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Biomedical subjects

M Nakata

Publications and source records attributed to M Nakata.

At least 361 records · Page 20Linked to original sources
PubMed1981

[A case of intracranial hypoglossal neurinoma (author's transl)].

A case of intracranial hypoglossal neurinoma was reported and the literature was reviewed. A 46-year-old woman with a three-year history of suboccipital pain, dizziness, blurred vision, swallowing disturbance, cysarthria, dysesthesia and weakness of the four extremities, was admitted to our hospital on July 9, 1978. General physical examination on admission were unremarkable. Café au lait spot was not found. Neurological examination revealed nystagmus, ataxic gait and impairment of the finger to nose test on the right side. Atrophy of the tongue with fasciculation on the right side was also noted. Computed tomography demonstrated a round low density area in the posterior fossa which was enhanced with contrast medium. Antero-posterior view of the skull tomography showed enlargement of the right hypoglossal canal. Vertebral angiography and ventriculography disclosed a mass located at the foramen magnum. On July 27, suboccipital craniectomy with C1 laminectomy was performed in prone position. The encapsulated tumor which arouse from the right hypoglossal nerve was totally removed. Histological diagnosis was Antoni A type schwannoma. Postoperative course was uneventful and she was discharged on August 31. Intracranial hypoglossal neurinomas are very rare and our case marks the 17th reported observation of such a lesion. because of their rarity, complicated natural history and variety of associated symptoms and signs, early diagnosis of these lesions is difficult. Among the diagnostic procedures, computed tomography, vertebral angiography and skull tomography were useful for localizing the lesion. Especially skull tomography which showed enlargement of the right hypoglossal canal could offer very important informations. It should be recognized that early diagnosis by means of neurological and radiological maneuvers mentioned above is essential to good prognosis.

Cranial Nerve Neoplasms↗
PubMed1980

Audiotoxicosis in newborn guinea pigs from pregnant ones given sisomicin.

The audiotoxic effect of sisomicin was investigated in guinea pig injected intramuscularly with 6.25, 12.5, 25 and 50 mg/kg of sisomicin sulfate (potential) during weeks 1--5 or 5--9 of pregnancy, and the transplacental effect on their offspring was studied. While, in some dams the higher doses of sisomicin produced a limited effect on the pinna reflex and the outer hair cells of the organ of Corti, no such effects were noted in any of the offspring during the 3-week postnatal observation period.

Animals↗
PubMed1980

Traumatic arteriovenous fistula between the middle meningeal artery and cortical vein.

A 53-year-old man with a traumatic dural arteriovenous fistula between the middle meningeal artery and the adjacent cortical vein is reported. The lesion was absent on the initial angiogram after his head injury and was first recognized nine months later. It enlarged during the succeeding ten months. Symptoms were convulsions and a transient aphasic episode. Surgical repair was successful.

Arteriovenous Fistula↗
PubMed1980

A genetic study of anodontia in X-linked hypohidrotic ectodermal dysplasia.

Dental examinations and tooth measurements were conducted on 16 mothers, 10 fathers, and 23 affected males in 15 families with X-linked hypohidrotic ectodermal dysplasia. Small teeth and congenital missing teeth were sufficiently consistent findings in obligate heterozygotes to suggest that carriers can usually be recognized by clinical criteria.

Anodontia↗
PubMed1978

Clinical, genetic, and histologic features of the trichoonychodental (TOD) syndrome.

A syndrome of scanty, fine, curled hair, thin dysplastic nails, taurodontic molars, hypoplastic-hypomature enamel, dysplasia of dentin, and hypohidrosis segregating as an autosomal dominant trait is described in a Japanese family. The patients received repeated doses of tetracycline during odontogenesis which fortuitously assisted in interpreting the pathogenesis of the dentin defect. The condition was previously described as familial congenital ectodermal dysplasia by Westerholm and as odontogenesis imperfecta by Siirilä and Heikinheimo. It is suggested that the name trichoonychodental (TOD) syndrome is a better descriptive term for the condition.

Chromosome Aberrations↗
PubMed1977

A clinical and histologic observation of enamel hypoplasia in a case of epidermolysis bullosa hereditaria.

The nature of the enamel defects in a patient with epidermolysis bullosa hereditaria dystrophica was observed by light microscopy, microradiography, and scanning electron microscopy (SEM) so that by these means more detailed information could be obtained. The random distribution of the round shape of the hypoplastic defects of enamel with hypomineralization, in which there is a continuity of the incremental lines, suggested the possibility that some ameloblasts maintained hypoactivity of their secretory function during their total life span.

Child↗
PubMed1977

Clinical and histologic observations of opalescent dentin associated with enamel defects.

The rare variant of opalescent dentin associated with enamel defect was found in a 1 1/2-year-old boy. The pulp chambers and root canals of the affected deciduous teeth were very large, with no sign of obliteration. The enamel layer of those teeth was markedly reduced in thickness, and the enamel prisms were not recognized even by scanning electron microscopy. The mantle dentin was abnormal, as were other portions of dentin.

Amelogenesis Imperfecta↗