Search PubMed⌕ Search

SEARCH · Search PubMed

Results for “Sinoatrial Block”

Search indexed PubMed citations on genomics, clinical trials, systematic reviews and public health. Explore titles, authors and supplied subject terms, then open the PubMed record.

Quote a phrase for an exact phrase match. Source license links do not imply unrestricted reuse.

At least 19 recordsLinked to original sources
PubMed1976

First-degree sinoatrial heart block: sinoatrial block in the sick-sinus syndrome.

Sinoatrial conduction time (SACT) was estimated from the delay in the atrial recovery period after premature depolarization applied in that portion of atrial diastole when increasing prematurity resulted in a constant recovery interval. In 20 normal patients SACT was 169 msec. +/- 91 (2 S.D.). At least nine of 19 patients with "sick-sinus syndrome" (SSS) demonstrated SACT that were longer than seen in these normal subjects. SACT was prolonged in seven of nine SSS patients with abnormal A-V nodal conduction. Among 10 SSS patients with normal A-V conduction, only two had prolonged SACT. This study identifies first-degree sinoatrial block as a frequent manifestation of SSS associated with the presence of A-V node conduction abnormalities.

Arrhythmia, Sinus↗
PubMed1985

Sinoatrial block with complicating sinoatrial reciprocation.

This presentation reflects an atypical manifestation of sinoatrial block in a patient with chronic renal failure and hyperkalemia (7.8 mEq/L). An allorhythmic distribution of P-P intervals permits the interpretation of sinoatrial block complicated by sinoatrial reciprocation.

Electrocardiography↗
PubMed1985

Amiodarone-induced sinoatrial block.

We observed sinoatrial block due to chronic amiodarone administration in a 5-year-old boy with primary cardiomyopathy, Wolff-Parkinson-White syndrome and supraventricular tachycardia. Reduction in the dosage of amiodarone resulted in the disappearance of the sinoatrial block and the persistence of asymptomatic sinus bradycardia.

Amiodarone↗
PubMed1985

Atrioventricular and sinoatrial block in thyrotoxic crisis.

A 55 year old woman in thyrotoxic crisis developed atrial fibrillation, atrioventricular block, and sinoatrial block in rapid succession. All of these abnormalities resolved completely after antithyroid treatment. This course of events illustrates the profound effect of thyroid hormones on cardiac function. In view of the potential aggravation of atrioventricular conduction disturbance by beta adrenergic blocking agents, thyrotoxic patients should be carefully screened for electrocardiographic evidence of conduction disturbance before the administration of such drugs.

Electrocardiography↗
PubMed1991

Wenckebach periods in sinoatrial block: experimental and clinical evidence.

The reality of sinoatrial Wenckebach periods (WP) has been suggested, but not proven, in the literature. We report experimental and clinical data showing WP in sinoatrial blocks. Experimental sinoatrial blocks were induced by superfusion of bepridil (10(-5) M) in 15 preparations of isolated rabbit right atria. Different types of block were observed, including Blumberger I block, i.e., sinoatrial WP. The recordings showed that the typical pattern of Blumberger type IA block and sinoatrial WP may be due to transient acceleration of the sinus rate, without change in the increment. We also observed sinoatrial WP in a 72-year-old patient on direct recordings of the sinus node (SN) electrical activity. In this case, transient acceleration of the sinus rate also seemed to be involved in the genesis of sinoatrial WP. Analysis of these clinical and experimental data showed similarities that may explain the mechanism of the WP. Usually type IA Blumberger block is said to involve a decrease in the sinoatrial increment to explain the atrial sequence (decrease in PP interval followed by a pause shorter than twice the value of the preceding cycle, then a cycle longer than the one preceding the pause). In fact, this pattern can be observed when the acceleration of the higher structure, the SN, induces block within the lower structure, i.e., the atrium.

Action Potentials↗
PubMed1993

Infra-His blocked premature atrial contractions simulating 2:1 sinoatrial block in a patient with an atrio-His bypass tract.

Blocked atrial premature contractions simulated 2:1 sinoatrial block because those were superimposed on the T wave of the preceding sinus beats and were not visible on the surface electrocardiogram of our patient. Additionally, His bundle recordings and premature atrial stimulation demonstrated the presence of an atrio-His anomalous pathway. The premature atrial contractions traveled anterograde via the anomalous pathway and were blocked distally to the His bundle. The term, pseudosinoatrial block, is used to describe the arrhythmia because there was no evidence of an intrinsic abnormality of sinus node function and sinoatrial conduction.

Bradycardia↗
PubMed1982

Alternation of sinoatrial block and left bundle branch block.

A case of alternating second degree sinoatrial block and left bundle branch block is described. Electrophysiologic evaluation of this patient revealed impaired sinoatrial, atrioventricular and intraventricular conduction (panconductional defect). Alternation of intraventricular conduction disturbances with sinoatrial block can be explained by the tachycardia-dependent (phase-3-dependent) occurrence of the left bundle branch block.

