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Biomedical subjects

L Babiuch

Publications and source records attributed to L Babiuch.

At least 37 records · Page 2Linked to original sources
PubMed1990

[HIV-1 infection in blood donors and blood recipients].

The results of the tests on HIV-1 infection in blood donors in the years 1985-1989 were presented. Out of 3.222.345 serum samples, in 66 donors HIV-1 antibodies were detected. In 1989 the significant raise of HIV-1 carriers, among whom there were 17 intravenous drug users, was noticed. The transmission of HIV-1 infection to 9 blood recipients was proved. Medical examination of 20 infected blood donors revealed in all of them the enlargement of peripheral lymph nodes. In 8 donors candidosis of oral cavity was diagnosed. The undertaken analysis showed the low efficacy of self deferral and pointed out that medical examination before blood donation should be more carefully done.

Acquired Immunodeficiency Syndrome↗
PubMed1989

[Course of acute hepatitis B in alcoholism].

Clinical course of the acute hepatitis B has been analysed in 29 men chronically abusing alcohol. The most frequent source of the infection was surgery and hospitalizations. Clinical course and biochemical findings were similar in this group and in 30 men not drinking alcohol. A sole difference concerned higher GGTP activity in alcoholics. The persistence of HBs antigen was similar in both groups. Chronic alcohol abuse does not seem to have a significant effect on the course and convalescent phase of the acute hepatitis B.

Acute Disease↗
PubMed1989

[Severe alcoholic hepatitis].

A case of severe alcoholic hepatitis was observed in a man aged 30 years. Attention is called to possible diagnostic and therapeutic difficulties. Despite abstinence lasting 9 months the results of biochemical tests failed to return to normal values.

Adult↗
PubMed1989

[Liver damage in alcoholics].

The authors made a liver biopsy in a group of 42 persons chronically abusing alcohol. In the majority of patients the authors found various forms of hepatopathology: fatty degeneration--8 cases (19%), alcoholic hepatitis--10 cases (24%), chronic active hepatitis--11 cases (26%), and hepatocirrhosis--2 cases (5%). The authors found no correlation between the kind and degree of liver damage and the amount of alcohol drunk, which suggests the role of individual hypersensitivity in the development of hepatopathology in alcoholics. In some of the alcoholics liver damage was connected with infection by HBV--5 cases (12%) and the participation of alcohol in the pathogenesis of chronic active hepatitis seems likely but it still remains not verified. The results of the investigation carried out point to the need of histological examination of the liver in alcoholics.

Adult↗
PubMed1988

Prevalence of hepatitis B virus markers among Polish urban alcoholics.

124 consecutive patients (mean age: 41.1 years; range: 20-63 years) attending an outpatient alcoholic clinic were tested for the presence of markers of HBV infection. HBsAg was found in 26 (21%), anti-HBs and/or anti-HBc in the absence of HBsAg in 35 (28%). Altogether, at least one marker was present in 61 (49%), these results being significantly higher than in a matched control population. In 70 cases HBV status was compared with epidemiological data. No relationship was found with past blood transfusions, hospitalization or jaundice. HBV infection was, however, more common when parents of the subjects were alcoholics, which points to a possible role of family spreading. Furthermore, chronic infection with HBV was related to high alcohol intake.

Adult↗
PubMed1988

Prevalence of hepatitis B virus markers among Polish alcoholics.

102 consecutive alcoholics attending an outpatient alcoholic clinic were tested for hepatitis B virus (HBV) markers in serum. HBsAg was found in 21, anti-HBs in 22 and anti-HBc in 42; at least 1 marker was present in 49. These results were significantly higher than in a matched control population. Out of 21 HBsAg positive cases 7 had markers of ongoing viral replication as judged by the presence of HBeAg and HBV DNA.

Adult↗