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Results for “Erysipelothrix Infections”

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At least 19 recordsLinked to original sources
PubMed1975

Salmonella and Erysipelothrix infection in swine: a laboratory summary.

The occurrence of salmonellas and Erysipelothrix rhusiopathiae in tissues of swine was determined by bacteriologic and fluorscent antibody examination of 5,297 specimens from herds investigated for hog cholera. Of the specimens examined, 35.5% were positive for salmonellas, 21.1% were positive for E rhusiopathiae, and 8.3% were positive for both salmonellas and E rhusiopathiae.

Animals↗
PubMed1986

[Pathomorphological changes in experimental Erysipelothrix infection in lambs].

Investigations were carried out with eight lambs treated i/v with a broth culture of Erysipelothrix rhusiopathiae at the rate of 10(8) microbial cells per 1 cm3. Following infection the lambs developed polyarthritis. Studied were the morphologic changes in both dead and slaughtered lambs at various intervals after the infection was performed. It was found that from the 2nd up to the 7th day the lesions were characterized by moderate infiltration (neutrophil granulocytes) of the parenchymal organs as well as by dystrophic- and necrotic foci in the liver and hyperemia, edema, and hemorrhages in the spleen, lung, and brain. After the 7th day were found reticulohistiocytic proliferations in the liver and heart, and intracapillary glomerulonephritis, thromboendocarditis, and polyarthritis.

Animals↗
PubMed1978

Effect of experimentally induced trichinellosis on the infection with Erysipelothrix rhusiopathiae in rats.

Infection with Erysipelothrix rhusiopathiae in rats infested 20 days earlier with Trichinella spiralis developed more slowly, the clinical and pathoanatomic changes in the joints were expressed to a less extend, and the mortality rate was lower. The erythrocyte sedimentation rate, the precipitin formation and the phagocytic activity of the macrophages did not considerably change. Experiments carried out to elucidate this fact did not reveal any antigenic or antagonistic relationships between parasite and bacterium nor any protective effect of the host's serum. The inhibitory influence of corticosteroids on the defence forces was not completely manifested in the rats infested. This fact might be explained by interrelations depending both on the cycle of helminth development and on the non-specific immunological reactivity of the organism, the latter being stimulated by the helminth invasion.

Adrenocorticotropic Hormone↗