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Results for “Auditory Fatigue”

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At least 19 recordsLinked to original sources
PubMed1988

[Evaluation of the effect of auditory fatigue on the human ear by auditory brain stem responses (ABR). I. Effect of auditory fatigue on temporary threshold shifts of ABR].

The effects of auditory fatigue on auditory brainstem responses (ABR) were investigated in 23 normal-hearing subjects. ABR was recorded before and after exposure to fatiguing noise. Behaviourally, the auditory fatigue was tested using a temporary threshold shifts (TTS) paradigm. The latency of waves I, III, V and interwave intervals I-III, III-V, I-V were analysed as functions of stimulus intensity. In post-exposure recordings a threshold elevation, significant increase of all waves latency, increase of interwave-interval III-V at 40,50,60 dB nHL and unaltered intervals I-III, I-V were observed.

Adult↗
PubMed1988

[Evaluation of the effect of auditory fatigue on the human ear based on the measurement of brain stem auditory potentials (ABR). II. Relation of temporary auditory threshold shift and changes in the latency of wave V].

The relationship between temporary threshold shift (TTS) and latency changes of auditory brainstem responses (ABR) induced by noise exposure was studied in 23 normal-hearing subjects. Shift in latency wave V at 40 dB nHL of stimulus intensity as a result of auditory fatigue showed no significant correlation with TTS at 4 kHz. The great intersubject variability of shifts in latency wave V as a result of auditory fatigue suggests that this shift may be a useful measure of individual susceptibility to noise.

Acoustic Stimulation↗
PubMedDate not supplied

Developmental changes of susceptibility to auditory fatigue in young hamsters.

Susceptibility to auditory fatigue was studied in young hamsters by using an evoked-potential criterion of sound-induced threshold shift. Animals aged 15, 28, 40, 54 and 85 days were anesthetized and stimulated with a continuous tone (3 kHz, 110 dB SPL) for 10 min. Threshold shifts 1 min post-exposure were highest in animals aged 40 days, and lowest in animals aged 15 or 85 days. Threshold shifts recovered within 100 min in 15- and 85-day-old animals, but required considerably longer to recover in the other age-groups. The data suggest that young hamsters pass through a critical period of susceptibility to auditory fatigue. Comparison of this critical period with various indices of the development of hearing in the hamster suggests that the developmental events underlying the critical period do not occur in the middle ear.

Acoustic Stimulation↗
PubMed1983

Effects of auditory fatigue on speech intelligibility and lexical decision in noise.

The influence of weak auditory fatigue on speech identification and lexical decision (word/nonword) was studied. It appears that, at a low listening level and in presence of a strong masking noise, auditory fatigue gives rise to: (1) a decrease in identification scores, (2) more frequent confusions for fricatives, (3) a reduction in correct lexical decision scores, (4) an increase in the tendency to respond "word" more often than "nonword" (5) a slight increase in reaction times for incorrectly repeated items. The intelligibility impairment can be attributed to a combination of masking and fatigue effects. Errors in lexical decisions and changes in response times are explained by an adaptation of the lexical decision processes to the degraded perceptual representations of the stimuli. There is no clear evidence in this experiment that a very central component of auditory fatigue influences lexical decisions.

Adult↗
PubMed1984

Effects of the ovarian and contraceptive cycles on absolute thresholds, auditory fatigue and recovery from temporary threshold shifts at 4 and 6 kHz.

Absolute thresholds at 4 and 6 kHz were tested in three sessions before and after 20 min of exposure to 105 dB(A) pink noise in 12 young normal-cycling females, 11 young females on oral contraceptives and 8 young men. Women on contraceptives showed lower resting thresholds, larger temporary threshold shift (TTS) and higher recovery rates than normal-cycling females. The analysis of resting thresholds, auditory fatigue and recovery from auditory fatigue did not evidence any sex-linked difference. Significant differences linked to the phases of the menstrual cycle and of the contraceptive cycle were observed during recovery from auditory fatigue at 4 kHz and in resting thresholds at 6 kHz. Application of Kendall's coefficient of concordance confirmed these results. Absolute thresholds were highest at menstruation and lowest in the postovulatory phase in normal-cycling females and in women on oral contraceptives, during the days of pill ingestion. Oral contraception is probably a more important factor of change in hearing performance than the phases of the menstrual cycle.

