Search PubMed⌕ Search

Biomedical subjects

A Encke

Publications and source records attributed to A Encke.

At least 199 records · Page 11Linked to original sources
PubMed1977

[Treatment of intrahepatic cholesterol stones by chenodeoxycholic acid (author's transl)].

Intrahepatic gallstones were demonstrated by operative cholangiography in two men, aged 34 and 46 years, respectively. The cholesterol stones were dissolved in both patients within eight and two months, respectively, by retrograde instillation of chendeoxycholic acid via a T-drain. Additional endoscopic papillotomy was performed because of a papillary stenosis in one instance, and extrahepatic occlusion during treatment in the other. During treatment there was a rise of transaminases of 5-fold levels, of gamma-glutamyl transferase up to 10-fold of normal. Since chenodeoxycholic-acid instillations both patients have been receiving the drug orally to prevent recurrence of gallstones. So far all biochemical values are within the normal range, the patients are without symptoms and have a normal exercise tolerance.

Adult↗
PubMed1977

[Pathophysiology of venous thrombosis (author's transl)].

The pathophysiology of venous thrombosis is determined mainly by the triad of Virchow (hypocirculation, endothelial lesion of vessels, coagulation disturbances). Various predisposing factors are known. Thrombocytes are of utmost importance in the initial phase of thrombus formation, while fibrin and red cells influence the length of the clot and the extent of pulmonary embolism. Postoperative venous thrombosis occurs more frequently and earlier than hitherto thought (radiofibrinogen test). General prophylaxis is indicated for its effective prevention.

Adult↗
PubMed1976

[Surgical treatment of malignant melanoma (author's transl)].

The primary lesion is widely excised under a general anesthetic. The resulting defect is covered by a free graft. Prophylactic lymph-node dissection seems to be reasonable for stage-I melanoma with deep invasion (microstage III-V) according to recent investigations. Stage II is an absolute indication. Stage III patients received an individual palliative therapy. The value of additional measures is not yet clearly defined.

Germany, West↗
PubMed1976

[Histological findings after transabdominal ligation-resection of the esophagus ad modum boerema (author's transl)].

The esophagus was histologically studied in ten patients, who died after transabdominal ligation-resection of the esophagus ad modum Boerema because of bleeding esophageal varices. The patients had survived the operation for various periods of time. The questioning was, if with this operation with a higher incidence of parietal leakage following mediastinitis and peritonitis has to be calculated. We could not confirm that hypothesis. On the contrary we recognized at the level of the ligation simulataneously degenerated muscles fibres and a regeneration of the esophageal wall by fibrous tissue compensation.

Adult↗
PubMed1976

[Double-blind study for the prevention of postoperative thrombosis].

In a double-blind study a randomized group of 96 patients with abdominal operations received a placebo, dipyridamol/acetylsalicylic acid or ASA alone. Thrombi were verified by the 125-I-fibrinogen test. In 38% of the placebo group deep venous thrombosis (DVT) was found, while only 10% of the dipyridamol/ASA group (P less than 0,05) and 28% of the ASA treated patients (not significant) showed radioactive thrombi. DVT were localized in 83.8% in the calf region. 48.6% of increasend activities were registered up to the end of the first postoperative day, 78.3% up to the third day. The results emphasize the importance of a general antithrombotic prophylaxis. Dipyridamol/ASA seems to be efficient in this regard for abdominal procedures.

Abdomen↗
PubMed1975

[Local gastric fibrinolysis activation].

Hemorrhagic gastric juice contains a higher fibrinolytic activity than normal gastric juice. It is liberated from damaged muco and possibly from hemolytic erythrocytes.

Fibrinolysis↗
PubMed1975

The influence of resection of the small intestine on gastrin levels.

In 16 dogs, serum gastrin levels were determined after resection of the distal and proximal half of the small intestine. The response to feeding was significantly increased after intestinal resection. Higher postresectional levels were reached after resection of the proximal half of the intestine. This may be evidence that the proximal half of the small intestine, in addition to the liver and kidney, plays an important role in the inactivation of endogenous gastrin.

Animals↗