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Results for “Hearing Loss, Bilateral”

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At least 19 recordsLinked to original sources
PubMed2003

[Glasgow Benefit Plot for evaluation of stapedectomy result].

In a group of 153 patients, the result of unilateral stapedectomy was evaluated using Glasgow Benefit Plot. Pure tone average at frequencies 0.5; 1; 2; and 3 kHz of operated and non-operated ear were used to distribute patients to pre- and post-operative groups. In 26 (79%) of 33 patients with unilateral hearing loss, bilateral normal hearing was achieved. Thirty one (46%) of 68 patients with asymmetric bilateral hearing loss and 37 (71%) of 52 patients with symmetric bilateral hearing loss had unilateral normal hearing after the operation. Twenty (29%) patients of the group III had bilateral symmetrical hearing loss after surgery. Stapedectomy was less beneficial for 17 (25%) of 68 patients with asymmetric hearing bilateral loss and 15 (29%) 52 of patients with symmetric bilateral hearing loss, who still had asymmetric hearing loss after the operation. Evaluation of hearing tests using the Glasgow Benefit Plot enables to evaluate patient's hearing disability and to predict possible benefit from surgery for individual cases.

Adolescent↗
PubMed1997

CT screening for temporal bone abnormalities in idiopathic bilateral sensorineural hearing loss.

Bilateral sensorineural hearing loss can be caused by a variety of temporal bone abnormalities including primary cochlear otosclerosis, local and systemic bony diseases and some metabolic conditions. These may be identified using computerized tomography (CT), with attenuation recordings taken across the cochlear capsule (CT densitometry). Eighty patients with bilateral sensorineural hearing loss were screened over a period of six and a half years using this technique, and only three cases (3.8 per cent) of treatable disease were detected. Positive yields may be increased by screening selected cases with other clinical or biochemical stigmata of temporal bone disease.

Absorptiometry, Photon↗
PubMed1981

Acute bilateral hearing loss during a pop concert: consideration for differential diagnosis.

A 40-year-old male administration employee suffered an acute acoustic trauma while attending a pop concert. This paper discusses the differential diagnosis. The patient's mother was also hard of hearing following bomb attacks during the last war. The degree of hearing loss did not correlate with the effect of the noise in either case. It is therefore assumed that the inner ear has developed greater susceptibility to noise. The origin of this is attributed to a genetically induced weakness of the cochlea to exogenous factors.

Adult↗
PubMed2001

[Unilateral hearing loss in childhood. An empirical analysis comparing bilateral hearing loss].

BACKGROUND: Unilateral cochlear hearing loss is considered as a risk factor for auditory, verbal-communicative, behavioral and academic development. An early diagnosis is therefore necessary. METHOD/PATIENTS: 182 consecutive patients with an age up to 10 years were diagnosed with permanent hearing loss in the mild to profound range in a defined 5-years-period (1.10.1994-30.9.1999) in the Pedaudiologic Outpatient Clinic of the University Göttingen. Fifty children (27.5%) had a unilateral hearing loss (30 sensorineural, 20 conductive caused by aural atresia with or without microtia), 132 a bilateral one. This paper presents the data of the consecutive series of the 30 sensorineural unilaterally hearing-impaired children (> 25 dB). RESULTS: A slight male predominance was present (53.3%). The left ear was affected in 43.3%, the right ear in 56.7%. In the majority of the cases the hearing loss was severe and profound. The hearing-impairments were diagnosed by the median age of 69.5 months and all aided by the median age of 70 months. The etiology remained unknown in 60 per cent of the cases. Hearing aid acceptance at the first follow-up (on average after 6 months) was found to be 79 per cent. CONCLUSIONS: The data suggest the relevance and necessity of a pedaudiometric prevention. They demonstrate the urgent necessity of a molecular genetic cause investigation. Recessive sensorineural hearing loss with onset in infancy may exist with no antecedent family history.

Child↗
PubMed2000

[A case of brain stem infarction with bilateral hearing loss].

