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At least 163 records · Page 9Linked to original sources
PubMed1985

Spontaneous regeneration of the parotid salivary gland following juvenile recurrent parotitis.

Sialograms of an 18-year-old female patient known to suffer from juvenile recurrent parotitis (JRP) disclosed the destructive glandular changes typical of the disease. During the 10 years subsequent to the sialographic examination, the patient did not experience any further attacks of JRP. Sialograms carried out when the patient was 28 years of age demonstrated a normal gland. This case illustrates that a damaged parotid gland is capable of regeneration following JRP.

Adolescent↗
PubMed1986

Juvenile recurrent parotitis: clinicoradiologic follow-up study and the beneficial effect of sialography.

In twenty-two children juvenile recurrent parotitis (JRP) was diagnosed on the basis of age at onset, duration of parotid swelling episodes, frequency of attacks, lack of pus formation, and typical sialograms. Age at onset and severity of the disease varied widely. There was a high incidence of upper respiratory tract infection, and in some cases a familial history of JRP was noted. The sialograms revealed acinar and ductal atrophy and severe sialectasis. A dominant feature was impaired glandular function. The follow-up period was notable for a striking decrease in the number of incidents of glandular swelling regardless of the patient's age at the time of his or her first visit to our clinic. Since drug treatment was administered prior to our first examination and no medicaments were prescribed by us, the improvement in the clinical status of the patients is attributed to the sialographic procedures.

Child↗
PubMed1991

Acute suppurative parotitis: a forgotten cause of upper airway obstruction.

A patient had threatened upper airway obstruction as a result of bilateral acute suppurative parotitis. Because of the rarity of this condition in recent times, accurate diagnosis was delayed and fiber-optic intubation in the intensive care unit was required to safeguard the airway. Management is described with reference to the surgical anatomy and previously reported cases.

Acute Disease↗
PubMed1992

Chronic obstructive parotitis. Report of ninety-two cases.

Clinical, sialographic, and sequential quantitative scintigraphic characteristics of 92 cases of chronic obstructive parotitis (in 117 diseased parotid glands) are reported, and the treatment investigated. The cardinal feature of this disease is obstruction or retarded salivary flow resulting in recurrent swellings and retrograde infection. The main characteristic seen in sialograms is irregular dilation of the main and branching ducts. On the basis of sialographic findings, the ducts are classified into four types. This classification reflects the severity of the disease and can also be used as a guide to treatment.

Adolescent↗
PubMed1995

Acute suppurative parotitis and parotid abscess in children.

Acute suppurative parotitis and parotid gland abscess are infrequently encountered among children. Fourteen children were diagnosed in the Sheba Medical Center as suffering from these infections and their clinical features are described. Discussion reveals neither significant differences nor predominating factors to differentiate between these two entities.

Abscess↗
PubMed1976

Priapism: an unusual complication of parotitis without orchitis.

A 10-year-old white boy is described in whom priapism developed as a complication of parotitis without orchitis. Medical study failed to reveal any other etiology. The syndrome was assumed to be related to a mumps virus. Treatment by corpora cavernosa-spongiosum shunt relieved the acute symptoms as well as preserved the patient's ability to have an erection.

Child↗
PubMed1999

Acute suppurative parotitis with spread to the deep neck spaces.

This report describes the case of an elderly, diabetic woman who became dehydrated and developed acute suppurative parotitis, which caused marked swelling of her left face and neck. The parotid infection also extended by continuity into the lateral pharyngeal space and contiguous deep neck spaces, causing airway-threatening, extensive inflammation and swelling of the epiglottis and parapharyngeal soft tissues. The differential diagnosis and diagnostic rationale is discussed. The anatomy of the stylomandibular area is reviewed to explain how infection of the parotid can spread to the pharynx.

Acute Disease↗
PubMed1992

Sialographic changes related to clinical and pathologic findings in chronic obstructive parotitis.

This paper presents the results from a study of 23 patients with chronic obstructive parotitis (COP) in whom the sialographic changes were related to the clinical and histopathologic features. The sialographic changes were divided into four types. The stimulated parotid flow rate (SPFR) was normal in sialography Type I patients, while SPFR was significantly reduced in Types II, III, and IV patients, as compared with healthy persons. The changes observed by light microscopy were irregular dilation and chronic inflammation of main and branching ducts with saliva deposits in the enlarged luminal system. Ultrastructural changes included degeneration of ductal epithelial cells, marked proliferation of periductal fibrous tissue, and moderately electron-dense deposits in the lumen of the dilated peripheral ducts. The extent of the histopathologic changes largely paralleled the severity of the sialographic changes. These findings indicate that COP is a relatively common inflammatory disease and that the sialographic classification reflects the severity of the disease.

Adult↗
PubMed2004

[Neonatal acute suppurative parotitis].

Acute suppurative parotitis is highly uncommon in neonates. Approximately 100 cases have been reported in the literature. Dehydration and prematurity are important predisposing factors. Diagnosis is based on clinical signs. White blood cell count and parotid ultrasonography are useful. The most commonly isolated causative organism is Staphylococcus aureus. Initial treatment consists of antistaphylococcal empiric antimicrobial therapy for 7-10 days. The prognosis is good. The illness is not usually associated with recurrences. We describe two 12-day-old newborn infants who presented with fever, unilateral swelling of the parotid region with erythema, warmness and pain, and purulent discharge from Stensen's duct. We also provide a literature review.

