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At least 19 recordsLinked to original sources
PubMed1994

[Abdominal pain in the differential diagnosis of acute abdomen-- common imitators of acute abdomen].

The authors analysed abdominal pains in diagnosis of acute abdomen, and the importance of that problem for the beginning of therapy in time. Here were elaborated the anamnestic, symptomatologic, diagnostic and differential diagnostic details, which make sure the recognition of acute abdomen and an early diagnosis of this state. The numerous data, rightly interpreted, enable an early diagnosis of acute abdomen, also by the general practitioner who is the first to meet those patients. We reported especially cases who imitate acute abdomen. These diseases are very important in differential diagnosis of acute abdomen. Both doctors, the general practitioner and the clinical one, have a big responsibility for an early recognition of the disease, in order to be able to do the in time and prognostically good surgical therapy.

Abdomen, Acute↗
PubMed1997

Gynecologic causes of the acute abdomen and the acute abdomen in pregnancy.

Evaluation of a female patient who presents with an acute abdomen must always consider surgical and gynecologic disorders. Laparoscopy and pelviscopy have had a major impact on the surgical approach in gynecology. Most acute abdomens can now be approached laparoscopically. Certain conditions that are discussed require the traditional laparotomy. Preservation of reproductive capability has a major impact on the wellness of a woman.

Abdomen, Acute↗
PubMed1989

The genetics of abnormal abdomen, incomplete abdomen, and bobbed in Drosophila buzzatii.

Five stocks of Drosophila buzzatii with superficially similar abdominal disruptions including partial tergite and sternite loss were isolated by inbreeding. Three of the stocks have indistinguishable phenotypes, the inheritance of which is maternally influenced. This phenotype and its mode of inheritance bear similarities with those of Abnormal abdomen in D. melanogaster. The phenotype in the fourth stock is slightly different and is due to a single autosomal recessive gene, which we denote incomplete abdomen. In the fifth stock the trait is limited to females, and in appearance and mode of inheritance resembles bobbed in D. melanogaster. Furthermore, only in this stock are rDNA deletions evident. The combined frequencies of the three types of abdominal aberration were found to be around 1% in several samples from wild and laboratory populations of D. buzzatii.

Abdomen↗
PubMed1986

The molecular through ecological genetics of abnormal abdomen. II. Ribosomal DNA polymorphism is associated with the abnormal abdomen syndrome in Drosophila mercatorum.

Restriction endonuclease cleavage analyses of cloned and genomic DNA samples indicate that the structure of the DNA encoding the large cytoplasmic RNAs (rDNAs) is altered in Drosophila mercatorum lines which exhibit an abnormal abdomen (aa) phenotype. In a majority of the rDNA repeat units from aa flies, the 28S coding sequence is interrupted by a large [5-6 kilobase pairs (kbp)] insert. A subclone containing this inserted DNA (ins 3) hybridizes primarily to rDNA-containing sequences in in situ and genomic blot hybridization experiments. Additionally, genomic nitrocellulose blot hybridization analyses show that ins- containing rDNA repeat units are clustered in a spontaneously arising aa mutant. This rDNA alteration in D. mercatorum flies with the aa phenotype more closely resembles the bobbed (bb) defect of D. hydei than the bb defect of D. melanogaster, which involves alterations in rDNA copy number. By analogy with the other Drosophila systems, we propose that the altered D. mercatorum rDNA repeat units are defective in rRNA production at a critical stage. The lowered levels of rRNA ultimately would limit the concentration of ribosomes needed to produce large quantities of a protein (in these cases, juvenile hormone esterase) needed for normal development.

Abdomen↗
PubMed1986

The molecular through ecological genetics of abnormal abdomen. III. Tissue-specific differential replication of ribosomal genes modulates the abnormal abdomen phenotype in Drosophila mercatorum.

The abnormal abdomen (aa) syndrome in Drosophila mercatorum is controlled by two major X-linked genetic elements. We have previously shown that the major X-linked element of aa is associated with the presence of large inserts in the 28S gene of the ribosomal RNA (rDNA) genes. We show that, in polytene tissue of wild-type D. mercatorum, the uninterrupted rDNA repeats are overreplicated relative to interrupted repeats. Uninterrupted rDNA repeats are also overreplicated in polytene tissue of hybrid larval offspring from wild-type and aa parents. This overreplication of uninterrupted repeats is not observed in diploid tissues of wild-type hybrids (of wild-type and aa parents) and homozygous aa larvae or in polytene tissue of aa larvae. Furthermore, molecular analysis of an aa line that has reverted to the wild type indicates that the reversion phenomenon is associated with the ability to overreplicate uninterrupted rDNA repeats in polytene tissues. The patterns of differential replication of rDNA genes in wild-type hybrids and aa larvae of D. mercatorum offer a possible mechanism for the tissue-specific control of the aa phenotype and suggest that the molecular basis for the second X-linked genetic element of aa is involved in the control of differential replication in polytene tissues.

Abdomen↗
PubMed1981

The acute abdomen. Part II: Acute abdomen arising from vascular disorders in the elderly.

The frequency of the "acute abdomen" has increased with the ageing of the population. In this chapter, we have examined syndromes of primary vascular origin, caused by vascular dysfunction, such as alterations in the vascular walls or haemodynamic disorders. We have left aside secondary vascular problems such as strangulation, obstruction or compression of vessels by tumours, which are easier to treat and diagnose and have a better prognosis. The symptoms of acute ischaemia caused by intrinsic vascular deficiency are not specific and clinical tests are untrustworthy. Despite progress in our knowledge of splanchnic haemodynamics, in radiology, vascular surgery and endoscopy, the prognosis remains poor. In view of the cataclysmic nature of the haemorrhage or the damage to the intestinal walls which evolves swiftly towards gangrenous necrosis, very rapid diagnosis of any of these various syndromes is essential if the patient is to be saved.

Abdomen, Acute↗