The study case was a 66-year-old man who had bilateral neurosensory hearing impairment due to brain stem infarctions. He noticed mild hearing loss, frequent vertigo and tinnitus. About one month later, his hearing took a sudden turn for the worse, and he suffered from dysarthria, dysphagea and abasia. Neurological examination revealed pseudobulber palsy, left hemiparesis, cerebeller ataxia, disturbance of pain and temperature sensation on the right face and left side of the body. Brain stem auditory evoked potentials (BAEPs) showed a delayed small wave V with the abscence of previous waves on the right side and no significant waves on the left side. Brain magnetic resonance images (MRI) revealed infarctions in the bilateral middle cerebellar peduncles, including in the right lateral portions of pons, and the right lower pontine base. We believe that not only peripheral, but also central auditory pathways adjacent to infarctions were damaged. Magnetic resonance angiography (MRA) showed severe stenosis or occlusion of left vertebral artery and basilar artery. We concluded that hypoperfusion of the vertebrobasilar artery territories caused ischemia of the cochlear nerve and the auditory tracts in the brain stem, which resulted in bilateral hearing loss.

Aged↗
PubMed1986

Sudden bilateral hearing loss after spinal anaesthesia. A case report.

A case of bilateral hearing loss following an otherwise uncomplicated spinal anaesthesia with bupivacaine is presented. It is suggested that a drop in intracranial pressure played a principal role in the development of this complication. Unless specifically sought for, minor auditory losses following spinal anaesthesia may be overlooked.

Aged↗
PubMed1978

Sudden bilateral hearing loss and meningitis in adults.

Sudden profound bilateral sensorineural hearing loss is a serious complication of bacterial meningitis. With high dosage Dexamethasone it is possible to halt, and, in some instances, to reverse the progression of the hearing loss. The efficacy of treatment depends upon early diagnosis. Hearing may worsen upon steroid cessation and low dosage oral steroid may be required for several months. The eventual hearing loss may be considerably worse than anticipated. Two cases illustrating differing aspects of the disease are described.

Adult↗
PubMed2000

[Sudden bilateral hearing loss with vertigo due to vertebral artery occlusion].

We report a case of sudden bilateral hearing loss associated with an occlusion of the right vertebral artery. A 66-year-old man was admitted to hospital suffering from sudden onset vertigo, deafness, and vomiting. He could not walk due to truncal ataxia. There was positional nystagmus to the right; spontaneous and gaze-evoked nystagmus were absent. He had no facial nerve palsy, dysarthria, pyramidal tract signs, limb ataxia, and sensory impairment. Pure tone audiometry demonstrated a profound sensorineural deafness of both ears. A tentative diagnosis of sudden deafness was made. He was treated with intravenous infusion of corticosteroid; hearing loss of the left ear slightly improved. Cranial MR imaging demonstrated fresh small infarcts in the right cerebellar tonsil, the left cerebellar medulla, and the left middle cerebellar peduncle. MR angiography demonstrated an occlusion of the right vertebral artery. It is possible that reduced perfusion of the anterior inferior cerebellar arteries and internal auditory arteries on both sides resulted in multiple "border zone" infarcts and damage to the inner ear. Clinicians should be aware of the possibility of vertebrobasilar occlusive disease in case of sudden bilateral hearing impairment, even when brainstem or cerebellar signs are absent.

Aged↗
PubMedDate not supplied

Bilateral hearing loss due to viral infection.

We encountered 2 patients with bilateral hearing loss due to the mumps virus in one and the varicella-zoster virus in the other. Each virus was identified by a serological test, and virus-specific IgG and IgM antibodies were detected in the sera. In both patients, hearing loss on the contralateral side occurred a few days after that on the initially affected side, but vestibular function was normal. Although therapy included the administration of steroids, antiviral agents and hyperbaric oxygen, the bilateral hearing loss remained unchanged in these 2 patients. Both required the use of a hearing aid after discharge.

Adult↗
PubMed2002

[Sudden bilateral hearing loss disclosing meningeal carcinomatosis].

We report the case of a patient presenting a sudden bilateral hearing loss. Four years before, a bladder carcinoma was resected and a chemotherapy was started. Gadolinium-enhanced magnetic resonance images revealed enhancement of both acoustic nerves. Cerebrospinal fluid analysis showed malignant cells consistent with the initial bladder cancer. Meningeal metastases from bladder carcinoma are extremely rare. Systemic chemotherapy and its low meningeal diffusion may enhance the incidence of this complication. Bilateral hearing loss is a rare initial manifestation of meningeal carcinomatosis.

