[Plasmatic enzymes (GOT, GPT, ALD, LDH, CPK) in experimental myocardial infarct in dogs with inhibition of thyroid function].
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Animals↗
Biomedical subjects
G Lombardo
Publications and source records attributed to G Lombardo.
[Electrocardiograms and plasmatic enzymes (SGOT, SGPT, ALD, LDH, CPK) in experimental myocardial infarct in the dog].
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Animals↗
[Blood lipids, lipoproteins and blood cholesterol fractions in experimental myocardial infarct in the dog with inhibited thyroid function].
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Animals↗
[Electrocardiograms in experimental infarct in dogs with inhibited thyroid function].
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Animals↗
[Electrocardiography in experimental myocardial infarct in dogs with induced hyperthyroidism].
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Animals↗
[Plasmatic enzymes (GOT, GPT, ALD, LDH, CPK) in experimental myocardial infarct in rats with induced hyperpthyroidism].
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Alanine Transaminase↗
[Blood lipids, lipoproteins and cholesterinehemic fractions in experimental myocardial infarct in rats with induced hyperthyroidism].
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Animals↗
[Serologic research on diffusion of leptospirosis among cattle].
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Animals↗
[Serologic research on diffusion of leptospirosis among horses].
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Animals↗
[Behavior of erythrocytic leucine aminopeptidase in the dog subjected to splenectomy].
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Animals↗
[Behavior of hematic GSH and of the GSH stability test in the dog subjected to splenectomy].
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Animals↗
[Behavior of pyruvicokinase and lactic dehydrogenase of the erythrocytes in the dog subjected to splenectomy].
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Animals↗
[Behavior of G-6-PDH in erythrocytes of dogs exposed to splenectomy].
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Animals↗
[Behavior of erythrocytic adenylic derivatives (ATP, ADP, AMP) in dogs exposed to splenectomy].
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Adenine Nucleotides↗
[Activity of fructose-1, 6-phosphate aldolase in the erythrocytes in the splenectomized dog].
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Aldehyde-Lyases↗
[Activity of acid phosphatase in the erythrocytes of the splenectomised dog].
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Acid Phosphatase↗
[Hyperthyroidism in childhood].
Pathogenesis of Graves' disease, the most frequent cause of infantile hyperthyroidism, is not definitely clarified. The role of thyroid stimulating immunoglobulins as the cause of hyperthyroidism, goiter and exophthalmos in Graves' disease is analytically considered. Genetic factors are also discussed. The Authors report some personal clinical observations emphasizing the polymorphic clinical course of the infantile Graves' disease. The diagnostic procedures and the therapeutical management are successively analyzed. Pathogenesis and clinical picture of neonatal thyrotoxicosis and Plummer's disease, two not frequent conditions, are finally examined.
Adenoma↗
[Changes in the lower dental arch during growth].
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Adolescent↗