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Results for “Heart Septal Defects, Ventricular”

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At least 19 recordsLinked to original sources
PubMed1979

Surgical technique to reduce the risks of heart block following closure of ventricular septal defect in atrioventricular discordance.

Traumatic heart block remains a major concern after ventricular septal defect (VSD) closure in patients with atrioventricular discordance. A technique of closing the VSD, placing the suture line on the morphologically right side of the septum without opening the systemic ventricle, is described. This was used in 13 consecutive patients. The VSD was closed through the right atrium in eight patients, the left ventricle in three patients, the right atrium and the left ventricle in one patient, and through the right ventricle in one patient with atrioventricular disordance and ventriculo-arterial concordance. All patients were in sinus rhythm preoperatively, two exhibited atrioventricular dissociation before intracardiac manipulation began, and 11 patients were in sinus rhythm postoperatively. No major arrhythmia could be attributed to the closure of the VSD.

Adolescent↗
PubMed1979

Muscular ventricular septal defects: a reappraisal of the anatomy.

Among 79 autopsy specimens of heart with an isolated ventricular septal defect, there were 29 cases of muscular defect. Among 60 hearts with complete transposition of the great arteries and a ventricular septal defect, there were 13 cases with a muscular defect. All muscular defects could be classified in three different types, based on the specific pathologic anatomy of the ventricular septum. The central and posterior defects were usually large and single, the marginal defects were frequently small and multiple. In hearts with transposition, central muscular defects were extremely rare, whereas these defects were by far the most frequent muscular defects in isolated ventricular septal defect. Alternatively, the posterior type was more common in cases of transposition. Marginal muscular defects were rare in both groups of malformations.

Heart↗
PubMed1977

[Rhabdomyomatosis of the heart and ventricular septal defect (author's transl)].

An unusual case of cardiac rhabdomyomatosis associated with a ventricular septal defect without signs of tuberous sclerosis is described. On 3 different angiocardiographies the region of the tumor seemed to be a right ventricular aneurysm. The tumor was identified as such only on operation and its nature defined histologically. Subtotal excision of the tumor and closure of the large ventriccular septal defect and debanding (pulmonary banding at the age of 8 month) was successfully undertaken at the age of 10 years.

Heart Neoplasms↗
PubMedDate not supplied

Acute bacterial endocarditis following traumatic ventricular septal defect.

Traumatic ventricular septal defect occur rarely although penetrating wounds of the heart rate are fairly common. The urgency for the surgical correction of traumatic ventricular septal defects has been debated. In life-saving instances where left heart failure or cardiac tamponade occur, the decision is simple. A case report of early bacterial endocarditis complicating traumatic ventricular septal defect in the first 12 hours following injury has been presented. Avoidance of this complication by early surgical correction is advocated.

Acute Disease↗
PubMed1976

Left heart volume characteristics following ventricular septal defect closure in infancy.

Left ventricular and left atrial volume, left ventricular ejection fraction, and left ventricular muscle mass were determined preoperatively and postoperatively in 13 patients who underwent surgical closure of ventricular septal defects in the first two years of life. Left ventricular end-diastolic volume and systolic output averaged 255 +/- 19% (+/- SEM) and 240 +/- 19% of normal, respectively, before operation but fell to within normal limits postoperatively. Left ventricular ejection fraction was normal preoperatively (100 +/- 4% of normal) and remained so after correction (106 +/- 3%, NS). Left ventricular mass was mildly elevated at the preoperative catheterization (271 +/- 21%) and decreased significantly following repair (P less than 0.001). However, the postoperative left atrial volume (147 +/- 14%) remained abnormal (P greater than 0.05). These data suggest that when early surgical closure of a ventricular septal defect is necessary because of failure of medical management, good results with regard to postoperative left ventricular size and function can be expected.

Cardiac Catheterization↗
PubMed1979

Embryology of the univentricular heart.

