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F X Carton

Publications and source records attributed to F X Carton.

30 records · Page 2Linked to original sources
PubMedDate not supplied

[Duhring's and coeliac's diseases (author's transl)].

An atrophy of villi without digestive troubles is found in a 3 1/2 years old boy with a typical Duhring's disease. This observation proves the interest to research an enteropathy in case of dermatitis herpetiformis, and on the other hand the gluten free diet's efficacity on the cutaneous lesions. The authors consider cutaneous lesions's pathogeny and the antigenic relations between these two diseases.

Antibodies↗
PubMedDate not supplied

[Economic aspects of the treatment of leg ulcers].

The authors have compared the healing time and the cost of leg ulcers of venous origin according to whether they were treated on an out-patient basis or by admission to hospital. The healing times were extremely similar, but the cost of cure by hospital admission was twenty times as great. Although one cannot claim to cure all leg ulcers on an out-patient basis, it is clear that significant public economies could be achieved by avoiding or shortening some costly hospital admissions.

Adult↗
PubMedDate not supplied

[Lichen amyloidosus].

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Amyloidosis↗
PubMedDate not supplied

[Duhring and pemphigus].

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Dermatitis Herpetiformis↗
PubMedDate not supplied

[Vasculitis in children's dermatomyositis (author's transl)].

Vascular lesions have been discovered by a number of authors in children's dermatomyositis, when they are not ordinary found in adults. Affecting capillaries, arterioles and veinlets, vasculitis appears as an hyperplasia of intima, then as an endothelial necrosis and formation of thrombi. Usually seen with light microscope, these lesions are sometimes only revealed by electron microscope. These vascular lesions are observed in the muscle and mainly in the perimysial interstitial tissue, in the skin, and in the various visceral localisations of the disease: pulmonary, ocular, renal, nervous and mainly digestives. The ulcerations of the digestive tube are due to little infarcts secondary to submucous arteriolar occlusions. The presence of necrotizing microangiopathy in the muscle, in the skin and in the viscera, and the observation by an author of macroglobulin, gammaglobulin and C3 deposits in the vascular walls allows to issue an aetiopathogenic hypothesis. The vascular lesions could be due, at least partly, to immune complex deposits, explained by an allergic mechanism of type III of Gell and Coombs. This process could be released by a viral factor which has to be determined. This pathogeny could bring children's dermatomyositis nearer to the other circulating immune complexe collagenosis: S. L. E., periarteritis nodosa with which many transition form exist, and could separate it to the adults dermatomyositis, which would rather be a disease due to an exacerbation of the delayed hypersensitivity type IV of Gell and Coombs.

Child↗