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At least 1,135 records · Page 63Linked to original sources
PubMed1992

Ophthalmic laser surgery: therapeutic implications for the primary physician.

This paper represents the results of a retrospective study on the diagnosis and treatment of 102 consecutive patients who received laser surgery in a general ophthalmic practice during the period of August 1989 to September 1991. The purpose of the study is to document and interpret the type of eye disorders that lend themselves to definitive laser treatment. The goal of this article is to enhance the awareness of non-ophthalmologists in their pivotal and vital role in referring patients to an ophthalmologist for evaluation of the basic disease processes which, left untreated, can lead to vision impairment or blindness.

Cataract Extraction↗
PubMedDate not supplied

[Analysis of the prevalence of distichiasis in the dog breed Elo].

The Elo is a recently established dog breed in which the eye anomaly distichiasis has been frequently observed. Out of a total of 234 Elo dogs ophthalmologically examined over four years, 27.8% of the dogs showed between 1 and 31 distichia. There were twice as many distichia in the upper than in the lower eyelid. Litter size, the number of examinations of Elo dogs per ophthalmologist and the proportion of genes of the Eurasian dog had a significant influence on the prevalence of distichiasis. The error probability for the inbreeding coefficient and the proportion of genes of the Old English Sheepdog were just above the significance limit of p = 0.05. No significance was found for the remaining effects. Heritability for distichiasis was estimated using a linear animal model. After transformation of the linear observed estimate onto the underlying liability scale the heritability estimate was h2 = 0.43.

Animals↗
PubMed2006

Comparative lymphatic, ocular, and metabolic phenotypes of Foxc2 haploinsufficient and aP2-FOXC2 transgenic mice.

FOXC2 mutations cause the lymphatic/ocular disorder Lymphedema-Distichiasis (LD), and Foxc2 haploinsufficient mice mimic this disorder. To determine if FOXC2 overexpression might also cause lymphatic and/or ocular abnormalities, we performed dynamic lymphatic imaging (Evans blue dye), ocular tissue examination, and metabolic profiles in mice: transgenic for FOXC2 with an adipocyte (aP2) promoter (aP2-FOXC2 Tg), heterozygous for targeted disruption of Foxc2 (Foxc2+/-), or compound heterozygous and transgenic (Foxc2+/-, Tg) compared to wild-type controls (WT). Foxc2+/-; aP2-FOXC2 Tg; and Foxc2+/-, Tg, exhibited LD's distinctive hyperplastic lymphatic phenotype characterized by increased number of lymphatic channels and lymph nodes as well as retrograde lymph reflux. Foxc2+/-, and Foxc2+/-, Tg but not aP2-FOXC2 Tg or WT showed an abnormal ocular phenotype. Previously described alterations in brown/ white fat distribution and lean phenotype in aP2-FOXC2 transgenics were confirmed. AP2-FOXC2 Tg immunohistochemistry disclosed aberrant FOXC2 expression in ectopic sites, especially embryonic heart. Lymphatic system links with fat metabolism are discussed.

Adipocytes↗
PubMed1990

The management of trachomatous trichiasis.

Trachoma is the cause of one-quarter of world blindness. Corneal damage from trachoma is related to trichiasis, which may affect over 10% of adults in endemic communities. Relief of trichiasis is believed to prevent corneal damage and the provision of community-based lid surgery is a central strategy for trachoma control programs. This strategy is weakened by the dearth of information on the effectiveness of different trichiasis procedures. A systematic review of trichiasis surgery is presented and reports of treatment efficacy are reviewed. Conclusions are drawn as to which procedures seem most appropriate for trachomatous trichiasis in prevention of blindness programmes. It is suggested that further controlled clinical trials of appropriate trichiasis procedures are needed.

Blindness↗