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Biomedical subjects

A Kondo

Publications and source records attributed to A Kondo.

At least 433 records · Page 24Linked to original sources
PubMed1982

Suppression of bladder instability by penile squeeze.

The effect of penile squeeze upon bladder instability was examined in 20 males and the results compared with those of anal dilatation. An uninhibited contraction of the bladder was successfully suppressed by squeezing the glans penis in 17 out of 20 patients and by dilating the anal sphincter in 20. It is concluded that this mechanical stimulus, when given to the glans penis of a normal man suffering from bladder instability, suppresses the detrusor contraction during the contracting phase but does not affect bladder tonus during the noncontracting phase.

Adolescent↗
PubMed1982

A rare variant of ureteric ectopia: opening in vagina and vesico-uretero-vaginal communication.

A rare variant of ureteric ectopia, i.e. an opening in the vagina and a vesico-uretero-vaginal communication, is herein reported. This type of congenital anomaly, found in 2 females, has not been reported before. One patient had a free, 2-way communication between the bladder and the vagina via the lower segment of the right ectopic ureter. The other was found to have a one-way communication from the vagina to the bladder through the ectopic distal ureter. Surgical intervention was undertaken in both cases. It is suggested that vaginography after distending the bladder with air is important in the detection of this bizarre anomaly.

Adult↗
PubMed1982

Urinary retention caused by impaction of enlarged uterus.

2 cases of urinary retention were reported which was caused by retroverted gravid uterus and a large fibroid uterus, respectively. A chain cystourethrography and an endoscopic examination were the most informative to disclose the pathogenesis of urinary retention. Urodynamically, bladder compliance was normal without any pathological contraction. Urethral function was characterized by a shortened functional profile length.

Adult↗
PubMed1982

Hemodynamic and natriuretic responses to intravenous infusion of dopamine in patients with essential hypertension.

In order to clarify the role of dopamine on the pathophysiology of essential hypertension, mean arterial pressure (MAP), heart rate (HR), urine volume (UV), urinary sodium excretion (UNaV), endogenous creatinine clearance (Ccr), fractional excretions of sodium (FENa), inorganic phosphorus (FEP) and potassium (FEK), plasma renin activity (PRA), plasma aldosterone concentration (PAC) and plasma noradrenaline concentration (PNA) were measured before and after intravenous infusion of dopamine (3 micrograms/kg/min, 60 min) in normotensive (NT) and essential hypertensive subjects (EHT). Following dopamine infusion, a significant decrease of MAP and an increase of HR were observed in EHT but not in NT. UV, UNaV, Ccr, FENa, FEP and FEK increased significantly in both NT and EHT, and changes in these except for Ccr were significantly greater in EHT than in NT. In EHT, following dopamine infusion, PNA was clearly elevated, but no remarkable change was found in PRA and PAC. A significantly positive correlation was found between delta UNaV and delta FENa or delta FEP, and between delta FENa and delta FEP, while no significant relation was observed between delta UNaV and delta Ccr, delta MAP or MAP before dopamine infusion. A significant inverse correlation between supine PRA before dopamine infusion and delta FENa or delta FEP and a positive correlation between age and delta FENa or delta FEP were also observed in these patients. The changes in UNaV positively correlated with delta FENa and delta FEP in both low renin (group L) and normal renin EHT (group N) and with delta Ccr i group N but not in group L. The mean values of delta FENa, delta FEP and delta FEK were significantly higher in group L as compared with those in age-matched group N. These results suggest that, since the enhanced response to infused dopamine may reflect reduced dopaminergic activity, attenuation of renal dopaminergic activity might exist and be involved through a distribution of water-sodium metabolism, at least in part, in the pathophysiological mechanism in EHT, particularly in group L.

Adult↗
PubMed1982

[An autopsy case of moyamoya disease (author's transl)].

An autopsy case of severely progressed "Moya Moya" disease was presented. A 38-year-old male was admitted because of unconsciousness. On examination, he showed spider-like extremities, suggesting Marfan's syndrome. In the past history, he experienced acute rt. hemiparesis at 6 years old and generalized convulsion, lt. hemiparesis and aphasia at 7 years old. After these episodes, he spent an uneventful life until 38 years old, when he was found unconscious in his room by the family. Lt. CAG showed severe stenosis at the siphon of lt. ICA and fine vascular networks in the basal ganglia and many transdural collateral arteries. Three days later, he died and an autopsy was performed. On histological examination, proliferation of the intima, elongated elastica interna and lost of the media were identified in the stenotic arteries. In the vessels of other organs, especially of the heart, proliferated intima and lost of the media were also noticed. From these findings, "Moya Moya" disease was thought to be one of the manifestations of the systemic vascular diseases. The autopsy cases of "Moya Moya" disease in Japan were reviewed. According to the review, most of these autopsy cases died from intracerebral bleeding attack. Recently, EC-IC bypass operations have been performed in order to improve ischemic condition of "Moya Moya" disease. According to the finding of the review, however, the financial efficiency of such by-pass operations for "Moya Moya" disease must be elucidated after long-term follow-up study.

Adult↗