microliter in the iris, 59 microliter in the ciliary body, 198 microliter in the choroid and 38 microliter in the retina. The value obtained in the iris or ciliary body was similar to that in the cardiac muscle or intestine, and the value in the choroid was similar to that in the kidney, lung or liver. The value in the retina was greater than that reported in the brain.

Animals↗
PubMed1981

[Superior Foville syndrome after clipping of basilar bifurcation aneurysm--case report (author's transl)].

The patient was a 48-year-old housewife, who had a sudden onset of severe headache followed by loss of consciousness for a few hours on the day of admission. Initially she showed slight restlessness due to headache, neck stiffness and subhyaloid hemorrhage. Four-vessel study revealed a basilar aneurysm on right retrograde brachial angiography and anterior communicating aneurysm on left carotid angiography. Two weeks after the onset, when she had no neurological deficit except for intermittent appearance of disorientation, both aneurysms were successfully clipped through right pterional approach of Yasargil. The subarachnoid hemorrhage was apparently due to basilar bifurcation aneurysm. Postoperatively, she showed right hemiparesis including her face, aniscocoria (left, 4 mm, oval: right, 1.5 mm, round) and conjugate deviation toward the left. The disturbance of conjugate eye movement and the hemiparesis completely disappeared in 2 and 7 days respectively. The patient was discharged 4 weeks postoperatively with mild left 3rd nerve palsy. At present, one year postoperatively, she is fully engaged in her housewife life without any neurological deficits. A case of superior Foville syndrome combined with Weber syndrome after clipping of basilar bifurcation aneurysm was reported and its anatomicoclinical mechanism was reviewed. The pathogenesis was supposed to be left midbrain ischemic lesion due to circulatory disturbance of P-1 perforators (P-1: proximal posterior cerebral artery); e.g., occlusion on clipping of vasospasm. This P-1 perforator syndrome after aneurysmal clipping has been reported only little. The importance of preservation of these perforators with careful dissection and manipulation under microscopy was emphasized.

Basilar Artery↗
PubMed1981

[A case of hematoma localized to midbrain tegmentum following closed head injury (author's transl)].

UNLABELLED: CASEs of primary brain stem lesion following closed head injury, verified on CT scan, have been increasingly reported recently. However, most of them have supratentorial lesions in addition to brain stem, resulting in a poor outcome. In this paper, a case of localized brain stem hematoma following closed head injury, is reported based on CT findings. CASE: A 26-year-old man slipped down on his back and hit the right occipital area with following loss of consciousness for several minutes. Since then, he continued to have gait disturbance and visited our hospital on the next day. On admission, he presented truncal ataxia, left trochlear nerve palsy, right Horner's syndrome and left hemihypesthesia. CT scan revealed a small hematoma localized to the right midbrain tegmentum at the level of inferior colliculus, well correlating with his clinical presentation. He gradually improved on conservative treatment and returned to his former work one month after the accident. This case suggests that there may be cases of primary brain stem injury with no other intracranial lesion based on CT findings and with a good prognosis. Shear strain is said to be a probable mechanism for explaining brain stem injury, which is usually combined with other parenchymal lesions. In our case, however, more focal factor--nervous and/or vascular compression against the tentorial edge--is suspected for producing this localized midbrain lesion.

Adult↗