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Results for “Alcoholic Intoxication”

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PubMed1991

Alcohol intoxication and sialic acid in erythrocyte membrane and in serum transferrin.

Microheterogeneity of serum transferrin as well as erythrocyte membrane sialic acid content were examined in alcoholic patients and healthy controls. Both the sialic acid content of erythrocyte membranes and of the circulating transferrin were significantly lower in alcoholic patients than in controls. A moderate daily ethanol intake (less than 80 g) allowed to observe a proportional relationship between alcohol intake and the carbohydrate deficient forms of transferrin, and also a correlation between alcohol intake and the membrane sialic acid content. This supports the hypothesis of ubiquitary alterations of glycosylations in connection to ethanol intoxication. Additional disturbances could explain the absence of correlations between membrane sialic acid, pattern of abnormal forms of serum transferrin, and alcohol intake in heavy alcoholic patients.

Adult↗
PubMedDate not supplied

[Biological markers in the accurate diagnosis of chronic alcohol intoxication--the significance of metabolic disorders (I)].

Continuing the series of papers dedicated to determining some objective markers for chronic alcohol poisonings, this time we insist on the significance of metabolic disturbances in the series under study. The hepatic changes are useful in the diagnosis, in determining the consequences and in following up the therapeutical management. The study was based on the marked alterations in the lipid and protein metabolisms, these alterations being strongly connected with ethanol metabolization stages.

Alcoholism↗
PubMed1992

[Effects of alcoholic intoxication in the antenatal period on the development of male germ cells].

During 1-20 days of pregnancy rats were given 20% alcohol orally in a dose 0.4 mg/kg bw. This led to a significant increase in the nuclear volume of germ cells in male 19-day embryos. A karyological analysis of 2-month offspring germ cell generations at stage VII of seminiferous epithelium cycle revealed a reliable decrease in the number of spermatids on step 7 of development after introduction of antenatal alcohol. The increased proportion of spermatocytes with meiotic chromosomes pathological behaviour due to alcohol was determined by the proposed quantitative analysis of meiotic divisions.

Animals↗