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Enzootic ataxia in red deer, Cervus elaphus.
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Copper methionate for parenteral copper therapy in sheep.
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Methods of copper supplementation.
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Breed, bodyweight and age age as factors in the mortality rate of sheep following copper injection.
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Concurrent enzootic ataxia and white muscle disease in a flock of lambs.
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Nutrition and the nervous system in farm animals.
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Histopathology of enzootic ataxia in Sika deer (Cervus nippon Temminck).
Thirteen Sika deer (Cervus nippon Temminck) showing ataxia on a stock farm in the north eastern district of China were examined histopathologically. The principal pathological changes were spongy vacuolation and myelin deficiency in the white matter of the spinal cord and brain stem, fibrosis and rupture of the elastic lamina of the spinal arterioles, and mesothelial hyperplasia in the spinal arachnoid. Other findings included defective formation of the elastic laminae of the aorta, and the blood vessels in the kidney and lung, hemosiderosis in the spleen and liver, and lymphocyte depletion in the systemic lymph nodes. Copper concentrations were low in the serum and liver. In the white matter of the spinal cord and brain stem, demyelination appeared to coexist with dysmyelination and secondary myelin breakdown. It was inferred that decreased activity of copper containing enzyme induced various lesions. The possible role of copper deficiency in the pathogenesis of the ataxic conditions is discussed.
Degenerating myelin: comparative histochemical studies using classical myelin stains and an improved Marchi technique minimizing artifacts.
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Enzootic ataxia in a Norwegian red deer herd.
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Enzootic ataxia in farmed red deer.
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Enzootic ataxia from copper deficiency in sheep in Colorado.
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[Porencephalia of the greater hemispheres of the brain in enzootic ataxia in lambs].
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Sheep as animal models in biomedical research.
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A rapid immunologic test for scrapie in sheep.
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[Demyelination diseases in the animal].
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Parenteral copper therapy in sheep.
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