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Results for “SHEEP DISEASES”

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At least 163 records · Page 9Linked to original sources
PubMed1977

White muscle disease of sheep and associated tissue selenium levels in Western Australia.

White Muscle Disease of sheep occurs in Western Australia as distinct syndromes in lambs and weaner sheep. Mean liver selenium concentrations of lambs and weaners with WMD and unaffected by WMD were 0.19 and 0.46 ppm, and 0.20 and 0.41 ppm (dry weight) respectively. There was considerable overlap between levels suggesting that selenium status was not the only factor involved in the development of WMD.

Animals↗
PubMed1985

Border disease of sheep: spinal cord morphometry.

Semi-automated planimetry was used to determine cross-sectional areas of spinal grey and white matter by direct microscopy of paraffin sections of spinal cord from Border disease (BD) and normal lambs at segments C4, C8, T6 and L4. Spinal cord cross-sectional area was significantly reduced in cases of BD produced by either intramuscular or intranasal inoculation of ewes in the first half of pregnancy with Weybridge strains of BD virus. The reduction was apparent at all 4 levels and in both grey and white matter, though the white matter was the more severely affected. Spinal cord areas tended to be smaller in the progeny of ewes exposed earlier in gestation or to larger doses of virus, but these differences were not statistically significant.

Animals↗
PubMed1995

An outbreak of Aujeszky's disease in sheep in Northern Ireland.

An outbreak of Aujeszky's disease occurred in a flock of 160 ewes which had been shorn and housed adjacent to pigs which were excreting Aujeszky's disease virus. The 29 affected sheep developed either the classical 'mad itch' signs associated with Aujeszky's disease in ruminants or signs of encephalitis. Aujeszky's disease virus was isolated from the central nervous system of the affected ewes. Five farm cats also became ill and died. Histological lesions of viral encephalitis were observed in one of the cats and Aujeszky's disease virus was isolated from its brain.

Animals↗
PubMed2002

Ovine progressive pneumonia (Maedi-Visna): an emerging respiratory disease of sheep in Ethiopia.

A serological study was done to assess the role of Maedi-Visna (MV) infection in sheep from flocks with high respiratory tract disease morbidity in Ethiopia. Of 105 sheep examined from central Ethiopia 78 (74%) were positive for MV-infection. However, antibodies to the virus were not detected in 48 sheep and 70 goats from elsewhere in Ethiopia. The infection was detected in all breeds of sheep examined (Awassi, Hampshire, Corriedale, indigenous Menz breeds and their crosses) but with a significant breed difference (chi 2 = 20, p < 0.001) varying from 48% in imported Awassi sheep to 92% in the indigenous Menz sheep. This suggests that Menz sheep are more susceptible to infection, which may support the observation of a higher incidence of clinical disease in these sheep compared to exotic breeds and their crosses. It also supports recent studies indicating that MV is becoming one of the most important respiratory tract diseases in sheep in central Ethiopia. Our findings indicate that MV was introduced into Ethiopia via sheep imported into the central highlands and that it now constitutes an important emerging disease is discussed. Measures to control the disease are suggested.

Animals↗
PubMed1981

Fatal hemolytic disease in sheep attributed to Leptospira interrogans serotype hardjo infection.

An outbreak of acute hemolytic disease in a large flock of sheep resulted in a mortality of approximately 5% in lambs. A diagnosis of Leptospira interrogans serotype hardjo infection was established on the basis of the presence of leptospires in the urine of one lamb, serologic findings in a guinea pig inoculated with this urine, L. hardjo titers in lambs, and pathologic findings. Prominent necropsy findings included severe icterus, thin watery blood, dark red to black kidneys and severe hemoglobinuria.

Animals↗
PubMed1991

Border disease of sheep--aspects for diagnostic and epidemiologic consideration.

Border Disease (BD) is a condition of newborn sheep that results from congenital infection by a non-cytopathic pestivirus, occurring during the first half of gestation. The variations in expression of the virus directly relate to the age of the fetus at the time of infection. There are four distinct disease syndromes: (1) early embryonic death, (2) abortion and stillbirth, (3) birth of lambs with malformations, and (4) birth of small, weak lambs, lacking characteristic clinical signs, but bearing features of immunosuppression. In the newborn, the BD virus may be recovered from all tissues and teratogenic lesions are found in the endocrine, nervous, skeletal, integumentary and immune systems. These effects of virus infection are manifest in the clinical signs characteristic of the disease, such as tremors, ataxia, hairy birthcoat, low birth weight, facial bone malformations, short-boxy stature, and eye abnormalities. The consequences of the BD compromised immune system is an increased susceptibility to infection, a failure to produce specific antibody to BD virus, and an inability to clear the virus; features characteristic of the immuno-tolerant state. The lifelong shedding and persistence of virus is of epidemiologic importance. The persistently infected BD ewe remains a source of infection for the flock both through horizontal transmission (virus shedding) and congenital transmission (a persistently infected ewe will always bear a BD lamb). Detection of persistently infected individuals within a flock is difficult: clinical signs abate with time and most frequently no antibody to BD is produced.

Animals↗
PubMed1977

Histological and histochemical studies of fetal skin follicles in Border disease of sheep.

The structure of the epidermis and dermis, and the distribution of reticulin, glycogen, lipid and sulphydryl groups in the skin have been examined in control and Border disease affected sheep fetuses at 95 and 115 days' gestation. In Border disease, there was a temporary retardation of development of the epidermis and skin follicles at 95 days. There was no difference in the structure of the dermis between control and experimental fetuses at either age. No evidence was found to suggest that the keratogenous process was abnormal in Border disease, but the sulphydryl zone of the primary fibres of affected fetuses was situated nearer the skin surface than in control fetuses. This may have been a consequence of the larger size of primary follices in Border disease. Glycogen present in the undifferentiated epidermis of both groups at 95 days' gestation was absent from the stratified squamous epithelium of the 115 days's fetuses. Glycogen and lipid droplets were present in the outer root sheath of primary follicles of all fetuses examined, but their significance is unknown.

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PubMed2005

Malignant catarrhal fever-like disease in sheep after intranasal inoculation with ovine herpesvirus-2.

A malignant catarrhal fever (MCF)-like disease was induced experimentally in 3 sheep after aerosol inoculation with ovine herpesvirus-2 (OvHV-2). Each of 3 OvHV-2-negative sheep was nebulized with 2 ml of nasal secretions containing approximately 3.07 X 10(9) OvHV-2 DNA copies from a sheep experiencing an intensive viral-shedding episode. Ovine herpesvirus-2 DNA became detectable by polymerase chain reaction in the peripheral blood leukocytes of all 3 sheep within 3 days, and all 3 seroconverted between 6 and 8 days postinfection (PI). The sheep developed clinical signs, with copious mucopurulent nasal discharge and fever around 14 days PI. One of the 3 clinically affected sheep was euthanized at 18 days PI. Major lesions at necropsy were multifocal linear erosions and ulcers in mucosa of the cheeks, tongue, pharynx, and proximal esophagus and mild disseminated pneumonia. Microscopically, there was extensive moderate superficial histiocytic-lymphocytic rhinitis with epithelial dissociation and degeneration. Moderate multifocal histiocytic bronchointerstitial pneumonia was associated with loss of terminal bronchiolar epithelium. Lymphocytic vasculitis was present only in the lung. The remaining 2 sheep recovered clinically, approximately 25 days PI. The study revealed that clinical signs and lesions resembling MCF can develop when uninfected sheep are exposed to a high dose of aerosolized OvHV-2.

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