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Biomedical subjects

D Storck

Publications and source records attributed to D Storck.

At least 55 records · Page 3Linked to original sources
PubMed1976

[Severe hemolytic jaundice and Wilson's disease].

The onset of spontaneous hemolytic jaundice in a young subject should lead to the search for Wilson's disease when clinical examination reveals cirrhosis. This hemolysis may evolve in the form of severe jaundice to a stage where the cirrhosis remains usually latent or well tolerated. The intervention of a toxic, allergic of infective factor liable to produce a hepatic lesion which frees a dose of copper sufficient to trigger off hemolysis, is discussed. The mechanism of the latter, that of the coagulation disorders observed, liver cell failure and widespread intravascular coagulation, are analysed in this paper and compared with data in the literature. The dramatic character of the case indicates that it is necessary to treat as a routine with penicillamine all homozygous forms of Wilson's disease.

Adult↗
PubMed1976

[Endomyocardial biopsy by venous route. Our initial results].

The authors report their experience of right ventricular endomyocardial biopsy by the technic of Caves and Schulz. The instrument which consists of a spiral metallic catheter covered with synthetic material, includes at one end a hand lever and at the other end a biopsy forceps 3 mm in diameter. It is introduced through the skin into the right internal jugular vein then pushed under direct vision with an image intensifier upto the tip of the right ventricle. The angle of the distal part of the bioptome may be freely modified before use. Passage across the tricuspid orifice from the right atrium is usually easy. 30 good quality samples were thus taken from 10 patients with various forms of heart disease. There were no complications. The simplicity, rapidity and benign nature of this technic are emphasised together with its use in the diagnosis of myocardiopathies.

Biopsy↗
PubMed1975

[Trypsin-induced polyseritis: action of di-iso-propyl-fluorophosphate; evolution of the proteas activity of effusion; comparison with elastase].

1. Whereas the injections of trypsin in the peritoneum of the rat cause every time a polyseritis (ascites and pleuritis) the trypsin inactivated by di-iso-propyl-fluoro-phosphate remains without effect; this fact proves therefore that the proteasic properties of the trypsin are responsible of this exsudative effect on peritoneum and pleura. 2. The intra-peritoneal injections of elastase cause also a polyseritis in the rat, but it is complicated by shock. 3. The dosage of the proteasic activity in the effusions caused by the trypsin and the elastase, shows a progressive reduction of this activity. At the beginning there is more protease in the ascites than in pleural serositis and the volume of the ascites is greater; later, the findings about the proteases are opposite and the protease content becomes greater in the pleural effusion. This evolution is a new proof of the transdiaphragmatic propagation of the proteasic polyseritis.

Animals↗