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Biomedical subjects

A Prasad

Publications and source records attributed to A Prasad.

At least 181 records · Page 10Linked to original sources
PubMed1981

In vitro corrosion and tarnish characterization of typical dental gold compositions.

Analysis of the anodic polarization and alternate immersion test response of 16 precious metal alloys with compositions similar to those of commercial dental golds indicate that alloys containing high (Ag + Cu) contents are likely to be more corrosion and tarnish prone. It also appears that the alloys containing high precious metals contents (Au, Pt, and Pd) are characterized by immunity or anodic film protection over a range of anodic overpotentials, indicating superior corrosion resistance.

Copper↗
PubMed1981

In vitro corrosion and tarnish analysis of the Ag-Pd binary system.

Binary compositions over the entire range of compositions of the Ag-Pd system are characterized by anodic polarization in Ringer's solution and alternate immersion in 0.5% Na2S solution. The results show that a wide range of protection and passivation tendencies are introduced with Pd alloying of Ag.

Corrosion↗
PubMed1981

Biochemical changes in the cervical mucus of buffalo after induction of oestrus with prostaglandin F-2 alpha and cloprostenol.

Prostaglandin F-2 alpha and cloprostenol were given as an i.m. injection on Days 9 or 15 of the cycle. There was a significant decline (P less than 0.01) in the concentration of protein and activities of acid and alkaline phosphatase and peroxidase in cervical mucus after treatments, and the change was more marked in animals that responded by becoming oestrous within 4 days of treatment. Values in control animals remained steady or increased.

Alkaline Phosphatase↗
PubMed1980

Effect of prostaglandin F(2alpha)- thamsalt and Estrumate (ICI 80996) on plasma progesterone levels in Indian water buffaloes (Bubalus bubalis ).

Thirty six buffalo showing normal reproductive cyclas were given a single intramuscular injection of prostaglandin F(2alpha)- thamsalt (PGF(2alpha)) and Estrumate on day 9 or 15 of the estrous cycle in order to induce estrus. In PGF(2alpha) treated animals, plasma progesterone dropped from 2.99+/-0.29 to 0.10 +/- 0.02 ng/ml and from 5.53 +/- 0.53 to 0.09 +/- 0.03 ng/ml (mean +/- SEM) within 72 hours of treatment, on day 9 and 15, respectively. Similarly, the treatment with Estrumate on day 9 or 15 of the cycle reduced the level of proges--terone from 3.02 +/- 0.03 to 0.14 +/- 0.03 ng/ml and from 4.26 +/- 0.47 to 0.21 +/- 0.03 ng/ml, respectively, 72 hours aPter each treatment. It was inferred that both drugs induce estrus in buffalo without any observed residual effect.

Journal Article↗
PubMed1977

Serum UDP-galactose: glycoprotein galactosyltransferase in diabetics with microangiopathy.

1. Levels of serum UDP-galactose:glycoprotein galactosyltransferase in 117 unselected diabetics were compared with those in 60 non-diabetic healthy controls. 2. Enzyme activity (mean +/- 2 S.D.) of control sera was found to be 90.2 +/- 21.5 etamoles/ml/hr at 37 degrees. In 30 of the 117 diabetic sera (26%) enzyme activity was elevated (greater than mean + 2 S.D. of the controls). Sixteen of 19 (84%) patients with retinopathy, 16 of 26 (62%) patients with peripheral vasculopathy and 13 of 26 (50%) patients with neuropathy had higher levels of serum enzyme. When serum enzyme levels of groups of diabetics with retinopathy, peripheral vasculopathy and neuropathy were compared with the enzyme level in all diabetics, there was a significant difference with p values of 0.001, 0.05 and 0.05 respectively.

Adolescent↗
PubMed1975

Intestinal enterokinase deficiency. Occurrence in two sibs and age dependency of clinical expression.

Intestinal enterokinase deficiency in 2 sibs in described. A boy failed to gain weight and had vomiting, diarrhoea, oedema, hypoproteinaemia, and anaemia in early infancy. His duodenal juice contained very low or absent proteolytic enzyme activity, which increased markedly after addition of enterokinase. He was treated with pancreatic extract and gained weight rapidly. At 44 months of age he is normal, apart from some development delay, and no longer needs pancreatic extract. His older sister, who had had similar symptoms in early infancy but then grew normally, had the same abnormality in her duodenal juice when seen at 4 years of age. Enterokinase activity was virtually absent in the duodenal mucosa of both patients. Mucosal morphology was normal. The findings suggest that enterokinase deficiency is an inherited congenital defect and not the result of mucosal damage. Affected patients may show spontaneous improvement and normal growth after the age of 6 to 12 months. This phenomenon may be related to the decreasing growth volocity during the first 2 years of life and the concimitant decrease in protein requirements per unit bodyweight.

Amylases↗