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At least 1,657 records · Page 92Linked to original sources
PubMed1984

Neurovascular decompression for hemifacial spasm.

Results of 23 cases of facial spasm operated on by posterior fossa exploration were reviewed. In all cases, but one, vessels were found to impinge on the facial nerve root exit zone from the brain stem. All cases in which the facial nerve had been liberated from the vessel had satisfactory results and there was no recurrence of spasm. Significant sensorineural hearing loss occurred postoperatively in 11 cases. The hearing loss greatly improved within a month and persisted as permanent loss greater than 10 dB in 6 cases (26.0%). The complication has been lessened by approaching to the nerve from caudal direction and by retracting the cerebellum to oblique direction to the acoustic nerve. Microvascular decompression is the best procedure available for hemifacial spasm.

Adult↗
PubMed1983

[Strategy for the prevention and treatment of post-paralytic facial syncinesis].

The existence of facial heimspasm and post-paralytic syncinesia is in general interpreted as the result of aberrant reinnervation following a Bell's palsy. In a certain number of cases, electrophysiological tests reveal synaptic abnormalities in the facial nucleus. These findings must be taken into consideration, together with the possibility of ephapsic stimulation of the proximal part of the facial nerve, when explaining the regression of syncinesia which is found in certain patients during the reinnervation phase. Prevension is based principally upon the quality of treatment and the rapidity with which both electrophysiological testing is undertaken and treatment started. It is important to avoid treatment aimed at accelerating reinnervation (neuronotrophic factors, dielectrolysis, etc . . . The process should take place naturally. Electrical stimulation, administered under conditions of choice of current on the basis of the lesion, experimentally prevents dissemination of reinnervation. Repeated evaluation of possible diffusion of the blink reflex can be used to detect sub-clinical stages of progression to syncinesia. It would seem necessary to review from a particular standpoint the organised programming of physiotherapy and its association with biofeedback-EMG techniques. If signs of syncinesia develop, appropriate physiotherapy, biofeedback-EMG techniques and contralateral strio-motor electrotherapy combined with sedative and anti-paroxysmal therapy should be started.

Electric Stimulation Therapy↗
PubMed1983

Symptomatic hemifacial spasm: case report and review of literature.

The purpose of this article is to discuss a case of hemifacial spasm due to a fusiform basilar aneurysm. The hemifacial spasm was successfully treated by decompression of the facial nerve in the facial canal 14 months before death due to aneurysmal rupture. The literature on symptomatic hemifacial spasm is reviewed, the pathophysiology discussed and the therapeutic options presented. Since the pathophysiology is poorly understood, conservatism is recommended.

Basilar Artery↗
PubMed1984

Blepharospasm--oromandibular dystonia (Meige's syndrome) misdiagnosed as secondary hemifacial spasm.

Orofacial dyskinesia (Meige's syndrome) is a rare clinical entity. This disorder was first described in 1910 by Henry Meige as "spasm facial median," a disabling spasm of the facial musculature which had frequently been misdiagnosed and inappropriately treated. This report concerns a patient who presented with tonic hemifacial spasm twenty-two years after contracting Bell's palsy. The condition was initially thought to be secondary to faulty regeneration of the facial nerve until Meige's syndrome became fully manifested over a period of two years. The diagnostic characteristics as well as the forms of treatment for this unusual disorder are presented.

Blepharospasm↗
PubMed1984

[A case of paroxysmal tinnitus and nystagmus accompanied by facial spasm].

Trigeminal neuralgia and facial spasm are usually caused by cross vascular compression of the trigeminal root entry zone and facial nerve exit zone. A similar mechanism is believed to affect the acoustic nerve as well, giving rise to tinnitus and nystagmus. We present one case of paroxysmal tinnitus and nystagmus, accompanied by facial spasm, and discuss the mechanism and the significance of the neurotological examination.

Arteries↗
PubMed1982

[Centronuclear myopathy. Complete review of the literature apropos of a case].

A 19 years old woman suffered from claw feet and mild disorders of gait since infancy. Physical examination disclosed parseris of pelvis girdle, diffuse areflexia, mild dorsolumbar scoliosis, claw feet and a high arched palate. The facial muscles and cranial nerves were not involved. Serum enzymes were normal, EMG was consistent with a myopathy. Family examination and laboratory data were normal. On muscle biopsy, there were many muscle fibers with central nuclei and clear perinuclear areas, and a varying increase of adipose tissue. Histochemical studies showed type I fiber predominance and atrophy ; the central part of fibers was not stained by ATPase reactions but was strongly reactive with phosphorylase and oxidative stains. By electromicroscopy, central nuclei were separated by strands of glycogen ; there were no myofibrillar abnormalities. From a literature review, there is a large heterogeneity in genetic, clinical and pathological findings. Any attempt to class the different kinds of this disease is difficult, other than by the age of onset : --Early onset cases are characterized by neonatal hypotonia, severe disability and sometimes early death by pulmonary involvement. --Infantile and late onset cases have slower evolution. The nature of the disease remains unknown.

Adult↗
PubMedDate not supplied

Hemifacial spasm: report of nine cases treated by posterior fossa microsurgical decompression.

Nine cases of hemifacial spasm have been treated, in our department until 1979, by posterior fossa microsurgical decompression, as described by Jannetta, without mortality or significant postoperative complications. A blood vessel was found crossing or looping the facial nerve in 8 cases. The relief of hemifacial spasm was immediate and complete in 7 cases, and partial in 1 case. This technique seems to be an effective and safe treatment, in this particular disease.

Adult↗