Biomedical subjects
B W Haynes
Publications and source records attributed to B W Haynes.
Massive hemorrhage for diverticulosis of the colon: guidelines for therapy based on bleeding patterns observed in fifty cases.
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The Evans formula revisited.
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Mafenide acetate in burn treatment.
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Current problems in burns. A perspective.
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Burn therapy--a current appraisal.
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Today's challenge in burn therapy.
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Early excision and grafting in third degree burns.
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Sulfamylon therapy in severe burns: two hundred forty-six cases compared to previous experience.
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Early fluid treatment of severe burns: hemodynamic alterations and the use of dextran and blood.
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Burn coma: a syndrome associated with severe burn wound infection.
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Sixth National Burn Seminar. Electrical burns.
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Probit analysis of burn mortality in 1,831 patients: comparison with other large series.
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Hospital isolation system for preventing cross-contamination by staphylococcal and pseudomonas organisms in burn wounds.
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Factors significantly affecting mortality in the burned patient.
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Magnesium metabolism in surgical patients; exploratory observations.
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Infections in burned patients.
Prevention and treatment of burn wound infection requires knowledge of the epidemiology of such infections. Prevention of infection rests on removal of reservoirs or sources of microorganisms from the burn patient's environment and interruption of transfer of microorganisms to the surface of the wound. When prevention fails and burn wound infection develops, successful therapy may depend on an understanding of the epidemiology of the burn wound during therapy. Contrary to the oft stated concept that antibiotics penetrate the avascular burn wound poorly, our study demonstrated that gentamicin and tobramycin achieved therapeutic concentrations in burn wound tissue. As in other types of infections, susceptible microorganisms were eradicated and resistant microorganisms persisted. Of most importance was the observation that resistant microorganisms may repopulate the wound within four days of starting therapy. It would appear that failure of therapy is not due to failure of antibiotics to penetrate the burn wound but rather to rapid development of superinfection during therapy.