Adult↗
PubMedDate not supplied

Psychoacoustic correlates of susceptibility to auditory fatigue.

The present study was designed to explore psychoacoustic correlates of susceptibility to auditory fatigue. Fifty-six normal-hearing subjects were given two monaural auditory fatigue tests. The high-frequency test consisted of a 3-min exposure to a 110 dB SPL, 2 kHz pure tone, with temporary threshold shift (TTS) measured at 4 kHz. The low-frequency test consisted of a 3-min exposure to a 115 dB SPL, 500 Hz pure tone, with TTS measured at 1 kHz. Amount of TTS and TTS recovery time were compared with performance on a test battery consisting of: (1) masking level differences; (2) brief-tone audiometry; (3) speech discrimination in noise; and (4) the threshold of octave masking test. A small negative correlation was found between amount of TTS and results of the threshold of octave masking test and the results of brief-tone audiometry. Subjects with larger amounts of TTS tended to have lower thresholds of octave masking and flattened threshold-duration functions.

Adolescent↗
PubMed1988

Effects of intake and rejection tasks on auditory fatigue.

The effects of sensory intake and rejection tasks on auditory fatigue were examined in 14 male subjects. Auditory thresholds, psychophysical tuning curves and physiological measures of cardiovascular function were obtained before, during, and following a 7 min 110 dB SPL white noise exposure. Acceleration of heart rate was observed under the sensory rejection (mental arithmetic) condition, and poorer post-exposure auditory thresholds and larger Q10 tip values (measured during late post-exposure intervals) were seen when the task required counting of interruptions in the noise. However, Q10 tip values obtained 1 to 2 min post-exposure, when cochlear effects are maximal, failed to confirm a significant difference as a function of task.

Adult↗
PubMed1980

[Annoyance and loudness measurements in auditory fatigue and recovery under different sound exposures].

In the course of two experimental studies the individual evaluations of noise annoyance during sound exposure were compared with the experimental auditory fatigue (TTS) and the recovery functions. In both studies, in fourteen young men with normal hearing capacity, a temporary threshold shift was built up and then the subjects were submitted during the recovery period to different second-noise levels (narrow band noise and white noise). The loudness estimations and the evaluations of noise annoyance were scaled at the same points as TTS was measured. The poststimulatory recovery of hearing was significantly delayed, even at noise levels of 65 and 75 db. The evaluations of loudness and noise annoyance corresponded well with the recovery of hearing. In the first experiment, the mean values of noise annoyance ratings increased after 16 min of the second noise exposure, in spite of a further decrement of auditory fatigue. In the second experiment, the mean values of noise annoyance ratings remained almost unchanged during the secondary noise exposure. Regarding individual evaluations, however, the noise annoyance ratings of half of the subjects increased whereas the other half decreased. These results are interpreted as a habituation respectively a sensitization process.

Acoustic Stimulation↗
PubMed1975

Auditory fatigue: retrocochlear components.

Changes in auditory sensitivity were measured at the VII nerve, cochlear nucleus, and inferior colliculus after a fatiguing sound exposure. Losses in sensitivity progressively increased from peripheral to central auditory sites. The results suggest that there is a retrocochlear component to auditory fatigue when it is induced by low-level sounds of short duration.

Acoustic Stimulation↗
PubMed1979

The effect of the stapedius reflex on attenuation and poststimulatory auditory fatigue at different frequencies.

