Thin-layer chromatographic detection of barbiturates, narcotics, and amphetamines in urine of patients receiving psychotropic drugs.
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Amphetamine↗
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Results for “Narcotics”
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Addiction problems in the medical use of narcotic analgesics.
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Analgesics↗
Effects of some narcotics and antagonists on synaptosomal 3 H-norepinephrine uptake.
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Animals↗
Effects of narcotic antagonists on fluid intake in the rat.
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Amphetamine↗
Stereospecific increase by narcotic antagonists of evoked acetylcholine output in guinea-pig ileum.
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Acetylcholine↗
On the use of tryptic digestion to localize narcotic binding material.
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Animals↗
Investigations on drug produced and subjectively experienced discriminative stimuli. I. The fentanyl cue, a tool to investigate subjectively experience narcotic drug actions.
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Animals↗
Investigations on drug produced and subjectively experienced discriminative stimuli. 2. Loperamide, an antidiarrheal devoid of narcotic cue producing actions.
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Administration, Oral↗
Long-term narcotic exposure reduces caudate cyclic nucleotide levels, protein phosphorylation and tyrosine hydroxylase activities.
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Animals↗
Stereospecific aversive property of narcotic antagonists in morphine-free rats.
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Animals↗
The effects of morphine and various narcotic antagonist type analgesics on body temperature in rats.
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Analgesics↗
Evidence that opiate receptors mediate suppression of hypertonic saline-induced drinking in the mouse by narcotic antagonists.
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Animals↗
Discriminative stimulus effects of narcotics: evidence for multiple receptor-mediated actions.
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Animals↗
Enhancement of apomorphine-induced climbing in mice by reversible and irreversible narcotic antagonist drugs.
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Animals↗
The possible role of intracellular receptors in the expression of narcotic antagonist precipitated abstinence.
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Animals↗
Narcotic antagonists attenuate drinking induced by water deprivation in a primate.
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Animals↗
Proceedings of the 1982 International Narcotic Research Conference. Part I.
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Analgesia↗
Differential analgesic cross-tolerance to morphine between lipophilic and hydrophilic narcotic agonists.
Mice were rendered tolerant to morphine by the subcutaneous implantation of one 75 mg morphine pellet. Seventy-two hours post-pellet implantation, the animals were evaluated in the tail-flick assay for analgesic tolerance and cross-tolerance to subcutaneously administered morphine, normorphine, methadone, etorphine and intracerebroventricularly administered morphine. With the pellet remaining in situ during testing, there was the expected analgesic tolerance to peripherally administered morphine and analgesic cross-tolerance to normorphine. However, with the pellet in situ during testing, there was a surprising lack of analgesic tolerance to intracerebroventricular administered morphine and no analgesic cross tolerance to peripherally administered etorphine or methadone. In contrast, removal of the morphine pellet 3 hours prior to the analgesic evaluation apparently unmasked the expression of tolerance and cross-tolerance as evidenced by a three fold, parallel shift to the right of the analgesic dose-response curve for subcutaneously administered etorphine and methadone and a seven fold shift to intracerebroventricularly administered morphine. These data emphasize that a more rigorous evaluation of tolerance development methodologies need be explored and support the suggestion that removal of the morphine-inducing pellet prior to analgesic determinations results in a distinct state of "tolerance" quite different from that observed with the pellet remaining in situ during testing.
Analgesics, Opioid↗