Influence of diethylstilbestrol on Drosophila melanogaster tumors.
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Animals↗
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Results for “DIETHYLSTILBESTROL”
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The effects of trace amounts of diethylstilbestrol in rations of fattening steers.
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Animals↗
Antimicrobial action as a basis for measurement of diethylstilbestrol.
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Anti-Infective Agents↗
Effect of diethylstilbestrol on substrate metabolism in Aerobacter aerogenes.
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Diethylstilbestrol↗
Induction of aneuploidy in mouse bone marrow cells with diethylstilbestrol-diphosphate.
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Aneuploidy↗
Inhibition of a microbial glucose-6-phosphate dehydrogenase by diethylstilbestrol.
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Diethylstilbestrol↗
Exploratory studies of the liver glutamic dehydrogenase of the hagfish Myxine glutinosa. Lack of regulation of activity by ADP and diethylstilbestrol in a physiological saline.
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Adenine Nucleotides↗
The effect of diethylstilbestrol on nitrogen excretion in sheep.
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Inhibition of hypothalamic obesity in the mouse with diethylstilbestrol.
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Effects of administering cortisone acetate and diethylstilbestrol to normal force-fed rats.
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Animals↗
Influence of diethylstilbestrol on the hemopoietic system of young mice.
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Actions of diethylstilbestrol in the albino rat.
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Effect of diethylstilbestrol on utilization of energy by the growing chick.
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Animals↗
Effect of adrenal steroids and diethylstilbestrol on growth and fat content of cockerels.
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Adrenal Cortex Hormones↗
Effects of diethylstilbestrol with thyroxin or thiouracil on thyroid in male rats.
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Animals↗
Effects on reproductive functions of diethylstilbestrol administered orally to gilts.
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Animals↗
Physiological actions of diethylstilbestrol in lambs fed varying levels of protein and energy.
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Animals↗
Effect of steroid hormones and diethylstilbestrol on adrenomedullary catecholamine secretion.
Acetylcholine-induced catecholamine secretion from isolated, perfused bovine adrenal medulla was strongly inhibited by hydrocortisone (30 micro M), estradiol (30 micro M), and estriol (30 micro M). Diethylistilbestrol (DES) (10 and 30 micro M) inhibited acetylcholine-induced secretion, and the effect of the higher dose was prolonged. Inhibition of high potassium-induced secretion by DES (30 micro M) indicated that hormonal inhibition of evoked secretion was not by competition for acetylcholine-sensitive receptor sites. Dehydrotestosterone (30 micro M) was ineffective in inhibiting acetylcholine-induced secrection. Hydrocortisone, estradiol, and estriol treatments enhanced nonstimulated release. It is suggested that incorporation of certain steroid into plasma and granule membranes, thereby modifying membrane function, enhances nonstimulated release and may also block evoked release of secretoy material.
Acetylcholine↗