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Biomedical subjects

J J Baudon

Publications and source records attributed to J J Baudon.

53 records · Page 3Linked to original sources
PubMed1977

[Acrodermatitis enteropathica; zinc sulfate therapy].

We present the case of two siblings with acrodermatitis enteropathica. Zinc levels were low. Oral administration of 100 mg of zinc sulfate allowed total regression of all signs. When zinc therapy was interrupted, the manifestations reappeared and disappeared again when treatment was resumed. A defect of genetic origin in digestive zinc absorption is suggested. Mohnahan's advocated zinc treatment of acrodermatitis enteropathica in 1973; it is a non-toxic treatment, which is always active and be considered as a diagnostic test.

Acrodermatitis↗
PubMed1976

[Acute villous atrophy due to intolerance to soy bean protein].

In infancy, intolerance to soybean protein may be associated with cow's milk protein intolerance. In the reported case, the intolerance to soybean had clinical expression. In addition, this intolerance was expressed on the histological level with severe villous atrophy which subsided 18 days after the suppression of soybean from the diet. These facts invite to administer soybean proteins with caution in patients with cow's milk intolerance.

Atrophy↗
PubMed1975

[Digestive intolerance to cow's milk proteins in infants. Clinical study of 42 cases].

The criteria chosen for the diagnosis of gastro-intestinal intolerance to cow milk proteins were the following: occurrence of shock or anaphylaxis, reccurrence of gastro-intestinal symptoms as a consequence of repeated trials of reintroduction of cow milk proteins into the diet. Under these conditions the transient forms, including the secondary forms, of intolerance to cow milk, were eliminated. This diagnostic attitude may explain the early appearance of gastro-intestinal disturbances, the higher frequency in infancy than in childhood of the severe forms, characterized by shock, subacute malabsorption, exsudative enteropathy, bloody diarrhea, the authors are describing in their study report.

Animals↗
PubMed1975

[Digestive intolerance to cow's milk proteins in infants. Biological and histological study].

Biological and histological studies were performed in 42 cases of gastro-intestinal intolerance to cow's milk proteins. Hypoproteinemia was present in 2/3 of the cases, anemia in 3/4. Malabsorption syndrome (steatorrhea, xylose) was overt in less than 1 patient out of 2. Serum level of vitamin A was more frequently depressed than that of vitamin E and folinic acid. Intestinal biopsy, performed in 32 children, showed consistent abnormalities, a partial atrophy of the villi being most often seen (26 cases). Eosinophilia was present in 50% of the cases. A titer of anti-milk agglutinins of 1/64 or above was always found if the test was performed repeatedly when the diet of the children contained milk. Lymphocyte proliferation in culture, induced by milk proteins, was positive in 70% of the cases. None the less, no biological or histological findings were found to be specific for gastro-intestinal intolerance to milk proteins.

Agglutinins↗
PubMedDate not supplied

Lymphoblastic stimulation test with food proteins in digestive intolerance to cow's milk and in infant diarrheas.

The lymphoblastic stimulation test (LST) with cow's milk proteins was performed in 114 infants. In 42 infants, digestive intolerance to cow's milk proteins (CMI) was suspected; withdrawal-rechallenge test confirmed intolerance in 34, and disproved it in the other eight patients. Of the other patients, 17 had acute gastroenteritis, 11 had postgastroenteritis subacute diarrhea, 12 had gluten intolerance, 14 had intractable diarrhea, and 18 had no digestive disorders. Of the 34 infants with CMI, 27 (79%) had a positive LST to one or more cow's milk proteins. Of the 34 positive LST patients, 12 also had soya intolerance; nine of these 12 infants (75%) had positive LST to soya. Of the eight infants who had a negative cow's milk rechallenge test, five (62%) had a positive LST. In the other groups, results were also positive in 12-27% of those having diarrhea of infectious origin or gluten intolerance, and in none of the infants without digestive disorders. Of the 14 cases of severe intractable diarrhea, 12 (86%) were also LST-positive, but CMI could not be excluded. LST was positive, particularly in diarrheas of neonatal origin. Lymphoblastic stimulation was induced more frequently by casein than by beta lactoglobulin, and least frequently by alpha lactalbumin. In conclusion, LST is frequently positive in CMI, but is not sufficiently specific to be a reliable diagnostic examination.

Animals↗
PubMedDate not supplied

[Steroid-responsive nephrotic syndrome in infants: 2 familial case reports ].

Two cases of nephrotic syndrome discovered respectively at the ages of 3 and 7 weeks in 2 siblings are reported. Nephrosis was of the optically normal glomeruli-type in both cases. In spite of the very young age of the patients a good result of steroid-therapy was obtained after 6 and 8 weeks of treatment, respectively. Both children were not HLA-identical but had in common the A2-B12-DR4 haplotype.

Female↗