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At least 1,765 records · Page 98Linked to original sources
PubMed1984

Johne's disease in sheep.

Johne's disease was identified in sheep flocks on 6 farms in one area of the central tablelands of New South Wales. The sources of infection were not determined and no common source of all infections was identified. The disease was not diagnosed in cattle that were run on 4 of the farms.

Age Factors↗
PubMed1984

Serum biochemical changes in calves with Johne's disease.

Calves clinically affected with experimentally induced Johne's disease exhibited elevation of caeruloplasmin oxidase activity, and marked depression of alpha-mannosidase activity during the period when clinical signs of the disease were most prominent. Changes in serum copper levels and alkaline phosphatase activity were closely correlated with the elevation of caeruloplasmin oxidase activity, and depression of alpha-mannosidase activity. The pattern of these changes was similar to nutritional and metabolic changes described previously in acute infectious conditions in man and animals.

Alkaline Phosphatase↗
PubMed1996

Aggregate testing for the evaluation of Johne's disease herd status.

This paper examines methods for evaluating herd Johne's disease status that could be used in a survey of the cattle industry. Emphasis is placed on aggregate testing, a process whereby a random sample of cattle from a herd is assessed using an imperfect test, such as an ELISA for detecting antibody in serum. Important aggregate test parameters discussed include: sample size, herd-level sensitivity, herd-level specificity, the number of reactors used for declaring a positive herd result, and the expected within-herd prevalence of disease. Aggregate testing may be useful for several livestock diseases. However, problems arise when it is applied to Johne's disease because of the poor sensitivity of the available diagnostic tests, the low within herd prevalence of infection, and clustering of false positives within a herd.

Animals↗