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Biomedical subjects

J Jacquy

Publications and source records attributed to J Jacquy.

101 records · Page 6Linked to original sources
PubMedDate not supplied

[The value of cognitive evoked potentials in the diagnosis of senile dementia and dementia of Parkinson disease].

P300 latency is often delayed in dementia but some authors have questioned its interest in clinical practice, because of the relatively low specificity and sensibility of the method. Therefore, we studied numerous parameters (latency, duration, amplitude and surface) of N100, N200 and P300 waves in patients with senile dementia, patients suffering from Parkinson's disease with dementia and non demented elderly patients. Using discriminant analysis, more than 90% of the patients were correctly classified. This method could thus be contributive in clinical practice.

Aged↗
PubMedDate not supplied

[Acute maternal anterior poliomyelitis in a non-endemic zone].

The authors report the case of a 26 year old woman with acute anterior poliomyelitis contracted during the vaccination of her baby. Despite having been herself vaccinated in infancy she was not protected against the poliovirus. The clinical interest of this uncommon case is a severe paralytic state with definitive paraplegia. The authors suggest serologic testing of patients born before 1967 especially if they are at risk of encountering the virus.

Adult↗
PubMedDate not supplied

[Endocrinological and neurophysiological modifications in a case of atropinic delusional state].

In this case report, we observed a delirium probably owing to toxic effects of homatropine eye drops. We reproduced this syndrome and we reversed it with physostigmine salicylate as an antidote. We observed a decrease of cortisol at 9.00 a.m. and impairment of regional cerebral blood flow with increase in the previous right side. The unusual susceptibility of this patient to the anticholinergic effect of homatropine is perhaps related to a preexisting mild cognitive defect possibly related to a cholinergic neurotransmitter deficit.

Aged↗
PubMedDate not supplied

[Quantitative rheoencephalographu in normal and pathological ageing (author's transl)].

A method of quantitative rheoencephalography estimating the rapid cerebral blood flow, defined by the clearance of Xe 133 is applied to normal, hemiplegic and demented subjects. In the normal child the flow is twice that of the elderly. In adults a decrease of 13% is observed in the fifties. The flow is decreased in both hemispheres of the hemiplegics. The cerebral flow is decreased in the two clinical groups of demented patients (vascular and non-vascular); different values of flow are obtained in these two groups.

Adolescent↗
PubMedDate not supplied

[Cerebral blood flow and depression].

Regional cerebral blood flow is an index of cerebral metabolism. We have studied with the Xenon 113 inhalation method, regional cerebral blood flow (CBF) in patients with various types of depression and in remission. A left frontal hypervascularisation and a right posterior hypovascularisation have been found in primary major depression, but not in minor depression and bipolar depression in remission. These results confirm and complete results from other authors who have studied the physiopathology of depression with other technics.

Adult↗
PubMedDate not supplied

[A case of post-hepatitic Guillain-Barré-Strohl syndrome with extension to all pairs of cranial nerves and late papilledema. Review of recent literature (author's transl)].

The authors report the case of a 17 year-old female patient who presented acute ascending polyneuritis of the Landry-Guillain-Barré type extending to all cranial nerve pairs during the abatement of hepatitis of unspecified etiology. The patient remained in a state of coma vigil for five days prior to gradual but full recovery over a six-month period. The patient was given respiratory assistance for 55 days, together with corticosteroid therapy and above all plasmapheresis. After five weeks of treatment a transient late papilledema was noted for about six weeks. The authors discuss the diagnostic criteria of the Guillain-Barré-Strohl syndrome, its etiology, the possibility of central nerve involvement and late papilledema. Treatment with corticosteroids and plasmapheresis is also discussed.

Adolescent↗