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[Pulmonary emphysema--silicosis--cor pulmonale. Results of statistical investigations (author's transl)].
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[Experimental model of silicosis: a study using 22Na-labelled SiO2 (author's transl)].
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Pathogenesis of silicosis: current concepts and hypotheses.
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Effect of adrenocorticotropic hormone (ACTH) on beryllium granulomatosis and silicosis.
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The biochemistry of lungs in relation to silicosis. I. The amino acid content of normal guinea pig lung tissue.
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An immunologic study of experimental silicosis.
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Pathogenesis of pulmonary silicosis in rats fed stock and multideficient diet since weaning.
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Experimental studies on early stages of the development of pulmonary silicosis in pups.
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Silicosis in shipyard sandblasters.
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Changes of copper, iron, and zinc in the serum of patients with silicosis, silicotuberculosis, and active lung tuberculosis.
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Pulmonary fibrosis: bronchoalveolar cell types and impaired function of alveolar macrophages in experimental silicosis.
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The mechanism of hemolysis by silica and its bearing on silicosis.
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Lymph node test for silicosis by activation analysis combined with spectrophotometry.
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Histamine metabolism in experimental silicosis. I. Experiments on the isolated rat lung and on guinea pig lung homogenates and sections.
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Histamine metabolism in experimental silicosis. II. In vivo experiments in guinea pigs.
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Changes in mitochondrial enzyme activity of rat lung during the development of silicosis.
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Bronchoalveolar leukocyte response in experimental silicosis: modulation by a soluble aluminum compound.
The biological properties of quartz have been related to its surface reactivity. We have addressed the role of particle surface reactivity in mediating the biological activity of quartz in mixed dusts, by treating the quartz with aluminum lactate. Intratracheal instillation of untreated quartz in rats caused a rapid, sustained alveolitis and bronchoalveolar leukocytes obtained from these animals had enhanced activity in degrading fibronectin, but reduced ability to mount a respiratory burst. Quartz pretreated with aluminum elicited a markedly reduced inflammatory response; the reduced activity of the treated quartz was also reflected in the attenuated change in the key functional parameters, oxidant production and proteolysis of fibronectin. Late intratracheal dosing with aluminum after the quartz-induced alveolitis was well established reduced the inflammatory response and abrogated the effect of quartz on the respiratory burst, but did not alter fibronectin degradation by the leukocytes. Aluminum did not affect the inflammatory response to Corynebacterium parvum and thus the effect was on the quartz particles and not on the inflammatory leukocytes. These findings have implications for the likely pulmonary responses to mixed dusts containing quartz and aluminum silicate clays.