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[Role of catecholamine metabolism disorder in the pathogenesis of botulism].
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[Differential diagnosis of botulism].
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[Mechanism of the change in electrolyte and carbohydrate metabolism in the submandibular salivary gland in experimental botulism in rats].
Content of lactic and pyruvic acids, glycogen, sodium and potassium ions are well as the total LDH activity and its isoenzyme spectrum were not distinctly altered, when secretion of salivary glands was inhibited by administration of botulinic toxin into rats (within 48 hrs after the toxin treatment). But within 14 days the following alterations were observed in the impaired salivary glands: decrease in content of glycogen and potassium, increase in concentration of lactic and pyruvic acids, elevation in total LDH activity and change in its isoenzyme spectrum (increase in content of the fractions responsible for regulation of anaerobic metabolism).
Honey, infant botulism and the sudden infant death syndrome.
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[Mechanism of the contractile function of masseter muscles in experimental rat botulism].
Injection of the sublethal dose of botulinic toxin into one of the masseter muscles produces muscle weakness and impaired occlusion. The membrane polarization rate and action potential decrease primarily in high polarized muscle fibers of the injured muscle. Meanwhile the function of low polarized fibers does not undergo any significant changes.
[Botulism in Amur Province].
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[Various problems of clinical course, diagnosis and therapeutic tactics in botulism].
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[Characteristics of the resuscitation period in severe forms of botulism].
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[Mechanism of disruption of histamine metabolism in salivary glands in experimental botulism].
Content of histamine was increased in submaxillary salivary gland of rats in generalized and local forms of botulinic intoxication. Permeability of the histohematic barrier for blood serum proteins was not altered in the salivary gland. Content of histamine was normalized after treatment with pilocarpine. Botulinic intoxication was apparently responsible for short-term and unspecific alterations of histamine content in submaxillary salivary gland.
[Mechanism of changes in glycogen concentration and lactate dehydrogenase activity in respiratory muscles in rats with experimental botulism].
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A case of infant botulism.
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[A familial case of botulism with severe toxemia].
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[The risk of botulism in Mexico. 1st confirmed case].
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