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[Acute psychoses caused by digitalis poisoning].

The course of illness in a 76-year-old woman with a history of dyspnea being treated with medigoxin, 0.1 mg daily, is presented. Five days before the symptoms of acute psychosis developed she had used medigoxin, 0.2 mg per day. On the fifth day of that therapy, the patient developed acute psychosis and was admitted to the Department of Psychiatry. An electrocardiogram recorded monomorphous ventricular premature beats at a frequency of 50%, the S-T segment depression of the anterolateral location with the diphasic T-wave and the corrected Q-T interval of 0.377 to 0.355 seconds. Five days after the discontinuation of medigoxin therapy, serum digoxin concentration was 2.2 nmol/L, out of which follows that two days after digitalis was stopped it was 4.0 nmol/L and that concentration was probably higher at the moment when acute psychosis developed. The patient was free of symptoms of acute psychosis after a single promazine dose and discontinuation of medigoxin.

Acute Disease↗
PubMed1991

[Scopolamine poisoning as a cause of acute paranoid hallucinatory psychoses].

In 1988 two patients intoxicated with scopolamine were treated in the department of psychiatry. Clinically an acute paranoid-hallucinatory psychosis was to be observed. Similar cases were reported from other parts of the country, partly proved by toxicological findings. In the cases seen by us scopolamine was applicated secretly in drinks and food with criminal intention. Diagnostic, therapeutic and historical aspects of intoxication with scopolamine are discussed.

Adult↗
PubMed1989

[EEG findings in cortisone psychoses--a retrospective study in 10 patients].

The EEG of 10 patients, suffering from cortisone induced transit syndromes according to H.H. Wieck were studied retrospectively. Four patients with mild forms of transit syndromes and acute onset (appearance of symptoms within one month) showed normal EEG. EEG-alterations (i.e. parenrhythms and aidiorhythms according to H. Penin) were found in 5 patients, suffering from a severe transit syndrome with peracute onset (appearance of symptoms within 7 days). Their EEG became normal after remission of this severe transit syndrome. EEG-alterations were not influenced by the administered cortisone dosage. No epileptic seizures nor epileptic patterns in EEG were seen while under cortisone treatment. The data indicate a good correlation between syndrom severity of cortisone induced transit syndromes and the appearance of pathological EEG-patterns.

Adult↗
PubMed1989

[Acute and subacute organic psychoses with altered consciousness. Attempt at classifying severity].

We have retrospectively investigated 71 patients with ICD-9 diagnoses 293.0 or 293.1, who were treated in our clinic in 1987, with respect to the maximum severity of altered consciousness. On the basis of an available neurological grading into "clouding of consciousness", "confusional state", "delirium", "stupor", and "coma", and from knowledge of the underlying somatic disease, we have developed prognostic criteria: Disorders of heart and circulation associated with acute organic mental disorder (with altered consciousness) carry a fair prognosis for recovery of the mental disorder in approximately two-thirds of all patients affected, even in advanced age. The same statement is valid with respect to drug-induced acute organic mental disorders, whereas most cases of cerebrovascular disorders with mental disturbances proceed to chronic forms of organic mental disorder. We believe that the clinical descriptive terms referred to above allow easy grading of states of acutely/subacutely altered consciousness, thereby providing prognostic criteria for the course of the disease.

Adult↗