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Are the features of ankyloglossia limited to the lingual fraenum?

A 16-year-old male patient presented with ankyloglossia accompanied by a complaint of forceful spurting of saliva from the mouth and a unilateral swelling in the submandibular region. Following surgical relief of the ankyloglossia, the spurting of saliva had virtually ceased and the submandibular swelling disappeared without recurrence.

Adolescent↗

[The etiology of salivary calculi].

Ninety-four patients with chronic sialodochitis, 45 of these with sialolithiasis, have been followed up for a long time. Sialolithiasis develops in patients with congenital defects in the ductal system of the salivary gland presenting as ectasias and can be regarded as calculous sialoductitis. The etiological factors of sialolithiasis (calculous sialoductitis) are strictures (stenosis) of the ducts (distal portions) and specific topography of the duct (a "broken" duct with abrupt curves in which the concrements may form).

Adult↗

Intraoral biopsy of the parotid gland.

Although biopsy of the parotid gland is occasionally necessary for diagnostic purposes, surgeons hesitate to perform a biopsy because of the possible adverse effects, such as facial scarring or damage to facial nerves. An intraoral technique was used for biopsy of a parotid gland of a young woman. The procedure was successful; a diagnosis was made and there was no facial scarring. The potential hazards must be weighed against the benefits.

Adult↗

Submandibular duct sialolith of unusual size and shape.

An unusual sialoith within Wharton's duct was associated with an abscess of the submandibular space. Previously reported sialoiths of considerable size are reviewed, and etiology, symptomatology, and surgical removal are discussed. Emphasis is placed on the need for thorough examination of patients who have symptoms of sialoithiasis in an attempt to locate the sialolith before it assumes unusual dimensions.

Humans↗

[Calcified parotiditis].

Six cases of parotiditis with multiple concretions clearly seen on X-rays in front view and without contrast, and associated with parenchymatous cavitation, have led us to suggest the concept of calcified parotiditis. The process was seen to be bilateral each time such an eventuality was sought. Histological examination confirmed that the calcifications were situated in the small efferent canals and provides evidence in favour of the similarity between such forms of parotiditis and calcified pancreatitis. As with pancreatitis, it is difficult to say whether such calcifications are responsible for ascending infection or if they result from it.

Adult↗

Sjögren's syndrome.

Sjögren's syndrome (SS) is a progressive autoimmune rheumatic disorder. Its precise etiology is unknown, although several contributing factors have been identified. One theory is that the condition results from complications related to infection with the Epstein-Barr virus. Primary exposure to or reactivation of Epstein-Barr virus elicits expression of the human leukocyte antigen complex. This is recognized by T lymphocytes (CD 4+) resulting in the release of cytokines (tumor necrosis factor, interleukin-2, interferon-gamma, and others). A genetic marker specific for Sjögren's syndrome, HLA-DR4, has been identified. According to the World Health Organization, the prevalence of Sjögren's syndrome is unknown. A recent epidemiologic study in Sweden estimated the prevalence in the adult population to be 2.7%. In the United States, 10 years ago, the number of patients with Sjögren's syndrome was thought to be fewer than 100,000. This number today is estimated to be more than 1 million. Sjögren's syndrome has been reported in nearly every major country of the world, and the geographic distribution of cases appears to be relatively uniform. Sjögren's syndrome typically affects women (90%) during the fourth or fifth decade of life. Isolated cases of Sjögren's syndrome in children have been reported.

Adult↗

[Relapsing parotitis: onset of Sjögren's syndrome].

Report of a case of lymphocytic parotitis associated with important inflammatory syndrome, anti-salivary antibodies and rheumatoid factor. The study of the cases of literature suggests that it might be the mode of onset of a Sjögren's syndrome.

Antibody Formation↗