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At least 1,171 records · Page 65Linked to original sources
PubMed1987

[Initial paroxysmal manifestations of epilepsy in children].

The article is devoted to characteristics of the onset of epilepsy in children. First paroxysmal manifestations observed in 727 children and 600 adults have been subjected to a comparative analysis. The children are characterized by the predominance of primary generalized attacks over secondary generalized and focal paroxysms. Other age-related differences in the structure of paroxysms have been ascertained. Complicated initial nonconvulsive forms of paroxysms and their time course are described. The author calls attention to the difficulties of therapy in connection with a great variability of paroxysmal manifestations which necessitate further improvement in the existent classification of epileptic attacks.

Adolescent↗
PubMed1987

[Effect of cerebral organic changes on the age of manifestation of childhood epilepsy].

In the present study, the influence of organic alterations in the brain on the age of manifestation of epilepsy was investigated by means of cranial computer tomography (CCT) in 307 children and adolescents. 38% of these patients revealed abnormal morphological findings in the CCT. Whereas in children with primary generalized and partial epilepsy the vast majority (80% and 75% respectively) showed normal findings, this only applied to barely half of the patients with secondary generalized epilepsy. The pathological CCT findings were characterized mainly by diffuse alterations in primary generalized epilepsy. On the other hand, children with secondary generalized epilepsy showed above-average focal defects. The age of onset of the disease showed a close relation with the CCT findings. In patients with pathological CCT alterations, the epilepsy was manifested very much earlier than in the reference group. However, the special features of the age distribution in relation to the CCT findings only applied to secondary generalized epilepsy. Thus 80% of the diseases in the first year of life involved this form of epilepsy (in the vast majority, West syndrome), in which pathological CCT alterations were present almost without exception. These results made it evident that exogenous factors have a crucial effect on the age of manifestation, the clinical features and course of epilepsy.

Adolescent↗
PubMed1987

[Initial epileptic manifestations in adolescents].

Eighty-eight epileptic patients with a history of first attacks at the age of 12-17 years have been studied. The control group has been composed of 176 patients in whom the disease first manifested at other age periods. The disease in adolescents more often begins with generalized paroxysmal attacks without aura or other focal components. The etiology of epilepsy in adolescents versus young children is less associated with perinatal pathology. The overwhelming majority of adolescents show electroencephalographic changes already at the onset of the disease while the number of patients with marked organic cerebral alterations demonstrated by PEG or CT is relatively small. An insufficiently critical attitude of adolescents to the disease necessitates the inclusion of psychotherapeutic measures into multiple modality treatment.

Adolescent↗
PubMed1987

Metabolism of magnesium and zinc in patients treated with antiepileptic drugs and with magnesium lactate.

Fourty-nine patients were examined with epileptic seizures and values of serum and erythrocyte magnesium (sMg, RBC Mg) and serum zinc (sZn) were determined. Chronic deficiency in RBC Mg and sZn were confirmed. The percentage of patients with decreased RBC Mg increased with the time of antiepileptic treatment; significant lowering occurs after 5 years of treatment for RBC Mg and within 5 years for sZn. Various antiepileptics and their combinations have different effects on the degree of the lowering. The therapy of magnesium lactate produces significantly higher RBC Mg and sZn. The clinical, electroencephalographical and biochemical findings are improved.

Adult↗
PubMed1988

[Temporal lobe epilepsy with an onset at an age from 6 to 12 and from 12 to 16 years].

Temporal epilepsy features concerning its etiology, seizures distribution within sleep-wakefulness cycle, their rate, psychic changes, EEG characteristics, treatment effectiveness are considered as related to different ages of the disease onset: 6 to 12 and 12 to 16. Age-related forms of the primarily generalized epilepsy: pyknolepsy, juvenile and adolescent myoclonic epilepsy, are analyzed.

Adolescent↗
PubMed1988

Withdrawal of barbiturate anticonvulsant drugs: prospective controlled study.

A barbiturate (phenobarbital or primidone) was withdrawn over a period of 3 months from 25 institutionalized residents, all of whom had had three seizures or less in the past 6 months and were maintained on a nonsedating drug (phenytoin, carbamazepine, or valproic acid). Results were compared with a matched comparison group maintained on both drugs. Subjects withdrawn from primidone, but not those withdrawn from phenobarbital, had increased seizure frequency, probably due to withdrawal. After 14 months, seizure-free subjects withdrawn from barbiturates were no more likely to have had seizures than were comparison subjects. Barbiturates appear to be unnecessary and may be withdrawn.

Adolescent↗
PubMed1985

The electroencephalogram in patients with epilepsy.

The EEG is useful in evaluating seizure disorders by establishing or confirming the diagnosis of a seizure disorder, determining the type and focus of origin of the seizure, and helping make the distinction between an epileptic attack and a nonepileptic condition. The EEG also may show other abnormalities, such as focal slowing, that give a clue about the underlying disease process. A negative EEG does not exclude the diagnosis of epilepsy. Activating procedures such as hyperventilation, photic stimulation, and recording during sleep may help bring out epileptiform activity. Occasionally, repeated or more prolonged recordings are necessary to demonstrate the epileptiform abnormalities. On the other hand, epileptiform-like activity may be present in the EEG without being associated with a seizure disorder. As the diagnosis of epilepsy is a clinical one, the EEG, like any test, should not be used independently in making the diagnosis of epilepsy but should be interpreted in the context of the whole clinical setting.

Electroencephalography↗
PubMedDate not supplied

[Classification of clinico-electroencephalographic syndromes in epilepsy in the developmental age].

The author describes the clinical-electroencephalographic classification of eleven syndromes of epilepsy in developmental age used by himself, based on verification of 3613 successive patients aged from 0 to 18 years who were treated in the years 1970-1976 and followed up for 5 years in the Poznań Epilepsy Treatment Centre. On the ground of clinical status (seizures and neuropsychiatric findings) and serial EEG records 2746 patients (76%) could have been classified into these epilepsy types. The practical aim of the classification was identification of the focus and circuit of the seizure at a given structural-functional level or organization of the developing brain for guided comprehensive therapy.

Adolescent↗