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Results for “Mercury Poisoning”

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At least 19 recordsLinked to original sources
PubMed1992

Psychiatric manifestations of mercury poisoning.

Mercury and its compounds are toxic substances that are widely used in industry and agriculture. Mercury poisoning is an uncommon but important clinical entity that often presents with psychiatric disturbances as a prominent part of the clinical picture. In this paper, a case of mercury poisoning in a 12-year-old girl with prominent psychiatric manifestations is presented. Relevant literature concerning the psychiatric manifestations of mercury poisoning is discussed. The importance of considering mercury poisoning, and toxic conditions in general, in the differential diagnosis of psychiatric disorders is emphasized.

Child↗
PubMed1984

Prognosis of mercury poisoning in mercury refinery workers.

The prognosis of chronic metallic mercury poisoning in two groups of patients from the mercury refinery of a mercury mine was evaluated by reexamination which included an interview, physical and neurological examination and determination of urinary mercury. Group I consisted of 70 male patients, who had been exposed to metallic mercury for 1.6-17.8 years, 15 of whom had been diagnosed 10 years earlier as having severe chronic metallic mercury poisoning, and the rest moderate chronic metallic mercury poisoning. At the time of reexamination, they had been removed from mercury exposure for 2 months-17 years. None of them had been treated with any chelating agent. Group 2 comprised 84 male patients from the same mercury mine, who had been exposed to metallic mercury for 2-10 years after 1962 and had been previously diagnosed as having mild chronic metallic mercury poisoning. They were reexamined after 2 months of hospital admission and chelation treatment with unithiol or sodium dimercaptosuccinate (Na-DMS). Based on clinical evaluation, the condition of the patients in both groups had all improved, even in the severe cases. The overall prognosis of chronic metallic mercury poisoning in mercury refinery workers was encouraging after termination of mercury exposure. Chelation therapy with unithiol or Na-DMS was evidently beneficial for reducing urine mercury and some symptoms, but not for neurological and stomal signs.

Chelating Agents↗
PubMed1988

Acute mercury poisoning and mercurial pneumonitis from gold ore purification.

We describe four men who had symptoms of acute mercury poisoning following exposure to mercury vapor. They were attempting home gold ore purification using a gold-mercury amalgam and sulfuric acid. Three of the four patients required treatment with penicillamine. The clinical and laboratory data are presented along with pulmonary function test results. Long-term follow-up of one patient indicates residual morbidity, with continued reduction in pulmonary diffusing capacity. This suggests permanent impairment of pulmonary function despite prompt chelation therapy.

Acute Disease↗
PubMed2003

A new cutaneous sign of mercury poisoning?

Chronic mercury poisoning is becoming a health concern because of extensive pollution of water and fish, and the increasing consumption of fish in the human diet. Mercury is extremely toxic to the body, especially the central nervous system, but diagnosis is difficult because of the lack of specific signs. A total of 11 patients were observed to have a nonpruritic or mildly pruritic discreet papular and papulovesicular eruption that correlated with high blood mercury levels. The mercury evidently came from increased seafood consumption. All of the patients improved when they were placed on either a seafood-free diet or chelation therapy. Physicians should suspect mercury poisoning in patients who eat a high-seafood diet who present with an asymptomatic or mildly pruritic papular or papulovesicular eruption.

Adult↗
PubMed1989

[Atmospheric pollution and mercury poisoning caused by peasants mercury smelting].

The article reports the results of the investigation on atmospheric pollution and mercury poisoning caused by the peasants mercury smelting. Mercury-smelting is a sideline occupation of the peasants in the mining area. They carried out the production in their homes, which were not protected. The equipment used was simple and the technological level low. Much was done during the slack seasons in farming. Before mercury-smelting was carried out the mercury concentration of air inside the house of the key household was 0.099 2 mg/m3, and outside 0.060 9 mg/m3. During mercury-smelting, near the stove, inside the house of the key household and the next door neighbour the concentrations were high, reaching 0.6463, 0.3160, 0.1717 mg/m3 respectively. At 10m, 30m, 100m, from the stove the figures were 0.0628, 0.0377 and 0.0079 mg/m3 respectively. 126 persons in 36 key households and their neighbours were given physical examinations and the incidence of chronic mercury poisoning of the operators, family members and their neighbours' family members were 75.00%, 13.79%, 3.57%, the mercury absorptivity were 12.50%, 34.48% and 21.43%, while the concentrations of mercury in the urine were 59.18, 18.21, 9.62 micrograms/L respectively. The mercury absorptivity of children under 15 was high 45.45%. The youngest age that absorbed mercury was 3. The characteristics of the harm that the peasants mercury smelting caused were discussed and suggestions proposed for their remedy.

Adolescent↗
PubMed1981

A novel approach for heavy metal poisoning treatment, a model. Mercury poisoning by means of chelating microspheres: hemoperfusion and oral administration.

The chelating drugs BAL (2,3-dimercaptopropanol), EDTA (ethylenediaminetetraacetic acid), and penicillamine (2-amino-3-mercapto-3-methylbutanoic acid), which are used for metal poisoning, are toxic and there is a real need for alternatives, especially for severe cases. A novel approach for treatment of heavy-metal poisoning is under investigation in our group. The approach utilizes the synthesis of chelating microspheres specific for the desired metallic compound. The microspheres are suggested for use in severe cases by means of hemoperfusion, as a first aid, and then by oral administration. As a model this approach was tried for mercury poisoning. Polymercaptal microspheres of 0.8 micrometer average size were synthesized. The microspheres have a high surface area, have a high affinity toward organic and inorganic mercury compounds, and can compete easily with albumin and cysteine in the ability to bind mercury compounds. These microspheres also were encapsulated with agarose--a blood compatible polymer--and were tried successfully for plasma perfusion (in 10 min, 40% of CH3HgCl and of HgCl2 were removed from 20 ppm of poisoned plasma).

Administration, Oral↗