[Sheet-type angioendothelioma of the face and scalp].
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Facial Neoplasms↗
Biomedical subjects
C Beylot
Publications and source records attributed to C Beylot.
[Phagedenic pyodermitis of the buttocks and of the genital organs in an hypogammaglobulinemic in a 6-week-old infant].
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Agammaglobulinemia↗
[Juvenile xanthogranuloma. Apropos of 6 cases].
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Female↗
[Lichen planus eroding the folds of the skin].
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Humans↗
[Hamartoma caused by hyperplasia of the arrectores pilorum in a 1-month-old infant].
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Female↗
[Ehlers-Danlos syndrome with whip marks].
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Child↗
[Spitz's melanoma (multiple agminated form). 2 cases].
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Child↗
[Postvaccinal erythemato-purpuric acrodermatitis].
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Acrodermatitis↗
[Polymorphous eruption (lichenoid, papulo-necrotic and dermo-hypodermic) with important cervical adenopathy and synovial reaction].
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Adolescent↗
[Chronic cutaneous lesions as portal of entry of tetanus. Apropos of 37 patients, including 2 cases of congenital epidermolysis bullosa].
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Callosities↗
[Aspect of pseudoxanthoma elasticum accompanied by a subacute inflammatory syndrome].
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Fever↗
[Clinical trial of synalar ointment].
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Adolescent↗
[Incomplete form of Toraine, Solente, Gole pachydermoperiostosis associated with articular hyperlaxity in a 7-year-old girl].
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Bone Diseases↗
[Fetal ichthyosis in 2 children of the same family].
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Female↗
[Clinical trials of Betneval, a new dermo-steroid].
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Adolescent↗
[Clinical trials of Hexomilone ointment].
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Adult↗
[Lymphoproliferative syndrome with granular lymphocytes of CD8+ phenotype: a clonal pathology with a chronic course].
The syndrome of CD8 hyperlymphocytosis with neutropenia is a heterogeneous disorder ranging from reactive benign state to neoplastic pathology. The prognosis for LGL (Large Granular Lymphocyte) leukemia depends likely on its phenotype:-NK phenotype, extremely poor prognosis and rapidly fatal-T phenotype (CD8+), chronic disease with slow progression. Here, we report four cases of CD8+ hyperlymphocytosis with neutropenia, which are CD2+/-, CD3+, CD4-, CD8+, CD16-, CD56+/-, CD57+ phenotype. These lymphocytic proliferations were associated with clonal rearrangement of T-cell receptor b gene. In two cases, characteristic blood hyperlymphocytosis appeared only after splenectomy, but retrospective bone marrow analysis showed that the CD8+, CD57+ lymphocyte proliferation previously existed. These lymphocytes had a low natural killer activity against K562 cell line. HTLV1 proviral sequence was not integrated in leukemic cell DNA. This monoclonal pathology has a chronic clinical course, with a thirteen year evolution in one case. Splenectomy did not correct neutropenia but allowed the control of hemolytic anemia and auto-immune thrombocytopenia in one case.
Adult↗