The effect of the stapedius reflex on attenuation was measured in patients with unilateral facial palsy (Bell's palsy) and stapedius muscle paralysis. Poststimulatory auditory fatigue was determined in the same patients and in a group of normal test subjects with a normal bilateral stapedius muscle function. The attenuation for a 0.5 kHz tone was found to begin at the reflex threshold, to increase about 7 dB per a 10 dB increase in the stimulus tone and to reach a maximum in the vicinity of 20 dB. When a 2.0 kHz tone was used, no attenuation of note appeared until the sound had risen to about 10 dB above reflex threshold. The attenuation reached a maximum at a little less than 10 dB. A properly functioning stapedius muscle significantly reduced poststimulatory auditory fatigue at 0.75 kHz while at 3.0 kHz, it could do so only if it was stimulated into action by low-frequency noise.

Acoustic Impedance Tests↗
PubMed2001

Origin of cubic difference tones generated by high-intensity stimuli: effect of ischemia and auditory fatigue on the gerbil cochlea.

Cubic difference tone (CDT) otoacoustic emissions are thought to arise from the feedback loop allowing outer hair cells to enhance the sensitivity and tuning of the organ of Corti. The existence of residual CDTs during complete cochlear ischemia is therefore disturbing. That stimulus intensities must exceed 50-60 dB SPL for residual CDTs to be recorded and for level notches to be present in CDT growth functions is often cited as evidence for a two-component, "active/passive" model: one component, the residual one, would originate from a passive, hardly vulnerable mechanism and thus be unsuitable for hearing screening purposes. This model was probed in gerbil ears after complete interruption of the cochlear blood flow. Cochlear potentials and CDTs were controlled simultaneously through continuous monitoring of CDT level and phase for 50 and 60 dB SPL stimuli and group-delay measurements. After a clear initial decay, CDT levels elicited at 60 dB SPL plateaued for several minutes at about 20 dB below initial level, and when early level notches were observed, CDT phase changes remained minor. The CDT group delays decreased by less than 30%. Later CDT level notches were associated with sharp phase reversals but the similarity between CDT characteristics before and after a notch was hardly consistent with a two-component interpretation. When mild sound overexposure (pure tone, 90-95 dB SPL, 15-30 min) had been performed prior to ischemia, little or no ischemic CDT came from the frequency bands where auditory fatigue had been detected (within 1 kHz), irrespective of the stimulus intensity. It suggests that instead of being passive, residual ischemic CDTs were vulnerable and produced according to a near-normal tonotopy by the same mechanisms that were sensitive to auditory fatigue. All the results lined up with a simple feedback model of cochlear function assuming a single CDT source related to mechano-electrical transduction in outer hair cells. More parsimonious than a two-component model, it posits that although early stages of ischemia dramatically impair the overall performance of the cochlea, the nonlinear mechanical stages responsible for the existence of CDTs keep working albeit at higher intensities.

Acoustic Stimulation↗
PubMed1980

Underestimation of auditory fatigue as measured by the compound action potential.

The action potential (AP), summating potential (SP), and cochlear microphonic (CM) were measured in rats in response either to clicks or pure tones prior to and following 3 min of exposure to pure tones at a level 5 db less than that which produced maximum CM. The ratio, in decibels, between pre-exposure and post-exposure potentials, for the same exposure and probe stimulus parameters, was taken as an index of decrement. The relative reduction in voltage resulting from the exposure was greater for the SP than for the AP when these potentials were elicited with 20-msec probe tone bursts between 70-80 db SPL having instantaneous rise times. However, for weaker probe levels within 20 db of that yielding AP potentials of 1 mu V, the AP and SP bpth exhibited similar losses. The CM and the click-evoked AP showed essentially no decrement. These results suggest that the SP might be a better indicator of noise-induced auditory decrement (fatigue?) than the CM. Theories of central auditory fatigue may be based on incorrect interpretations of previously published data obtained from cochlear and neural recordings.

Action Potentials↗
PubMedDate not supplied

Central auditory fatigue.

The results of this study, based on evoked responses and single-neuronal responses, reveal that there is a central involvement in auditory fatigue. In these experiments, cochlear potentials (microphonic and whole-nerve action potential) and inferior colliculus electrical responses were simultaneously obtained before and after excessive sound exposure. In general, sound exposure produced a greater reduction of the collicular evoked responses than of the cochlear microphonics and action potentials. Recordings from single neurons support the evoked-response findings.

Animals↗