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At least 1,639 records · Page 91Linked to original sources
PubMed1991

[Changes in serotonin-containing EC-cells in the lamina epithelialis mucosae of the small intestine in young children with growth disorders caused by intestinal diseases].

Duodenal jejunal biopsy material from children with growth retardation due to enteric disease was investigated. The disease dependent mucosa types were classified into three groups according to Shmerling (1970). 5-hydroxytryptamine was demonstrated with an impregnation technique and histochemically, as well, in EC-cells in the crypt epithelium. The EC-cell content in specimens with subtotal villus atrophy (type III) was higher significantly compared to normal mucosa (type I). The findings were discussed with regard to the possible role of 5-hydroxytryptamine in the pathogenesis of the growth retardation due to enteric disease in children.

Biopsy↗
PubMedDate not supplied

[Small intestine intubation during operative treatment of primary or recurring intestinal obstructions (author's transl)].

The author reports eight cases of recurrent obstruction treated by intubation of the small bowel. This technic, possible in any cases, avoids other long and often difficult technics. The tube used in those cases intends to maintain a normal circumference to the small bowel on all its length and to secure its permanent evacuation till it begins to function normally.

Adult↗
PubMed1985

[Genetic problems of hereditary polyposis of the large intestine. I. An analysis of the interrelation with intestinal cancer].

An attempt has been made to estimate the correlation of oncological illnesses in the families within the limits of segregational and correlational analyses, considering the fact of accumulation in the families of probands with autosomal-dominant polyposes of colon of the cases of primary (without preceding polyposes) colon cancer. Significant correlation was found between the hereditary polyposes and primary colon cancer in the families studied. Based on these data, a hypothesis is discussed of the main gene and its modifications on the phenotypical version in the form of primary benign (polyposes) and malignant (cancer) pathology. No significant correlation was found between colon polyposes and cancer of stomach in the families.

Genetic Techniques↗