Acute Disease↗
PubMed1999

Acute pancreatitis and parotitis induced by methimazole in a patient with Graves' disease.

A wide variety of adverse effects of methimazole (MMI) have been reported. Here we report a new MMI-induced disorder, acute pancreatitis and parotitis. Three weeks after a woman started MMI treatment for Graves' disease, she developed a high fever, painful parotid swelling and dull pain in the upper abdomen with elevation of the serum levels of salivary and pancreatic enzymes. These abnormalities disappeared soon after the withdrawal of MMI. However, the same abnormalities were rapidly provoked when MMI was reintroduced. Marked increases in the leucocyte count and CRP were also observed during these episodes. The possible mechanisms of MMI-induced pancreatitis/parotitis are discussed.

Acute Disease↗
PubMed1992

[Chronic recurrent parotitis in childhood].

Juvenile recurrent parotitis (j.r.p.) is distinguished from the adult form by the course of the disease, therapeutic considerations and prognosis. Children suffering from j.r.p. are between 2 and 15 years of age; the male: female ratio is 1.5:1. In the present paper the author describes the different pathogenic theories of j.r.p. in accordance with the relevant literature. Besides malformation of the glandular duct like stenosis and ectasis, functional factors like the character of salivary secretion are suspected as being responsible for the disease. Other authors assume that a viral genesis, allergic factors, a physiological immaturity of the immune response, or family history may be the causes. In the evaluation of children with j.r.p. the patients' history with recurrent swelling episodes of the parotid gland(s) followed by quiescent periods is indicative. Further procedures like ultrasonography, sialography and MRI are discussed according to their diagnostic value. Therapeutic possibilities include antibiotics in the acute stage of the disease as well as parotidectomy in severe cases. Radiotherapy cannot be recommended because of its side effects (facial dysplasia, tumour induction). In most cases, the disease ends at puberty.

Child↗
PubMed1995

[Suppurative parotitis as a sequela of infection with Salmonella enteritidis].

Extraintestinal manifestations of Salmonella infection are rare events and occur primarily in immunocompromised hosts. We report on a 69-year old male patient hospitalized for absceding suppurative parotitis. From the swab taken on incision of the abscess, Salmonella enteritidis was cultivated. There was no preceding gastrointestinal infection in our patient. Risk factors in our patient included a poorly controlled non-insulin-dependent diabetes mellitus, chronic alcohol abuse, and advanced age. Quinolones are the therapy of choice in extraintestinal salmonellosis. On ciprofloxacin perorally, the patient showed rapid recovery.

Abscess↗
PubMed1990

[Acute purulent parotitis as a sequela of alkylphosphate (E 605) poisoning].

Acute suppurative parotitis is a possible consequence of poisoning by organophosphates (E 605). The endogenous accumulation of acetylcholine results--as in the acute pancreatitis caused by poisoning with organophosphates--in massive hyperemia and an increased secretory activity with leakage of saliva into the tissue, in an activation of the kallikrein-kinin system, the phospholipase A2, and, ultimately, in toxic tissue lesions caused by lysolecithin and the superoxide-radical anion. In spite of antibiotic therapy this is followed by bacterial inflammation. Treatment consists in the administration of aprotinin (Trasylol) and corticosteroids. In the case of abscess formation surgical drainage is necessary.

Acute Disease↗
PubMed1987

Facial paralysis in acute parotitis.

We report on a 12-year-old boy who developed facial paralysis secondary to acute parotitis. Awareness of this rare complication is emphasised. Lower motor neuron lesion of the facial nerve due to parotid enlargement is extremely rare in the absence of underlying neoplasia.

Acute Disease↗
PubMed2005

Extra-cranial metastasis of glioblastoma multiforme presenting as acute parotitis.

We present an unusual case of extracranial metastasis of glioblastoma multiforme (GBM) to the parotid gland and cervical lymph nodes. The patient had previously undergone two craniotomies to debulk a left frontal GBM, followed by radiotherapy. After the second craniotomy, while waiting for chemotherapy, the patient was re-admitted with a short history of a painful swelling of his left parotid gland. The initial diagnosis was infective parotitis; however, as there was no improvement with broad-spectrum antibiotics, CT was undertaken, which revealed a mass in the parotid gland with a necrotic centre and enlarged cervical lymph nodes. Parotid gland biopsy revealed a parotid GBM metastasis. This case illustrates how GBM behaves in an aggressive manner even outside the CNS. A brief review of the literature and of the theories, which might explain the extra-neural metastasis of this tumour is also presented.

Acute Disease↗
PubMed2000

Acute parotitis due to dengue virus.

Acute bilateral parotitis is a common clinical feature of various infectious and autoimmune, metabolic, and drug-related conditions. We describe a unique case of bilateral inflammatory enlargement of the parotid glands in an immunocompetent patient with dengue fever. Evidence of dengue virus in the saliva is also provided for the first time.

Acute Disease↗