Bundle-Branch Block↗
PubMed1975

Sinoatrial block. Autonomic influences and clinical assessment.

Seventeen patient with sinoatrial block and 16 healthy volunteers were investigated with recently developed autonomic tests of atrial pacemaker function. Only one patients was found to be entirely normal in comparison with normal subjects, while another patients had supernormal responses. Fifteen patients had reduced responses relative to the controls implying impaired atrial pacemaker function or sinoatrial disease. It is suggested that while in some cases sinoatrial block may be of physiological origin, it is more often due to sinoatrial disease. The latter group are liable to Adams-Stokes syncope and may have additional atricventricular conduction abnormalities. The possible mechanisms and treatment of sinoatrial block are discussed relative to the autonomic and pacemaker function abnormalities described.

Adams-Stokes Syndrome↗
PubMed1988

Two-to-one sinoatrial block. Normalization by carotid massage.

In a clinical case of 2:1 sinoatrial block, carotid massage, paradoxically, released the sinoatrial block and doubled the manifest sinus rate. This resulted from a deceleration of the sinus discharge rate which allowed consecutive sinus impulses to traverse the sinoatrial junction.

Aged↗
PubMed1994

[Electrocardiographic aspects of sinoatrial block].

There are many electrocardiographic changes in sinoatrial block. Only 2nd and 3rd degree blocks can be analysed on surface recordings. However, they manifest themselves by pauses, the descriptions of which are rich and varied. They also vary according to the circumstances of apparition and the escape rhythms which accompany them. Related ECG changes such as chronotropic insufficiency or carotid sinus syndrome have been described. However, their significance is not univocal and only a precise analysis of the ECG recordings allows correct interpretation of the clinical and paraclinical signs and the institution of appropriate therapy.

Cardiac Complexes, Premature↗
PubMed1986

Sinoatrial block induced by oral diltiazem.

A 58-year-old man with variant angina who had no history suggestive of sinus node dysfunction experienced palpitation with pulse deficit after 8 days of diltiazem treatment, 240 mg per day. Electrocardiogram (ECG) revealed frequent occurrence of sinoatrial block. After discontinuation of diltiazem, an ambulatory ECG demonstrated gradual reduction of the occurrence of sinoatrial block, and no recurrence 8 hours after the last dose of the drug. One should note that diltiazem induces not only sinus node suppression and atrioventricular conduction disturbance, but also sinoatrial block.

Administration, Oral↗
PubMed1996

[Sinoatrial block caused by gastroesophageal reflux. The role of simultaneous 24 hr. esophageal pH-metry and Holter-ECG in the differential diagnosis of angina pectoris].

UNLABELLED: The authors report on a 61-year-old female patient, who has suffered from recurrent angina-like chest pain for 30 years. The patient's complaints became intolerable, in spite of therapy with nitroglycerin, H2 receptor blockers and sedative medication. The echocardiography, the ECG exercise testing and Thallium scintigraphy were normal, the upper gastrointestinal endoscopy did not prove oesophagitis either macroscopically or microscopically. The simultaneous 24-hr Holter ECG monitoring and esophageal pH-metry demonstrated pathological acid gastro-oesophageal reflux and frequent sinoatrial blocks (Mobitz I) in painful periods. After monotherapy with proton pump inhibitor (omeprazole) the patient became complaint-free. Repeated combined 24-hr oesophageal pH-metry and Holter ECG monitoring indicated nonpathological acid reflux and insignificant number of sinoatrial blocks. During the course of 19 months the patient was asymptomatic. The acid pump inhibitor was stopped for a 10 day-period, while the chest pain returned. The combined 24-hr Holter ECG and esophageal pH-metry proved pathological acid gastro-oesophageal reflux and frequent sinoatrial blocks during chest pain period. After treatment with acid pump inhibitor the patient became asymptomatic again. CONCLUSIONS: 1. The acid gastro-oesophageal reflux may be a provocative factor of sinoatrial blocks and it can be influenced by proton pump inhibitor successfully. 2. Simultaneous 24-hr oesophageal pH-metry and Holter ECG monitoring can be contribute to the differentiation among causes of atypical chest pain.

Angina Pectoris↗
PubMed1999

Escape capture bigeminy: a manifestation of sinoatrial conduction block.

Sinoatrial block (SAB) is often difficult to identify in the presence of bradycardic rhythms. This study demonstrates several manifestations of so-called escape capture bigeminy in 14 patients. Although periods of 1:1 sinoatrial conduction can aid in the analysis of SAB, the electrocardiographic pattern of bigeminal rhythm may be the only electrocardiographic clue of SAB. In one case, both sinoatrial entrance and exit block were identified. In eight instances, digitalis or digitalis plus a beta or calcium blocking agent could be partially implicated as the cause of SAB. In 6 of 14 patients, a permanent pacemaker was required to correct the bradycardia, in spite of discontinuation of aggravating antiarrhythmic agents or electrolyte derangement.

Electrocardiography↗