Controversies remain concerning the nomenclature, nature and pathogenesis of the various forms of univentricular heart. Common ventricle may well represent a very large ventricular septal defect while the nature of the indeterminate or nonspecific form of univentricular heart remains in doubt. Univentricular heart, right ventricular type, could be ascribed to overshift of the embryonic atrioventricular canal to the right, resulting in both atrioventricular ostia entering a large ventricular chamber which has the morphological characteristics of a right ventricle. In univentricular heart, left ventricular type on the other hand, the rightward shift of the atrioventricular canal appears to have failed or remained incomplete, possibly due to an abnormal position of the atrioventricular canal relative to the bulboventricular septum. Normal septation of the atrioventricular canal results in both atrioventricular ostia giving access to a large ventricle which in the main has the morphological features of a left ventricle.

Adult↗
PubMed1979

[Surgical treatment of complications of myocardial infarct].

Complications of myocardial infarction in the form of aneurysm of the heart, mitral incompetence and ventricular septal defect cause considerable changes in intracardiac hemodynamics which lead to severe disturbances in the circulation system in different periods after myocardial infarction. The timely and properly made diagnosis allows the prognostic severity of the complication to be appraised and the optimum therapeutic tactics determined. The given classifications of this pathological conditions promote a differential approach in considering the indications and contraindications in each case. Analysis of 312 operations shows that correction of the abnormality under conditions of extra-corporeal circulation in combination with direct myocardial revascularization is the most adequate intervention.

Chronic Disease↗
PubMed1976

[Surgical correction of ventricular septal defects treated by banding the pulmonary artery. Apropos of 38 cases].

The authors report their experience with two-stage surgical treatment of severe ventricular septal defects in babies: initial banding of the pulmonary artery, followed by closure of the ventricular septal defect and removal of the band by an open-heart technique. Out of 162 operations to band the pulmonary artery, 90 were for cases of ventricular septal defect. The mortality was low (9.1%) when there was a ventricular septal defect with or without a patent ductus. It is considerably higher (31.4%) when there is a coincident coarctation of the aortic isthmus. So far 38 children have had the second operation, with 2 deaths. The authors have studied the surgical problems and the long-term results (6 months to 3 years follow-up) in the first 29 patients undergoing surgery. Although the results from this two-stage operation are good, when the authors take into account their current experience with one-stage closure of ventricular septal defects in the babies, they feel that this latter technique is to be preferred. Reservations must, however, be expressed when the child is very young (Weighing 3.5 kg or less), when there are multiple defects between the ventricles, and when the defect is severe, with a combination of VSD, patent ductus and coarctation of the aorta, and sometimes when the opertion must be carried out as an emergency.

Aortic Coarctation↗
PubMed1977

Double outlet right ventricle: clinical spectrum and prognosis.

Patients with double outlet right ventricle can be separated into four distinct groups. In the largest (Group III), patients have a subaortic ventricular septal defect and pulmonary stenosis and clinically resemble patients with tetralogy of Fallot. In the next largest group (Group I), patients have a subpulmonry ventricular septal defect and no pulmonary stenosis and clinically resemble children with D-transposition of the great arteries and a ventricular septal defect. These patients have a high rate of coarctation of the aorta leading to early congestive heart failure, and their overall prognosis is poor. In the next largest group (Group II), patients have a subaortic ventricular septal defect and no pulmonary stenosis. Their presentation is similar to that of children with a large ventricular septal defect and pulmonary hypertension. In the smallest group (Group IV), the ventricular septal defect is uncommitted. Survivors in this group also clinically resemble children with a large ventricular septal defect and pulmonary hypertension. When present, coarctation of the aorta and severe mitral valve abnormalities greatly influence the prognosis in double outlet right ventricle. Although the prevalence of associated cardiac abnormalities is large, asplenia, polysplenia, chromosomal abnormalities and other congenital noncardiac abnormalities occur in only 12.5% of patients with double outlet right ventricle.

Aortic Coarctation↗