Antineoplastic Agents↗
PubMed2004

[Etiology of severe/profound, pre/perilingual bilateral hearing loss in Cantabria (Spain)].

OBJECTIVE: To know the etiology of preiperlingual bilateral hearing loss in children. MATERIALS AND METHODS: All the patients diagnosed with bilateral severe/profound, pre or perilingual hearing loss at Sierrallana and Marqués de Valdecilla Hospitals (Cantabria, Spain) during the last 20 years were included in this study. RESULTS: A hundred patients were diagnosed with bilateral severe/profound pre/perilingual hearing loss. The most frequent etiology was hereditary (49%), followed by severe perinatal hypoxia (11%), ototoxicity (5%), meningitis (3%), hyperbilirubinemia (3%) and rubella (2%). In 21% of cases was not known. CONCLUSIONS: The two most frequent etiologies found in severe/profound hearing loss in children in our area were hereditary and non infectious perinatal problems. Infectious disease were scarce. Will decrease when genetic test were used as clinical basis.

Female↗
PubMed2006

Vestibular evoked potentials (VsEPs) in patients with severe to profound bilateral hearing loss.

OBJECTIVE: To confirm the exclusive vestibular dependence of the evoked potentials (P10 and N15) recorded in normal subjects, by recording the potentials evoked in response to bone-conducted sound stimulation in patients with bilateral hearing loss. METHODS: Fourteen patients with severe to profound bilateral hearing loss were stimulated via a B71 bone-vibrator above the mastoid with bone-conducted tone bursts (500 Hz, 6 ms) at fixed levels above their individual vestibular evoked myogenic potential (VEMP) thresholds. Surface potentials were recorded from scalp electrodes at F7, F3, Fpz, F4, F8, T3, Cz, and T4 and referred to linked earlobes. RESULTS: Seven of 14 patients had suitable VEMPs to the maximal stimulus. P10 and N15 potentials were present in each of these 7 patients, but were absent in patients with absent VEMP responses. The potentials were only detected above VEMP threshold, and were similar in size, shape and latency to those recorded in normal controls at a matched intensity above VEMP threshold. CONCLUSIONS: Normal P10 and N15 potentials can be recorded from patients with profound bilateral hearing loss using bone-conducted acoustic stimulation of the vestibular apparatus. SIGNIFICANCE: The P10 and N15 appear to be dependent purely upon vestibular activation.

Acoustic Stimulation↗
PubMed2006

[A case of infarction in brainstem and cerebellum as a initial symptom with bilateral hearing loss].

A 56-year old male presented with a sudden onset of bilateral hearing difficulty. He complained of dizziness and gait disturbance at the onset and subsequently developed bilateral hearing loss and tinnitus. Brain MRI revealed multiple infarcts in bilateral middle cerebellar peduncles, bilateral cerebellar hemispheres and the right cerebral peduncle. Three dimentional computed tomography angiography (3D-CTA) showed severe stenosis of bilateral vertebral arteries. Infarcts were located in the border zone between anterior inferior cerebellar artery (AICA) and superior cerebellar artery (SCA), suggesting hemodynamic infarctions. Auditory brain stem responses (ABR) were recorded three times. The initial ABR demonstrated all waves except for wave I on day 14. Wave I on the left was normal, while wave I peak latency on the right was prolonged. On day 61, all waves were recorded, although peak latencies of waves III to V and interpeak intervals of the wave I to III on the right side were prolonged. Involvements of the cochlear nerve and pontine auditory pathway were suggested from the ABR abnormalities in this case.

Anterior Cerebral Artery↗
PubMed2003

[Progressive bilateral hearing loss with superficial hemosiderosis of the central nervous system: contribution of cochlear implantation].

Two cases of progressive bilateral hearing loss associated with superficial hemosiderosis of the central nervous system are reported. This is a rare disease caused by repetitive hemorrhage in subarachnoid spaces with hemosiderin deposits on the brain surface and cranial nerves. MRI provided the etiologic diagnosis in both cases based on typical low-density signals from the brain, the brainstem, and the cerebellar surfaces on T1 and T2 sequences. In one case a fourth ventricle ependymoma, which was the probable cause of hemosiderosis, was also discovered. Based on a literature review, we discuss the pathophysiological hypotheses, the modalities for treating hearing loss including cochlear implantation despite retrocochlear sensorineural hearing loss.

Adult↗