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Results for “Color Vision Defects”

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At least 1,171 records · Page 65Linked to original sources
PubMed1966

Dalton bicentenary.

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Chemistry, Physical↗
PubMed1976

Binocular enhancement of color discrimination in a deutan.

A 31-year-old white male deutan produced reliably different profiles when examined binocularly and monocularly with a Farnsworth-Munsell 100-hue test. Discrimination in the long wavelengths improved under the binocular conditions. Intensive testing yielded no information to account for the phenomenon.

Adult↗
PubMed1979

Autosomal recessive incomplete achromatopsia with deutan luminosity.

Four patients in three different families had a form of autosomal recessive incomplete achromatopsia not previously described. The visual acuity was 6/18 to 6/60 (20/60 to 20/200) with minimal ophthalmoscopic abnormality and normal fluorescein angiogram. The photopic electroretinographic responses were present in all four patients; the fusion rate of 60 Hz was only slightly subnormal. The high-intensity scotopic response was subnormal. The patients failed color screening plates and accumulated over 400 errors with scotopic axis on the Farnsworth-Munsell 100-hue test. The Rayleigh match was abnormal, displaced toward the red primary, but with normal luminance. The photopic luminous efficiency function was similar to that of the deuteranope. Color matching revealed a trichromatic form of color vision mediated by long wavelength and short wavelength cones, and a rhodopsin receptor.

Adolescent↗
PubMed1982

Autosomal recessive vitreoretinopathy and encephaloceles.

We conducted a ten-year follow-up of an unusual pedigree with an autosomal recessive vitreoretinal degeneration, severe myopia, and congenital encephalocele. All five affected members (four girls and one boy) also had early, recurrent bilateral detachments. Color vision testing disclosed an acquired tritan dyschromatopsia and electroretinography showed subnormal photopic and scotopic amplitudes, delayed b-wave implicit times and 30-Hz flicker-phase relations, and absent scotopic b-wave oscillations.

Child↗
PubMed1982

Prosopagnosia.

Three patients with prosopagnosia, and acquired inability to recognize familiar faces usually resulting from cerebrovascular insufficiency, had left-sided visual field defects and color vision abnormalities of central origin. Prosopagnosia, although clinically associated with posterior right hemispheric disease in most cases, represents a disconnection of both occipital poles from the final processing center for facial recognition in the right temporal lobe. This problem can profoundly affect everyday activities requiring visual recognition.

Adult↗