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Biomedical subjects

G Strohmeyer

Publications and source records attributed to G Strohmeyer.

At least 235 records · Page 13Linked to original sources
PubMed1977

[Serum level of 25 hydroxy vitamin D3 in patients with gastric and duodenal ulcers and following billroth II gastric resection].

Serum 25-hydroxyvitamin D3 levels were determined in patients with gastric ulcer, duodenal ulcer, and subtotal gastrectomy (Billroth II); 35 healthy persons served as controls. 25-hydroxyvitamin D3 levels were correlated with the signs of osteopenia determined radiologically. 25-hydroxyvitamin D3 serum level was not different from normal in subjects with gastric or duodenal ulcer. Those who underwent gastric surgery had significantly lower values. Within this group those with osteopenia had lower values than those without.

Duodenal Ulcer↗
PubMed1977

Serum 25 -- hydroxyvitamin D3 levels in patients with liver disease.

Serum 25-hydroxyvitamin D3 levels were determined in chronic hepatic diseases by a radioreceptor assay and correlated with serum albumin, calcium and anorganic phosphate, 25-hydroxyvitamin D3 serum levels were significantly lower inall chronic hepatic diseses compared to normals. The low levels are correlated with the degree of parenchymal damage, not with the etiology of hepatic disease. In alcoholic liver disease thus 25=hydroxyvitamin D3 levels are significantly lower when cirrhosis is present than in mere fatty liver. Anorganic phosphate and calcium were close to the lower range of normal and significantly lower than in the control group studied.

Chronic Disease↗
PubMed1977

25-hydroxycholecalciferol serum levels in patients with Crohn's disease.

In 37 patients with Crohn's disease the 25-hydroxycholecalciferol (25-HCC) serum level, serum concentration of calcium and inorganic phosphate, and the enzyme activity of alkaline phosphatase were measured. Furthermore the activity index of Crohn's disease was determined in every patient. There was no statistically significant difference of 25-HCC serum levels in these patients compared to a healthy control group. Correspondingly most patients showed normal alkaline phosphatase enzyme activity and normal serum concentration of calcium and inorganic phosphate. No correlation between 25-HCC concentration and site of inflammation or activity index was found.

Alkaline Phosphatase↗
PubMed1976

[Internal aspects of diverticulosis (author's transl)].

Diverticular disease of the colon is, apart from the irritable colon syndrome, by now probably the commonest disorder of the large intestine. The incidence of the disease seems to be increasing, which might be due to fundamental dietary changes during the last 60-80 years. It has been suggested that the condition may be a dietary deficiency disorder caused by an inadequate intake of fiber in the diet. Undiagnosed diverticulosis can suddenly progress to painful diverticular disease or diverticulitis with no warning. Complications are sometimes life-threatening, and they demand immediately surgical intervention. The optimal medical and surgical management of diverticular disease and diverticulitis is not well defined as no controlled clinical trials are available.

Colitis, Ulcerative↗
PubMed1976

[Diabetes mellitus in idiopathic haemochromatosis (author's transl)].

In 28 out of 40 patients with idiopathic haemochromatosis manifest diabetes mellitus could be demonstrated 19 patients required insulin. Treatment of diabetes with or without insulin was problem-free. In only two patients there was an insulin resistance which required high doses of insulin some of the time. There was a family history of diabetes in eleven patients. Minimal diabetic retinopathy in two patients was the only typical complication specific to diabetes. Severe forms of microangiopathy are seldom seen in haemochromatosis diabetes. This form of diabetes is probably mainly of genetic origin. Liver cirrhosis and fibrosis and possibly pancreatic siderosis are additional factors to be considered. A sufficiently long and intensive venesection treatment leads to clinical improvement in the diabetes in only a small fraction of the haemochromatosis patients.

Adult↗
PubMed1976

[Effect of phenylalanine-vasopressin on the lower esophageal sphincter. Possible implications in the treatment of bleeding esophageal varices].

Vasopressin and its analogs are used inthe treatment of bleeding esophageal varices. Since gastrointestinal reflux may have a deleterious effect on variceal hemorrhage, the effect of 2,3-phenylalanine-8-lysine-vasopressin upon the lower esophageal sphincter (LES) was studies by rapid pull-through manometry in 24 persons. PLV infusion up to a dosis of 2.7 mU/kg/h raised LES pressure from 15.1 +/- 1.3 (SEM) to 17.9 +/- 2.0 mm Hg. Higher doses lowered LES pressure progressively to 12.1 +/- 0.7 mmHg at 54 mU/kg/h. The serum gastrin level did neither correlate with basal LES pressure not with LES pressure changes during PLV infusion. Therefore, PLV does not appear to act indirectly through serum gastrin. Because of the danger of systemic side effects and of the undesirable in LES pressure with the usual high doses of vasoactive substances, a continuous infusion of lower doses of vasopressin analogs appears to be advantageous.

Adult↗