A study of the relationships between carbon tetrachloride-induced lipid peroxidation and liver damage in rats pretreated with vitamin E.
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Administration, Oral↗
Biomedical subjects
M Comporti
Publications and source records attributed to M Comporti.
Fatty acid composition of the major lipid classes of very low density lipoproteins and of plasma in rats poisoned with carbon tetrachloride.
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Animals↗
Liver glutathione peroxidase activity and CCl4-induced lipid peroxidation in selenium treated rats.
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Aldehydes↗
Studies on in vitro peroxidation of liver lipids in ethanol-treated rats.
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Animals↗
Carbon tetrachloride induced peroxidation of liver lipids in vitamin E pretreated rats.
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Animals↗
Changes in microsomal lipids of rat liver after chronic carbon tetrachloride intoxication.
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Animals↗
Fatty acid composition of mitochondrial and microsomal lipids of rat liver after acute ethanol intoxication.
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Alcoholic Intoxication↗
[Changes in the fatty acid composition of lipids from the liver and hepatic microsomes of rats with carbon tetrachloride poisoning].
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Animals↗
Limitation of myocardial oxygen uptake under acetyl-beta-methylcholine.
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Animals↗
Effect of in vivo and in vitro ethanol administration on liver lipid peroxidation.
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Aldehydes↗
[Effect of some cations on the inactivation by CCl-4 of microsomial glucose-6-phosphatase in vitro].
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Animals↗
[Ascorbic acid content of certain rat organs and urine in experimental alloxan diabetes].
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Animals↗
The direct effect of carbon tetrachloride on subcellular particles.
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Acid Phosphatase↗
Effect of CCl-4 in vitro and in vivo on lipid peroxidation of rat liver homogenates and subcellular fractions.
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Animals↗
[Stimulation of the lipid peroxidation of rat liver homogenates by low concentration of CC1-4 in vitro].
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Animals↗
[Protective action of EDTA on inactivation by CCl-4 in vitro of microsome glucose-6-phosphatase].
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Animals↗
[Inhibition of lipid peroxidation by CCl-4 in vitro and in vivo].
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Animals↗
Regulation of ascorbic acid and of xylulose synthesis in rat-liver extracts. The effect of alloxan-diabetes on the glucuronic acid pathway.
1. The synthesis of l-ascorbic acid and of l-xylulose from d-glucuronolactone, d-glucuronate, l-gulonate and l-gulonolactone has been studied with liver extracts from normal and alloxan-diabetic rats. 2. In diabetic animals the synthesis of ascorbic acid is impaired, and more from glucuronolactone and glucuronate than from gulonate and gulonolactone, whereas the formation of xylulose from gulonate and gulonolactone is enhanced. These changes are reversed by insulin therapy. 3. The activity of the NAD-linked gulonate dehydrogenase is enhanced during diabetes.
Journal Article↗