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PubMed2006

[Diagnostic and treatment of necrotizing cervical fascitis. Clinical course after a Ludwig angina].

The Ludwig angina is an infection of the base of the mouth and submandibular region, frecuently after a dental extraction or a piece in bad state. When it surpassees the milohioid muscle it extends dissecting the superficial aponeurotic planes, and can evolve to a necrotisant fascitis. We present the case of a 67 years old man with a painful tumefaction of the mouth base and submandibular region. The CT reflected an heterogenous submaxilar lesion that extended by the deep cervical fascia introducing itself in the thorax. Under antibiotic treatment the pus was drained by cervicotomy, with daily cures during 3 weeks.

Aged↗

PubMed1977

Ludwig angina in childhood.

Two patients had Ludwig angina. Prompt recognition of this once common, but now rare, clinical entity is essential to save the life of the patient.

Child↗

PubMedDate not supplied

Ludwig's angina.

Ludwig's angina is a serious, often fatal infectious disease process that requires prompt intervention of life-saving therapies. The critical care nurses caring for a patient with Ludwig's angina must be able to recognize subtle changes in the patient's status and intervene quickly to prevent death by airway edema or profound sepsis.

Airway Obstruction↗

PubMed2000

[Ludwig's angina].

Ludwig's angina is a rare infectious condition originating in the oropharyngeal region, most commonly from the teeth. The principal symptoms consist of cervical pain, dyspnea, dysphagia, symmetrical neck swelling and fever. Serious complications include respiratory collapses, mediastinitis, pleural empyema, pericarditis and pericardial tamponade, and may result in the death of the patient. The most useful investigations in patients with suspected Ludwig's angina are clinical assessment, a cervico-thoracic CT-scan to determine the extent of the lesions, microbiological examination and panoramic radiography to detect possible dental focuses of infection. Treatment consists of ensuring adequate ventilation by securing the upper airways, broad spectrum antibiotic therapy, eradication of the source of infection, and often early surgical decompression may be necessary. Rapid diagnosis and appropriate treatment can reduce the mortality rate to 10%.

Adult↗

PubMed1980

Ludwig angina, empyema, pulmonary infiltration, and pericarditis secondary to extraction of a tooth.

A case of Ludwig angina after extraction of a mandibular third molar, progressing to pleural effusion and empyema, pericarditis, pulmonary infiltration, and pericardial effusion has been presented. The importance of early diagnosis and treatment as well as appropriate antibiotic and surgical therapy have been discussed. The practice of empirically prescribed antimicrobial agents has also been discussed, and the relevance of the organism E corrodans in oral infections has been emphasized.

Adult↗

PubMed1992

Ludwig's angina.

Ludwig's angina is a cellulitis frequently occurring as a result of infections of the second and third lower molar. Despite a decrease in mortality from 50% to less than 10% since the introduction of antibiotics, it remains a rare but life-threatening illness. The potential for rapid respiratory obstruction is the greatest concern. Familiarity with the anatomy of the neck and recognition of symptoms are essential for effective treatment. Treatment focuses on maintenance of an airway, antibiotic therapy, and surgery. Asphyxia, aspiration, mediastinitis, pneumonia, empyema, and septicemia are possible complications.

Female↗

PubMed2001

[Ludwig angina: a disease of the past century. Case report].

The case of a patient with Ludwig's angina, diffuse inflammation of the submandibular and sublingual spaces, a rare but life threatening disease, is described. This disorder can develop almost always as a complication of the dental infection. Causative bacteria include many Gram-negative, anaerobic organisms, streptococci and staphylococci. The potential for rapid respiratory obstruction is the greatest concern. Other serious complications include sepsis, mediastinitis, pleural empidema, pericarditis, pericardial tamponade. A cervico thoracic CT-scan and neck RNM, were performed to determine the extent of the inflammatory lesion. Treatment consists of ensuring adequate ventilation, with tracheostomy, broad spectrum antibiotic therapy and surgical drainage of the source of infection. The patient recovered without complications.

Anti-Bacterial Agents↗

PubMed2006

Did Ludwig's angina kill Ludwig?

Wilhelm Frederick von Ludwig first described in 1836 a potentially fatal, rapidly spreading soft tissue infection of the neck and floor of the mouth. The condition was later named 'Ludwig's angina', a term which persists in medicine to this day. A gold medallist at 19 and professor at 25, Ludwig also served as president of the Württemberg Medical Association and chief physician to the royal family. His outstanding contribution to medicine was rewarded with the title Excellence upon retiring in 1855. Ludwig died at the age of 75, ironically, days after developing an inflammation of the neck. Could it be that Ludwig died of his own condition? This article combines a biography of Wilhelm Frederick von Ludwig with an overview of his eponymous condition and its management.

Eponyms↗

PubMed2001