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[Scorpion stings].
This article begins by describing how Iberian Peninsula scorpion bites poison humans. Then the only potentially dangerous scorpion species in the peninsula is identified. Other types of scorpions or those which inhabit other continents are not included. Once identified, the author describes the characteristics and effects on the symptoms of this scorpion bite and how to treat and handle a victim, both at the moment when the victim is bitten and in hospitals.
[What should be done after bee or wasp stings?].
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The case for the use of verapamil in alarming Chironex stings.
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Temporal arteritis after Hymenoptera sting.
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[Clinical and epidemiological aspects of scorpion sting in a hyperendemic area].
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Myocarditis from scorpion sting. A clinical and electrocardiographic study of 50 cases.
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A piece of my mind: sometimes dying still stings.
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The Sting procedure and its complications: sonographic evaluation.
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Parasitoid wasp sting: a cocktail of GABA, taurine, and beta-alanine opens chloride channels for central synaptic block and transient paralysis of a cockroach host.
The wasp Ampulex compressa injects venom directly into the prothoracic ganglion of its cockroach host to induce a transient paralysis of the front legs. To identify the biochemical basis for this paralysis, we separated venom components according to molecular size and tested fractions for inhibition of synaptic transmission at the cockroach cercal-giant synapse. Only fractions in the low molecular weight range (<2 kDa) caused synaptic block. Dabsylation of venom components and analysis by HPLC and MALDI-TOF-MS revealed high levels of GABA (25 mM), and its receptor agonists beta-alanine (18 mM), and taurine (9 mM) in the active fractions. Each component produces transient block of synaptic transmission at the cercal-giant synapse and block of efferent motor output from the prothoracic ganglion, which mimics effects produced by injection of whole venom. Whole venom evokes picrotoxin-sensitive chloride currents in cockroach central neurons, consistent with a GABAergic action. Together these data demonstrate that Ampulex utilizes GABAergic chloride channel activation as a strategy for central synaptic block to induce transient and focal leg paralysis in its host.
[Stinging nettle in medicine and nutrition].
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The sting of salt on an old, but open, wound--is Na(+) the cause of mitochondrial and myocardial injury during ischemia/reperfusion?
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[Skin reactions from stings of the gnat Culicoides minutissimus Zett].
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Oh death, where is thy sting? Reflections on dealing with dying patients.
This paper examines the reactions of physicians and other health professionals when they become involved in decisions about the death of their patients... Four traditional views of death are explored. The problem that physicians have in helping patients die (be it by hastening death through pain control, assisting patients in suicide or by more active means) is analyzed. Physicians, in dealing with such patients, must be mindful of their own, and their patients beliefs as well as mindful of the community in which such dying takes place. They must try to reconcile these often divergent views but can neither paternalistically deny patients their rational will, hide themselves behind an appeal to the law or go against their own deeply held moral views. When such views cannot be reconciled, compassionate transfer to a more compatible physician may be necessary.
Insect venoms: identification of dopamine and noradrenaline in wasp and bee stings.
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