Biomedical subjects
G J Gilbert
Publications and source records attributed to G J Gilbert.
Pseudohemiparetic parkinsonism.
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Ibuprofen-induced meningitis in an elderly patient with systemic lupus erythematosus.
The use of ibuprofen by patients with systemic lupus erythematosus poses a major threat of aseptic meningitis. Although previously reported cases have all been in young adults, our case of ibuprofen-induced meningitis occurred in a 73-year-old woman. This report greatly expands the age population that may suffer meningitis from this analgesic, now available over the counter, and emphasizes the importance of recognizing this causative relationship in patients with headache.
Common carotid occlusion.
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Acute ammonia intoxication 37 years after ureterosigmoidostomy.
Ureterosigmoidostomy causes hyperammonemia, which can, especially in the presence of hepatic disease, lead to metabolic encephalopathy and coma. In the case reported here, acute hyperammonemic encephalopathy developed 37 years after the operation, and responded promptly to oral neomycin therapy, with clinical and electroencephalographic resolution. Magnetic resonance imaging of the brain, not previously reported in this disorder, showed symmetric lesions of the white matter.
Churg-Strauss syndrome: determining adequacy of corticosteroid dosage by the eosinophil count.
Prednisone therapy induced prompt clinical remission in a 38-year-old woman with hypereosinophilic vasculitis. Smoldering vasculitis accounts for the 38% fatality rate in treated patients with the Churg-Strauss syndrome. Complete suppression of the peripheral hypereosinophilia will reflect the efficacy of therapy more accurately than clinical response.
Turtle headaches and Parkinson's disease.
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An occurrence of complicated migraine during propranolol therapy.
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CSF eosinophilia following myelography.
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Hemorrhagic complications of cerebral arteritis.
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Cardiopulmonary resuscitation.
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Evidence of viral cause in granulomatous angiitis.
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Familial spasmodic torticollis.
Presented are four cases of familial spasmodic torticollis, comprising members of three families. The age of onset tended to be family-specific, and no afflicted family member had evidence of more widespread disease (dystonia musculorum deformans). Familial incidence supports the conclusion that spasmodic torticollis is organic in origin. Familial spasmodic torticollis occurs more often than has been generally recognized.
Hemorrhagic femoral neuropathy: report of a case due to anticoagulant.
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