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Biomedical subjects

H Shoji

Publications and source records attributed to H Shoji.

At least 199 records · Page 11Linked to original sources
PubMed1981

Expression of Forssman antigen of avian lymphoblastoid cell lines transformed by Marek's disease virus or avian leukosis virus.

The expression of Forssman-type heterophile antigen on Marek's disease (MD) virus (MDV)-transformed cell lines, MDCC-MSBI 1, -HP1, -RP1 and -BP1, and avian leukosis virus (ALV)-transformed cell lines, LSCC-1104B1 and -1104X5 was investigated by membrane immunofluorescence and complement-dependent antibody cytotoxicity tests. Forssman antigen was detected on a high percentage of the cells in two ALV-transformed cell lines and on a smaller percentage of splenic lymphocytes from normal chicken. Of the MDV-transformed cell lines tested only the RP1 and BP1 cell lines, derived from transplantable MD tumours, expressed Forssman antigen, while the MSB1 and HP1 cell lines, derived from MD lymphomas, did not. Forssman antigen appears to be unrelated to MD tumour-associated surface antigen (MATSA).

Animals↗
PubMed1981

Heel deformity in hereditary spastic paraplegia.

The association of pes cavus deformity and neurological disorder is well known. Varus deformity of the heel is often associated with, and may even precede the development of pes cavus. Clinical and radiographic examinations of the feet of members of three kindreds of hereditary spastic paraplegia, a rare neurologic disorder, suggested that the autosomal dominant form manifests a significantly higher incidence and degree of heel varus deformity than the autosomal recessive form of the disease. Therefore, heel varus deformity may be a sign suggestive of autosomal dominant inheritance of hereditary spastic paraplegia.

Adult↗
PubMed1980

A case of facial diplegi following herpes zoster ophthalmicus.

A 71-year-old man developed a facial diplegia following right ophthalmic zoster. Neurological examination also revealed loss of reflexes in the lower limbs with increase of CSF protein. Unusual cases of the Ramsay Hunt syndrome and Guillain-Barré syndrome seen with herpes zoster were reviewed. These cases including our case may suggest that the bilateral involvement or its wider spread throughtout nervous tissue can rarely occur in the Ramsay Hunt syndrome and its subgroups.

Aged↗
PubMed1980

Distribution of Forssman antigen in chickens.

The distribution of Forssman antigen in various tissues of chickens was studied by the indirect and direct immunofluorescence (IF) tests using rabbit immune serum against purified Forssman glycosphingolipid (GSL) [GalNAc (alpha, 1-3)GalNAc(beta, 1-3)Gal(alpha, 1-4)Gal(beta, 1-4)Glc-ceramide]. A general finding in all tissue sections was the presence of Forssman antigen in vascular endothelium and perivascular connective tissues. Erythrocytes gave a Forssman positive reaction, but the intensity of fluorescence was slight. Forssman antigen was found in about 20% of all peripheral leukocytes. Fractionation of lymphocytes from peripheral blood into T and B lymphocytes showed that about 30% of the B lymphocytes gave a positive reaction, while only 2-3% of the T lymphocytes gave a positive reaction. About 40% of bone marrow cells expressed Forssman antigens. On the other hand, about 40-50% of adherent cells in vitro cultures from spleen were Forssman-positive. The antigen was also abundant in hematopoietic organs, such as spleen, thymus and the bursa of Fabricius. Positive reactions in the digestive tract, ovary, testis and skin varied from portion to portion of the tissues. In brain and spinal cord, only the vascular endothelium gave a positive reaction. In peripheral nerves, only the connective tissue stroma around the nerve fibers gave a positive reaction. In other tissues, the distribution of the antigen was generally in accord with the results of Tanaka and Leduc (1956). These positive reactions in the IF test were specifically inhibited by addition of purified Forssman GSL. The distributions of Forssman antigen in tissues were found to be the same in chickens of various ages.

Animals↗
PubMed1979

Partial Klüver-Bucy Syndrome following probably herpes simplex encephalitis.

An autopsy case of probable herpes simplex encephalitis (HSE) showed the partial Klüver-Bucy syndrome from 1 to 7 months after the onset, which consists of strong oral tendencies, emotional changes and possible hypersexuality. Several cases of the syndrome in definite or probable HSE were reviewed in this paper. In these cases including our case, the partial or complete Klüver-Bucy syndrome has appeared from some interval (2 weeks--1 month) after the acute phase, so that the syndrome should be noticed in survived or prolonged cases of HSE, as significant clinical symptoms suggesting bilateral involvement of the temporal lobes and limbic areas.

Adult↗